Repeated cocaine administration induces gene expression changes through the dopamine D1 receptors. - Wikidata - awgldk - TriplyDB

Repeated cocaine administration induces gene expression changes through the dopamine D1 receptors.

Repeated cocaine administration induces gene expression changes through the dopamine D1 receptors.

http://www.wikidata.org/entity/Q46381667

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Targeting sigma receptors: novel medication development for drug abuse and addiction Cocaine inhibits dopamine D2 receptor signaling via sigma-1-D2 receptor heteromers Lack of self-administration of cocaine in dopamine D1 receptor knock-out mice Selective genomic targeting by FRA-2/FOSL2 transcription factor: regulation of the Rgs4 gene is mediated by a variant activator protein 1 (AP-1) promoter sequence/CREB-binding protein (CBP) mechanism Stimulants as specific inducers of dopamine-independent σ agonist self-administration in rats Cytosolic proteomic alterations in the nucleus accumbens of cocaine overdose victims Assessment of genome and proteome profiles in cocaine abuse Dopamine D1 and N-methyl-D-aspartate receptors and extracellular signal-regulated kinase mediate neuronal morphological changes induced by repeated cocaine administration Potential translational targets revealed by linking mouse grooming behavioral phenotypes to gene expression using public databases.Rats markedly escalate their intake and show a persistent susceptibility to reinstatement only when cocaine is injected rapidly.NK-3 receptor antagonism prevents behavioral sensitization to cocaine: a role of glycogen synthase kinase-3 in the nucleus accumbens.A Role for Sigma Receptors in Stimulant Self Administration and Addiction.Cocainomics: new insights into the molecular basis of cocaine addiction.Elevations of FosB in the nucleus accumbens during forced cocaine abstinence correlate with divergent changes in reward function.Cocaine-induced synaptic structural modification is differentially regulated by dopamine D1 and D3 receptors-mediated signaling pathways.Self-administration of cocaine induces dopamine-independent self-administration of sigma agonists.Distinct roles of dopamine D3 receptors in modulating methamphetamine-induced behavioral sensitization and ultrastructural plasticity in the shell of the nucleus accumbens.Comparison of dopamine D1 and D5 receptor knockout mice for cocaine locomotor sensitization.Fos regulates neuronal activity in the nucleus accumbens.Drosophila D1 dopamine receptor mediates caffeine-induced arousal.Addictive drugs modulate GIRK-channel signaling by regulating RGS proteins Augmented D1 dopamine receptor signaling and immediate-early gene induction in adult striatum after prenatal cocaine.Pharmacology and therapeutic potential of sigma(1) receptor ligands.Cocaine disrupts histamine H3 receptor modulation of dopamine D1 receptor signaling: σ1-D1-H3 receptor complexes as key targets for reducing cocaine's effects.Control of striatal signaling by g protein regulators.CNS genes implicated in relapse c-Fos is an intracellular regulator of cocaine-induced long-term changes.Alterations in subcellular expression of acid-sensing ion channels in the rat forebrain following chronic amphetamine administration.Zebrafish reward mutants reveal novel transcripts mediating the behavioral effects of amphetamine.Opposite regulation of cocaine-induced intracellular signaling and gene expression by dopamine D1 and D3 receptors.Exploring mechanisms underlying extinction of cue-elicited cocaine seeking Effects of ginseng saponin on acute cocaine-induced alterations in evoked dopamine release and uptake in rat brain nucleus accumbens.Transcriptomic profiling of the ventral tegmental area and nucleus accumbens in rhesus macaques following long-term cocaine self-administration.Long-lasting up-regulation of orexin receptor type 2 protein levels in the rat nucleus accumbens after chronic cocaine administration.Signaling via dopamine D1 and D3 receptors oppositely regulates cocaine-induced structural remodeling of dendrites and spines.Genetic or pharmacological inactivation of the dopamine D1 receptor differentially alters the expression of regulator of G-protein signalling (Rgs) transcripts.Acute amphetamine down-regulates RGS4 mRNA and protein expression in rat forebrain: distinct roles of D1 and D2 dopamine receptors.Active and passive MDMA ('ecstasy') intake induces differential transcriptional changes in the mouse brain.Everyday and prospective memory deficits in ecstasy/polydrug users.

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Repeated cocaine administration induces gene expression changes through the dopamine D1 receptors.

http://www.wikidata.org/entity/Q46381667

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Repeated cocaine administratio ...... ugh the dopamine D1 receptors.

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Repeated cocaine administratio ...... ugh the dopamine D1 receptors.

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Repeated cocaine administratio ...... ugh the dopamine D1 receptors.

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Repeated cocaine administratio ...... ugh the dopamine D1 receptors.

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Repeated cocaine administratio ...... ugh the dopamine D1 receptors.

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Neuropsychopharmacology

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Danwen Lou

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Dongsheng Zhang

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Frank R Sharp

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Jianhua Zhang

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Lu Zhang

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Ming Xu

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Qi Zhang

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Yang Tang

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Addiction and the brain: The neurobiology of compulsion and its persistence

Amphetamine and cocaine induce drug-specific activation of the c-fos gene in striosome-matrix compartments and limbic subdivisions of the striatum

Wrch-1, a novel member of the Rho gene family that is regulated by Wnt-1

Impaired B-lymphopoiesis, myelopoiesis, and derailed cerebellar neuron migration in CXCR4- and SDF-1-deficient mice

Addiction, dopamine, and the molecular mechanisms of memory

Involvement of sigma receptors in the behavioral effects of cocaine: evidence from novel ligands and antisense oligodeoxynucleotides

The molecular basis of CaMKII function in synaptic and behavioural memory

D1 and D2 receptor antagonists differently affect cocaine-induced locomotor hyperactivity in the mouse

Effects of dopamine D(1-like) and D(2-like) agonists on cocaine self-administration in rhesus monkeys: rapid assessment of cocaine dose-effect functions

Dopamine D1 receptor mutant mice are deficient in striatal expression of dynorphin and in dopamine-mediated behavioral responses

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