MKL1 mediates TGF-beta1-induced alpha-smooth muscle actin expression in human renal epithelial cells. - Wikidata - awgldk - TriplyDB

MKL1 mediates TGF-beta1-induced alpha-smooth muscle actin expression in human renal epithelial cells.

MKL1 mediates TGF-beta1-induced alpha-smooth muscle actin expression in human renal epithelial cells.

http://www.wikidata.org/entity/Q46612028

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From tissue mechanics to transcription factors.Mechanobiology of myofibroblast adhesion in fibrotic cardiac disease TGF-β-induced mesenchymal transition of MS-1 endothelial cells requires Smad-dependent cooperative activation of Rho signals and MRTF-A Fate-determining mechanisms in epithelial-myofibroblast transition: major inhibitory role for Smad3 Local delivery of novel MRTF/SRF inhibitors prevents scar tissue formation in a preclinical model of fibrosis.Prostaglandin E2 inhibits α-smooth muscle actin transcription during myofibroblast differentiation via distinct mechanisms of modulation of serum response factor and myocardin-related transcription factor-A Gene expression in nontumoral liver tissue and recurrence-free survival in hepatitis C virus-positive hepatocellular carcinoma Tissue geometry patterns epithelial-mesenchymal transition via intercellular mechanotransduction Immunocytochemical characterisation of cultures of human bladder mucosal cells Myocardin-related transcription factor-a controls myofibroblast activation and fibrosis in response to myocardial infarction.Distinct mesenchymal alterations in N-cadherin and E-cadherin positive primary renal epithelial cells The Purα/Purβ single-strand DNA-binding proteins attenuate smooth-muscle actin gene transactivation in myofibroblasts.Cell adhesion and shape regulate TGF-beta1-induced epithelial-myofibroblast transition via MRTF-A signaling.MKL1 inhibits cell cycle progression through p21 in podocytes RPEL proteins are the molecular targets for CCG-1423, an inhibitor of Rho signaling.Stereospecific Inhibitory Effects of CCG-1423 on the Cellular Events Mediated by Myocardin-Related Transcription Factor A.Regulation of Epithelial-Mesenchymal Transition by Transmission of Mechanical Stress through Epithelial Tissues Matrix stiffness-induced myofibroblast differentiation is mediated by intrinsic mechanotransduction.Transcriptional control of cardiac fibroblast plasticity.Regulation of myocardin factor protein stability by the LIM-only protein FHL2 Cell-cell contact and matrix adhesion promote αSMA expression during TGFβ1-induced epithelial-myofibroblast transition via Notch and MRTF-A Novel Rho/MRTF/SRF inhibitors block matrix-stiffness and TGF-β-induced fibrogenesis in human colonic myofibroblasts Sphingosine-1-phosphate and sphingosine kinase are critical for transforming growth factor-beta-stimulated collagen production by cardiac fibroblasts Activation of MRTF-A-dependent gene expression with a small molecule promotes myofibroblast differentiation and wound healing RhoA/ROCK signaling regulates TGFβ-induced epithelial-mesenchymal transition of lens epithelial cells through MRTF-A SAP domain-dependent Mkl1 signaling stimulates proliferation and cell migration by induction of a distinct gene set indicative of poor prognosis in breast cancer patients.TGFBR1 is required for mouse myometrial development.The actin-MRTF-SRF gene regulatory axis and myofibroblast differentiation.Biomechanics of TGFβ-induced epithelial-mesenchymal transition: implications for fibrosis and cancer Dynamic Interplay of Smooth Muscle α-Actin Gene-Regulatory Proteins Reflects the Biological Complexity of Myofibroblast Differentiation.Cytoskeletal signaling in TGFβ-induced epithelial-mesenchymal transition.Critical role of serum response factor in podocyte epithelial-mesenchymal transition of diabetic nephropathy.Cardiac Fibroblast Activation Post-Myocardial Infarction: Current Knowledge Gaps.A novel inhibitory mechanism of MRTF-A/B on the ICAM-1 gene expression in vascular endothelial cells.β-catenin and Smad3 regulate the activity and stability of myocardin-related transcription factor during epithelial-myofibroblast transition.Trichostatin A inhibits radiation-induced epithelial-to-mesenchymal transition in the alveolar epithelial cells.Transglutaminase-2 mediates calcium-regulated crosslinking of the Y-box 1 (YB-1) translation-regulatory protein in TGFβ1-activated myofibroblasts.Matrix Rigidity Mediates TGFβ1-Induced Epithelial-Myofibroblast Transition by Controlling Cytoskeletal Organization and MRTF-A Localization.Cardiac fibrosis: new insights into the pathogenesis

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MKL1 mediates TGF-beta1-induced alpha-smooth muscle actin expression in human renal epithelial cells.

http://www.wikidata.org/entity/Q46612028

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

@zh-cn

2008年学术文章

@zh-hans

2008年学术文章

@zh-my

2008年学术文章

@zh-sg

2008年學術文章

@yue

2008年學術文章

@zh-hant

name

MKL1 mediates TGF-beta1-induce ...... human renal epithelial cells.

@en

MKL1 mediates TGF-beta1-induce ...... human renal epithelial cells.

@nl

type

label

MKL1 mediates TGF-beta1-induce ...... human renal epithelial cells.

@en

MKL1 mediates TGF-beta1-induce ...... human renal epithelial cells.

@nl

prefLabel

MKL1 mediates TGF-beta1-induce ...... human renal epithelial cells.

@en

MKL1 mediates TGF-beta1-induce ...... human renal epithelial cells.

@nl

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AJPRENAL.00142.2007

P1433

American Journal of Physiology - Renal Physiology

P1476

MKL1 mediates TGF-beta1-induce ...... human renal epithelial cells.

@en

P2093

Charlotte J Logan

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Dorit Elberg

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Gerard Elberg

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Lijuan Chen

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Martin A Turman

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Michael D Chan

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P2860

Signal-regulated activation of serum response factor is mediated by changes in actin dynamics

Megakaryoblastic leukemia-1/2, a transcriptional co-activator of serum response factor, is required for skeletal myogenic differentiation

CHIP controls the sensitivity of transforming growth factor-beta signaling by modulating the basal level of Smad3 through ubiquitin-mediated degradation

Epithelial-mesenchymal transition and its implications for fibrosis

Megakaryoblastic leukemia factor-1 transduces cytoskeletal signals and induces smooth muscle cell differentiation from undifferentiated embryonic stem cells

Potentiation of serum response factor activity by a family of myocardin-related transcription factors.

Involvement of a human gene related to the Drosophila spen gene in the recurrent t(1;22) translocation of acute megakaryocytic leukemia

Myocardin is a master regulator of smooth muscle gene expression

Megakaryoblastic leukemia 1, a potent transcriptional coactivator for serum response factor (SRF), is required for serum induction of SRF target genes

Dual roles of myocardin-related transcription factors in epithelial mesenchymal transition via slug induction and actin remodeling

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F1116-28

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10.1152/AJPRENAL.00142.2007

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5

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294

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5513878

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18337547

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