Kisspeptin-GPR54 signaling is essential for preovulatory gonadotropin-releasing hormone neuron activation and the luteinizing hormone surge.
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Long-term effects of environmental endocrine disruptors on reproductive physiology and behaviorPI3K: An Attractive Candidate for the Central Integration of Metabolism and ReproductionThe excitatory peptide kisspeptin restores the luteinizing hormone surge and modulates amino acid neurotransmission in the medial preoptic area of middle-aged ratsComprehensive Review on Kisspeptin and Its Role in Reproductive DisordersPacemaking kisspeptin neuronsAfferent neuronal control of type-I gonadotropin releasing hormone neurons in the humanEstradiol Membrane-Initiated Signaling and Female ReproductionLoss of Fertility in the Absence of Progesterone Receptor Expression in Kisspeptin Neurons of Female MicePeripheral and Central Mechanisms Involved in the Hormonal Control of Male and Female ReproductionThe kisspeptin-GnRH pathway in human reproductive health and diseaseKisspeptin signaling in the brainSuppression of β1-integrin in gonadotropin-releasing hormone cells disrupts migration and axonal extension resulting in severe reproductive alterationsKisspeptin excitation of GnRH neuronsThe pros and cons of phytoestrogensStriatal GDNF Production Is Independent to Circulating Estradiol Level Despite Pan-Neuronal Activation in the Female MouseApplying gene silencing technology to contraceptionTargeted gene silencing to induce permanent sterilityCentral circadian control of female reproductive functionAblation of KNDy Neurons Results in Hypogonadotropic Hypogonadism and Amplifies the Steroid-Induced LH Surge in Female Rats.ERα in Tac2 Neurons Regulates Puberty Onset in Female Mice.Deletion of Vax1 from Gonadotropin-Releasing Hormone (GnRH) Neurons Abolishes GnRH Expression and Leads to Hypogonadism and Infertility.Absent Progesterone Signaling in Kisspeptin Neurons Disrupts the LH Surge and Impairs Fertility in Female MiceGonadotropin and kisspeptin gene expression, but not GnRH, are impaired in cFOS deficient mice.Positive, but not negative feedback actions of estradiol in adult female mice require estrogen receptor α in kisspeptin neuronsKisspeptin cell-specific PI3K signaling regulates hypothalamic kisspeptin expression and participates in the regulation of female fertility.Identification of neuropeptide receptors expressed by melanin-concentrating hormone neurons.Impaired GABAB receptor signaling dramatically up-regulates Kiss1 expression selectively in nonhypothalamic brain regions of adult but not prepubertal mice.Developmental GnRH signaling is not required for sexual differentiation of kisspeptin neurons but is needed for maximal Kiss1 gene expression in adult femalesRedundancy in Kiss1 expression safeguards reproduction in the mouse.Kisspeptin regulates gonadotropin genes via immediate early gene induction in pituitary gonadotropesAnalysis of multiple positive feedback paradigms demonstrates a complete absence of LH surges and GnRH activation in mice lacking kisspeptin signaling.Sexually dimorphic testosterone secretion in prenatal and neonatal mice is independent of kisspeptin-Kiss1r and GnRH signaling.Voltage-gated potassium currents are targets of diurnal changes in estradiol feedback regulation and kisspeptin action on gonadotropin-releasing hormone neurons in mice.Differential effects of hypothalamic IGF-I on gonadotropin releasing hormone neuronal activation during steroid-induced LH surges in young and middle-aged female rats.The neurobiology of preovulatory and estradiol-induced gonadotropin-releasing hormone surges.Beyond Leptin: Emerging Candidates for the Integration of Metabolic and Reproductive Function during Negative Energy Balance.Hypothalamic dysregulation and infertility in mice lacking the homeodomain protein Six6.Characterisation of arcuate nucleus kisspeptin/neurokinin B neuronal projections and regulation during lactation in the rat.Forebrain projections of arcuate neurokinin B neurons demonstrated by anterograde tract-tracing and monosodium glutamate lesions in the rat.Kisspeptin increases gamma-aminobutyric acidergic and glutamatergic transmission directly to gonadotropin-releasing hormone neurons in an estradiol-dependent manner.
P2860
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P2860
Kisspeptin-GPR54 signaling is essential for preovulatory gonadotropin-releasing hormone neuron activation and the luteinizing hormone surge.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh-hant
name
Kisspeptin-GPR54 signaling is ...... the luteinizing hormone surge.
@en
Kisspeptin-GPR54 signaling is ...... the luteinizing hormone surge.
@nl
type
label
Kisspeptin-GPR54 signaling is ...... the luteinizing hormone surge.
@en
Kisspeptin-GPR54 signaling is ...... the luteinizing hormone surge.
@nl
prefLabel
Kisspeptin-GPR54 signaling is ...... the luteinizing hormone surge.
@en
Kisspeptin-GPR54 signaling is ...... the luteinizing hormone surge.
@nl
P50
P1476
Kisspeptin-GPR54 signaling is ...... the luteinizing hormone surge
@en
P2093
Xavier d'Anglemont de Tassigny
P304
P356
10.1523/JNEUROSCI.1775-08.2008
P407
P577
2008-08-01T00:00:00Z