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Hexokinase II detachment from mitochondria triggers apoptosis through the permeability transition pore independent of voltage-dependent anion channelsThe mitochondrial permeability transition from yeast to mammalsDimers of mitochondrial ATP synthase form the permeability transition poreRegulation of the inner membrane mitochondrial permeability transition by the outer membrane translocator protein (peripheral benzodiazepine receptor)Cyclophilin D modulates mitochondrial F0F1-ATP synthase by interacting with the lateral stalk of the complexPhosphate is essential for inhibition of the mitochondrial permeability transition pore by cyclosporin A and by cyclophilin D ablationThe mitochondrial permeability transition, release of cytochrome c and cell death. Correlation with the duration of pore openings in situ.The mitochondrial effects of small organic ligands of BCL-2: sensitization of BCL-2-overexpressing cells to apoptosis by a pyrimidine-2,4,6-trione derivative.Mitochondria and cell death. Mechanistic aspects and methodological issues.The effects of idebenone on mitochondrial bioenergeticsProperties of the permeability transition pore in mitochondria devoid of Cyclophilin D.Melanocytes from Patients Affected by Ullrich Congenital Muscular Dystrophy and Bethlem Myopathy have Dysfunctional Mitochondria That Can be Rescued with Cyclophilin Inhibitors.Assessing the molecular basis for rat-selective induction of the mitochondrial permeability transition by norbormide.Enhancement of anxiety, facilitation of avoidance behavior, and occurrence of adult-onset obesity in mice lacking mitochondrial cyclophilin DPeptide-based carbon nanotubes for mitochondrial targeting.Properties of Ca(2+) transport in mitochondria of Drosophila melanogasterThe mitochondrial permeability transition from in vitro artifact to disease target.Calcium and regulation of the mitochondrial permeability transition.Respiratory complex I dysfunction due to mitochondrial DNA mutations shifts the voltage threshold for opening of the permeability transition pore toward resting levels.Overexpression of the stress protein Grp94 reduces cardiomyocyte necrosis due to calcium overload and simulated ischemia.Mitochondria are direct targets of the lipoxygenase inhibitor MK886. A strategy for cell killing by combined treatment with MK886 and cyclooxygenase inhibitors.Chloromethyltetramethylrosamine (Mitotracker Orange) induces the mitochondrial permeability transition and inhibits respiratory complex I. Implications for the mechanism of cytochrome c release.The permeability transition pore as a mitochondrial calcium release channel: a critical appraisal.Alisporivir rescues defective mitochondrial respiration in Duchenne muscular dystrophy.A p53-p66Shc signalling pathway controls intracellular redox status, levels of oxidation-damaged DNA and oxidative stress-induced apoptosis.Arachidonic acid causes cell death through the mitochondrial permeability transition. Implications for tumor necrosis factor-alpha aopototic signaling.Mitochondrial alterations induced by the p13II protein of human T-cell leukemia virus type 1. Critical role of arginine residues.Apoptosis to necrosis switching downstream of apoptosome formation requires inhibition of both glycolysis and oxidative phosphorylation in a BCL-X(L)- and PKB/AKT-independent fashion.The inner mitochondrial membrane contains ion-conducting channels similar to those found in bacteria.Arachidonic acid released by phospholipase A(2) activation triggers Ca(2+)-dependent apoptosis through the mitochondrial pathway.Induction of the mitochondrial permeability transition by the DNA alkylating agent N-methyl-N'-nitro-N-nitrosoguanidine. Sorting cause and consequence of mitochondrial dysfunction.Modulation of mitochondrial K(+) permeability and reactive oxygen species production by the p13 protein of human T-cell leukemia virus type 1.Discrimination between two steps in the mitochondrial permeability transition process.Species-specific modulation of the mitochondrial permeability transition by norbormide.The unique histidine in OSCP subunit of F-ATP synthase mediates inhibition of the permeability transition pore by acidic pH.Reciprocal relationships between the resistance to stresses and cellular aging.Ca2+ binding to F-ATP synthase β subunit triggers the mitochondrial permeability transition.A glutamine synthetase inhibitor increases survival and decreases cytokine response in a mouse model of acute liver failure.Commitment to apoptosis by GD3 ganglioside depends on opening of the mitochondrial permeability transition pore.Effects of fatty acids on mitochondria: implications for cell death.
P50
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P50
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P1053
C-3558-2015
P106
P21
P31
P3829
P496
0000-0003-4026-6404