Malarial infection develops mitochondrial pathology and mitochondrial oxidative stress to promote hepatocyte apoptosis.
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Understanding the Warburg effect and the prognostic value of stromal caveolin-1 as a marker of a lethal tumor microenvironmentN-acetyl-serotonin protects HepG2 cells from oxidative stress injury induced by hydrogen peroxideNovel anti-inflammatory activity of epoxyazadiradione against macrophage migration inhibitory factor: inhibition of tautomerase and proinflammatory activities of macrophage migration inhibitory factor.Stromal-epithelial metabolic coupling in cancer: integrating autophagy and metabolism in the tumor microenvironment.Liver pathology in Malawian children with fatal encephalopathySplenic architecture disruption and parasite-induced splenocyte activation and anergy in Plasmodium falciparum-infected Saimiri sciureus monkeys.Sterile protective immunity to malaria is associated with a panel of novel P. falciparum antigens.Tryptamine-gallic acid hybrid prevents non-steroidal anti-inflammatory drug-induced gastropathy: correction of mitochondrial dysfunction and inhibition of apoptosis in gastric mucosal cells.Safety and Reproducibility of a Clinical Trial System Using Induced Blood Stage Plasmodium vivax Infection and Its Potential as a Model to Evaluate Malaria TransmissionOxidative stress in malaria.Effect of Plasmodium falciparum malaria parasites on haematological parameters in Ghanaian childrenA key role for lipoic acid synthesis during Plasmodium liver stage developmentLipid peroxidation and antioxidant enzymes activity in Plasmodium vivax malaria patients evolving with cholestatic jaundice.Malaria evolution in South Asia: knowledge for control and elimination.Experimental study on the inhibitory effect of sodium cantharidinate on human hepatoma HepG2 cells.Liver changes in severe Plasmodium falciparum malaria: histopathology, apoptosis and nuclear factor kappa B expression.Redox sensing and signaling by malaria parasite in vertebrate host.Host-pathogen interactions in malaria: cross-kingdom signaling and mitochondrial regulation.Energy transfer in "parasitic" cancer metabolism: mitochondria are the powerhouse and Achilles' heel of tumor cells.Apoptosis induced by parasitic diseasesHost genetic variations in glutathione-S-transferases, superoxide dismutases and catalase genes influence susceptibility to malaria infection in an Indian population.Association of heme oxygenase 1 with the restoration of liver function after damage in murine malaria by Plasmodium yoelii.Impact of intravascular hemolysis in malaria on liver dysfunction: involvement of hepatic free heme overload, NF-κB activation, and neutrophil infiltration.Anti-plasmodial and anti-inflammatory activities of cyclotide-rich extract and fraction of Oldenlandia affinis (R. & S.) D.C. (Rubiaceae).Liver Injury in Uncomplicated Malaria is an Overlooked Phenomenon: An Observational StudyThe Impact of Nanochloroquine on Restoration of Hepatic and Splenic Mitochondrial Damage against Rodent Malaria
P2860
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P2860
Malarial infection develops mitochondrial pathology and mitochondrial oxidative stress to promote hepatocyte apoptosis.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Malarial infection develops mi ...... promote hepatocyte apoptosis.
@en
Malarial infection develops mi ...... promote hepatocyte apoptosis.
@nl
type
label
Malarial infection develops mi ...... promote hepatocyte apoptosis.
@en
Malarial infection develops mi ...... promote hepatocyte apoptosis.
@nl
prefLabel
Malarial infection develops mi ...... promote hepatocyte apoptosis.
@en
Malarial infection develops mi ...... promote hepatocyte apoptosis.
@nl
P2093
P921
P1476
Malarial infection develops mi ...... o promote hepatocyte apoptosis
@en
P2093
Athar Alam
Chinmay Pal
Kalyan Mitra
Manish Goyal
Mithu Guha
Samik Bindu
Uday Bandyopadhyay
P304
P356
10.1016/J.FREERADBIOMED.2008.10.032
P577
2008-10-30T00:00:00Z