ß-amyloid-related peptides potentiate K+-evoked glutamate release from adult rat hippocampal slices.
about
Levetiracetam suppresses neuronal network dysfunction and reverses synaptic and cognitive deficits in an Alzheimer's disease model.Amyloid-β acts as a regulator of neurotransmitter release disrupting the interaction between synaptophysin and VAMP2From single target to multitarget/network therapeutics in Alzheimer's therapyMemantine prevents memory consolidation failure induced by soluble beta amyloid in rats.Restored glial glutamate transporter EAAT2 function as a potential therapeutic approach for Alzheimer's diseasePreferential accumulation of amyloid-beta in presynaptic glutamatergic terminals (VGluT1 and VGluT2) in Alzheimer's disease cortex.Glutamate system, amyloid ß peptides and tau protein: functional interrelationships and relevance to Alzheimer disease pathologyGluN2B subunit-containing NMDA receptor antagonists prevent Abeta-mediated synaptic plasticity disruption in vivo.Reciprocal disruption of neuronal signaling and Aβ production mediated by extrasynaptic NMDA receptors: a downward spiral.Regulation of extracellular Zn(2+) homeostasis in the hippocampus as a therapeutic target for Alzheimer's disease.Is interaction of amyloid β-peptides with metals involved in cognitive activity?Glutamate transporter EAAT2: regulation, function, and potential as a therapeutic target for neurological and psychiatric disease.NMDA receptors and BAX are essential for Aβ impairment of LTP.Computational investigation of Amyloid-β-induced location- and subunit-specific disturbances of NMDAR at hippocampal dendritic spine in Alzheimer's disease.β-amyloid induces a dying-back process and remote trans-synaptic alterations in a microfluidic-based reconstructed neuronal network.β-amyloid and ATP-induced diffusional trapping of astrocyte and neuronal metabotropic glutamate type-5 receptors.Liver X Receptor Agonist GW3965 Regulates Synaptic Function upon Amyloid Beta Exposure in Hippocampal Neurons.Human Brain-Derived Aβ Oligomers Bind to Synapses and Disrupt Synaptic Activity in a Manner That Requires APP.Genome-wide association and interaction studies of CSF T-tau/Aβ42 ratio in ADNI cohort.Modulating the Balance of Synaptic and Extrasynaptic NMDA Receptors Shows Positive Effects against Amyloid-β-Induced Neurotoxicity.Differential Membrane Toxicity of Amyloid-β Fragments by Pore Forming Mechanisms.Synaptic dysfunction in Alzheimer's disease: the effects of amyloid beta on synaptic vesicle dynamics as a novel target for therapeutic intervention.
P2860
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P2860
ß-amyloid-related peptides potentiate K+-evoked glutamate release from adult rat hippocampal slices.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
ß-amyloid-related peptides pot ...... adult rat hippocampal slices.
@en
ß-amyloid-related peptides pot ...... adult rat hippocampal slices.
@nl
type
label
ß-amyloid-related peptides pot ...... adult rat hippocampal slices.
@en
ß-amyloid-related peptides pot ...... adult rat hippocampal slices.
@nl
prefLabel
ß-amyloid-related peptides pot ...... adult rat hippocampal slices.
@en
ß-amyloid-related peptides pot ...... adult rat hippocampal slices.
@nl
P2093
P1476
ß-amyloid-related peptides pot ...... adult rat hippocampal slices.
@en
P2093
P304
P356
10.1016/J.NEUROBIOLAGING.2008.08.009
P577
2008-09-25T00:00:00Z