Cardiomyocyte apoptosis induced by short-term diabetes requires mitochondrial GSH depletion. - Wikidata - awgldk - TriplyDB

Cardiomyocyte apoptosis induced by short-term diabetes requires mitochondrial GSH depletion.

Cardiomyocyte apoptosis induced by short-term diabetes requires mitochondrial GSH depletion.

http://www.wikidata.org/entity/Q46417518

about

Increased propensity for cell death in diabetic human heart is mediated by mitochondrial-dependent pathways Enhanced apoptotic propensity in diabetic cardiac mitochondria: influence of subcellular spatial location Reactive oxygen species, cellular redox systems, and apoptosis Diabetic Cardiomyopathy in OVE26 Mice Shows Mitochondrial ROS Production and Divergence Between In Vivo and In Vitro Contractility Mitochondrial glutathione, a key survival antioxidant.Metabolic and Biochemical Stressors in Diabetic Cardiomyopathy.The effect of Astragalus polysaccharides on attenuation of diabetic cardiomyopathy through inhibiting the extrinsic and intrinsic apoptotic pathways in high glucose -stimulated H9C2 cells.Protein kinase RNA-like endoplasmic reticulum kinase (PERK) signaling pathway plays a major role in reactive oxygen species (ROS)-mediated endoplasmic reticulum stress-induced apoptosis in diabetic cardiomyopathy.Glutathione and apoptosis.Updating experimental models of diabetic cardiomyopathy Altered glutathione homeostasis in heart augments cardiac lipotoxicity associated with diet-induced obesity in mice.Glutamine Reduces the Apoptosis of H9C2 Cells Treated with High-Glucose and Reperfusion through an Oxidation-Related Mechanism.Effects of N-acetylcysteine on nicotinamide dinucleotide phosphate oxidase activation and antioxidant status in heart, lung, liver and kidney in streptozotocin-induced diabetic rats.Glutathione and modulation of cell apoptosis.Glutathione efflux and cell death.Depletion of cellular glutathione modulates LIF-induced JAK1-STAT3 signaling in cardiac myocytes.Nonischemic heart failure in diabetes mellitus.Type II diabetes increases mitochondrial DNA mutations in the left ventricle of the Goto-Kakizaki diabetic rat.Effect of Exercise Training on Skeletal Muscle SIRT1 and PGC-1α Expression Levels in Rats of Different Age.Mitochondrial pathophysiology, reactive oxygen species, and cardiovascular diseases Moderate exercise attenuates caspase-3 activity, oxidative stress, and inhibits progression of diabetic renal disease in db/db mice.Glutathione Depletion by L-Buthionine-S,R-Sulfoximine Induces Apoptosis of Cardiomyocytes through Activation of PKC-δ.Effect of increasing glutathione with cysteine and glycine supplementation on mitochondrial fuel oxidation, insulin sensitivity, and body composition in older HIV-infected patients.Vestibular rehabilitation ameliorates chronic dizziness through the SIRT1 axis.Phospholipase C as a potential target for cardioprotection during oxidative stress.CYP3A5 mediates bioactivation and cytotoxicity of tetrandrine.Glutathione administration reduces mitochondrial damage and shifts cell death from necrosis to apoptosis in ageing diabetic mice hearts during exercise.Alterations in sarcoplasmic reticulum and mitochondrial functions in diabetic cardiomyopathy.Cardiomyocyte H9c2 Cells Exhibit Differential Sensitivity to Intracellular Reactive Oxygen Species Generation with Regard to Their Hypertrophic vs Death Responses to Exogenously Added Hydrogen Peroxide.Cardiovascular Protective Effects of Salvianic Acid A on db/db Mice with Elevated Homocysteine Level.Oxidative modification of citrate synthase by peroxyl radicals and protection with novel antioxidants.Impaired mitochondrial fatty acid oxidation and insulin resistance in aging: novel protective role of glutathione.Propagation of the cardiac impulse in the diabetic rat heart: reduced conduction reserve.Treatment of acute paraquat intoxication using recommended megadose of vitamin C: a reappraisal.Calcium-calmodulin-dependent protein kinase mediates the intracellular signalling pathways of cardiac apoptosis in mice with impaired glucose tolerance.Exercise restores coronary vascular function independent of myogenic tone or hyperglycemic status in db/db mice.Effects of Resveratrol Supplementation and Exercise Training on Exercise Performance in Middle-Aged Mice.

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P2860

Cardiomyocyte apoptosis induced by short-term diabetes requires mitochondrial GSH depletion.

http://www.wikidata.org/entity/Q46417518

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2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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2005年學術文章

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name

Cardiomyocyte apoptosis induce ...... s mitochondrial GSH depletion.

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Cardiomyocyte apoptosis induce ...... s mitochondrial GSH depletion.

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Cardiomyocyte apoptosis induce ...... s mitochondrial GSH depletion.

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AJPHEART.00038.2005

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American Journal of Physiology - Heart and Circulatory Physiology

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Cardiomyocyte apoptosis induce ...... s mitochondrial GSH depletion.

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P2093

Ashraf Abrahani

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Brian Rodrigues

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Dake Qi

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Ding An

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Girish Kewalramani

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Gloria Yuen

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Sanjoy Ghosh

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P2860

The pathophysiology of mitochondrial cell death

Direct evidence for tumor necrosis factor-induced mitochondrial reactive oxygen intermediates and their involvement in cytotoxicity

Hyperglycemia activates p53 and p53-regulated genes leading to myocyte cell death.

Redox regulation of cardiomyocyte survival and death.

Molecular mechanisms of apoptosis in the cardiac myocyte.

Detection of reactive oxygen and reactive nitrogen species in skeletal muscle.

Glucose and reactive oxygen species.

Cell death and diabetic cardiomyopathy.

Effect of chronic ethanol feeding on rat hepatocytic glutathione. Compartmentation, efflux, and response to incubation with ethanol

Prevention of mitochondrial oxidative damage as a therapeutic strategy in diabetes.

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