Theoretical study of L-type Ca(2+) current inactivation kinetics during action potential repolarization and early afterdepolarizations.
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Calcium movements inside the sarcoplasmic reticulum of cardiac myocytesCalmodulin regulation (calmodulation) of voltage-gated calcium channelsIon Channels in the HeartTowards a Unified Theory of Calmodulin Regulation (Calmodulation) of Voltage-Gated Calcium and Sodium ChannelsCa(2+) current facilitation is CaMKII-dependent and has arrhythmogenic consequencesPro-arrhythmogenic effects of CACNA1C G1911R mutation in human ventricular tachycardia: insights from cardiac multi-scale modelsMicrodomain-Specific Modulation of L-Type Calcium Channels Leads to Triggered Ventricular Arrhythmia in Heart Failure.Mechanistic Investigation of the Arrhythmogenic Role of Oxidized CaMKII in the HeartArrhythmogenesis in Timothy Syndrome is associated with defects in Ca(2+)-dependent inactivationβ-adrenergic stimulation activates early afterdepolarizations transiently via kinetic mismatch of PKA targets.Temperature-dependence of L-type Ca(2+) current in ventricular cardiomyocytes of the Alaska blackfish (Dallia pectoralis)Sensitivity of rabbit ventricular action potential and Ca²⁺ dynamics to small variations in membrane currents and ion diffusion coefficients.Do t-tubules play a role in arrhythmogenesis in cardiac ventricular myocytes?Rabbit-specific computational modelling of ventricular cell electrophysiology: Using populations of models to explore variability in the response to ischemia.Computational Cardiac Modeling Reveals Mechanisms of Ventricular Arrhythmogenesis in Long QT Syndrome Type 8: CACNA1C R858H Mutation Linked to Ventricular Fibrillation.Calmodulin mutations associated with long QT syndrome prevent inactivation of cardiac L-type Ca(2+) currents and promote proarrhythmic behavior in ventricular myocytes.Mechanism of doxorubicin cardiotoxicity evaluated by integrating multiple molecular effects into a biophysical model.Mitochondrial Dysfunctions Contribute to Hypertrophic Cardiomyopathy in Patient iPSC-Derived Cardiomyocytes with MT-RNR2 Mutation.Altered Ca and Na Homeostasis in Human Hypertrophic Cardiomyopathy: Implications for Arrhythmogenesis
P2860
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P2860
Theoretical study of L-type Ca(2+) current inactivation kinetics during action potential repolarization and early afterdepolarizations.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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2012年學術文章
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name
Theoretical study of L-type Ca
@nl
Theoretical study of L-type Ca ...... nd early afterdepolarizations.
@en
type
label
Theoretical study of L-type Ca
@nl
Theoretical study of L-type Ca ...... nd early afterdepolarizations.
@en
prefLabel
Theoretical study of L-type Ca
@nl
Theoretical study of L-type Ca ...... nd early afterdepolarizations.
@en
P2093
P2860
P1476
Theoretical study of L-type Ca ...... nd early afterdepolarizations.
@en
P2093
Aurora Summa
Eleonora Grandi
Kenneth S Ginsburg
Stefano Morotti
P2860
P304
P356
10.1113/JPHYSIOL.2012.231886
P407
P577
2012-05-14T00:00:00Z