Hypoxia-inducible factor 1-alpha reduces infarction and attenuates progression of cardiac dysfunction after myocardial infarction in the mouse.
about
Hypoxia-based strategies for angiogenic induction: the dawn of a new era for ischemia therapy and tissue regenerationCardiac regeneration: epicardial mediated repairHIF hydroxylase pathways in cardiovascular physiology and medicineComparative approaches to the study of physiology: Drosophila as a physiological tool.Hypoxia-inducible factor-dependent degeneration, failure, and malignant transformation of the heart in the absence of the von Hippel-Lindau proteinImpact of the phosphatidylinositide 3-kinase signaling pathway on the cardioprotection induced by intermittent hypoxiaMild hypoxia-induced cardiomyocyte hypertrophy via up-regulation of HIF-1α-mediated TRPC signallingThe impact of O2 availability on human cancerExperimental study of the effects of marrow mesenchymal stem cells transfected with hypoxia-inducible factor-1alpha geneA Phase I study of safety and pharmacokinetics of fruquintinib, a novel selective inhibitor of vascular endothelial growth factor receptor-1, -2, and -3 tyrosine kinases in Chinese patients with advanced solid tumors.Whole blood gene expression and atrial fibrillation: the Framingham Heart Study.Conditional HIF-1alpha expression produces a reversible cardiomyopathy.Impaired Ca(2+)-handling in HIF-1alpha(+/-) mice as a consequence of pressure overload.Understanding and managing toxicities of vascular endothelial growth factor (VEGF) inhibitorsAstragaloside attenuates myocardial injury in a rat model of acute myocardial infarction by upregulating hypoxia inducible factor‑1α and Notch1/Jagged1 signaling.Myocardial transfection of hypoxia-inducible factor-1α and co-transplantation of mesenchymal stem cells enhance cardiac repair in rats with experimental myocardial infarction.Manipulation of a VEGF-Notch signaling circuit drives formation of functional vascular endothelial progenitors from human pluripotent stem cells.Electron transport chain dysfunction in neonatal pressure-overload hypertrophy precedes cardiomyocyte apoptosis independent of oxidative stress.Management of side effects associated with sunitinib therapy for patients with renal cell carcinoma.Cardiac-specific overexpression of HIF-1{alpha} prevents deterioration of glycolytic pathway and cardiac remodeling in streptozotocin-induced diabetic mice.Regulation of phosphatase and tensin homolog on chromosome 10 in response to hypoxia.Loss of hypoxia-inducible factor prolyl hydroxylase activity in cardiomyocytes phenocopies ischemic cardiomyopathy.Period2 deficiency blunts hypoxia-induced mobilization and function of endothelial progenitor cells.l-Tetrahydropalmatine, an active component of Corydalis yanhusuo W.T. Wang, protects against myocardial ischaemia-reperfusion injury in rats.Exercise training restores cardiac protein quality control in heart failure.Redox signaling in cardiac myocytes.Hypoxia. 2. Hypoxia regulates cellular metabolismCardiomyocyte-specific prolyl-4-hydroxylase domain 2 knock out protects from acute myocardial ischemic injuryPreventive effect of yuzu and hesperidin on left ventricular remodeling and dysfunction in rat permanent left anterior descending coronary artery occlusion model.Oxygen Sensing, Cardiac Ischemia, HIF-1α and Some Emerging ConceptsDepletion of PHD3 protects heart from ischemia/reperfusion injury by inhibiting cardiomyocyte apoptosis.A-kinase anchoring proteins: scaffolding proteins in the heart.Systems analysis of gene ontology and biological pathways involved in post-myocardial infarction responses.Qiliqiangxin improves cardiac function and attenuates cardiac remodeling in rats with experimental myocardial infarction.PHD2/3-dependent hydroxylation tunes cardiac response to β-adrenergic stress via phospholamban.Hypoxia-inducible factor 1 transcriptional activity in endothelial cells is required for acute phase cardioprotection induced by ischemic preconditioning.Inhibition of the oxygen sensor PHD2 in the liver improves survival in lactic acidosis by activating the Cori cycle.Cardiac responses to hypoxia and reoxygenation in Drosophila.Somatic inactivation of the PHD2 prolyl hydroxylase causes polycythemia and congestive heart failure.Gene therapy progress and prospects: therapeutic angiogenesis for ischemic cardiovascular disease.
P2860
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P2860
Hypoxia-inducible factor 1-alpha reduces infarction and attenuates progression of cardiac dysfunction after myocardial infarction in the mouse.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Hypoxia-inducible factor 1-alp ...... rdial infarction in the mouse.
@en
Hypoxia-inducible factor 1-alp ...... rdial infarction in the mouse.
@nl
type
label
Hypoxia-inducible factor 1-alp ...... rdial infarction in the mouse.
@en
Hypoxia-inducible factor 1-alp ...... rdial infarction in the mouse.
@nl
prefLabel
Hypoxia-inducible factor 1-alp ...... rdial infarction in the mouse.
@en
Hypoxia-inducible factor 1-alp ...... rdial infarction in the mouse.
@nl
P2093
P1476
Hypoxia-inducible factor 1-alp ...... rdial infarction in the mouse.
@en
P2093
Christopher C Sullivan
Lingling Du
Masakuni Kido
Patricia A Thistlethwaite
Reena Deutsch
Stuart W Jamieson
Xiaodong Li
P304
P356
10.1016/J.JACC.2005.08.045
P407
P577
2005-11-09T00:00:00Z