A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation.
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Neuronal and Cardiovascular Potassium Channels as Therapeutic Drug Targets: Promise and PitfallsEmerging directions in the genetics of atrial fibrillationGain-of-function mutations in potassium channel subunit KCNE2 associated with early-onset lone atrial fibrillationNext-generation sequencing of nine atrial fibrillation candidate genes identified novel de novo mutations in patients with extreme trait of atrial fibrillation.Multi-walled carbon nanotubes impair Kv4.2/4.3 channel activities, delay membrane repolarization and induce bradyarrhythmias in the rat.Genetic determinants of P wave duration and PR segmentThe potential role of Kv4.3 K+ channel in heart hypertrophy.Atrial Fibrillation and SCN5A Variants.Expression changes of ionic channels in early phase of cultured rat atrial myocytes induced by rapid pacing.First de novo KCND3 mutation causes severe Kv4.3 channel dysfunction leading to early onset cerebellar ataxia, intellectual disability, oral apraxia and epilepsyYour Father and Grandfather's Atrial Fibrillation: A Review of the Genetics of the Most Common Pathologic Cardiac DysrhythmiaThe Role of Pharmacogenetics in Atrial Fibrillation Therapeutics: Is Personalized Therapy in Sight?Prevalence and Spectrum of TBX5 Mutation in Patients with Lone Atrial Fibrillation.Screening of the ito regulatory subunit klf15 in patients with early-onset lone atrial fibrillation.Auxiliary KChIP4a suppresses A-type K+ current through endoplasmic reticulum (ER) retention and promoting closed-state inactivation of Kv4 channelsAtrial fibrillation: the role of common and rare genetic variants.Atrial fibrillation: mechanisms, therapeutics, and future directions.Genomics of Atrial Fibrillation.Role of ATP-sensitive K+ channels in cardiac arrhythmias.Cardiac potassium channel subtypes: new roles in repolarization and arrhythmia.From iPSC towards cardiac tissue-a road under construction.Genetic Discoveries in Atrial Fibrillation and Implications for Clinical Practice.Atrial Fibrillation Genetics: Is There a Practical Clinical Value Now or in the Future?Identification of six new genetic loci associated with atrial fibrillation in the Japanese population.KCNE4 and KCNE5: K(+) channel regulation and cardiac arrhythmogenesis.Novel mechanism of transient outward potassium channel current regulation in the heart: implications for cardiac electrophysiology in health and disease.The L450F [Corrected] mutation in KCND3 brings spinocerebellar ataxia and Brugada syndrome closer together.Evolutionary analysis of voltage-gated potassium channels by Bayes method.Biological therapies targeting arrhythmias: are cells and genes the answer?A novel KCND3 mutation associated with early-onset lone atrial fibrillation.Strategies for Risk Analysis and Disease Classification in Atrial Fibrillation.
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P2860
A novel KCND3 gain-of-function mutation associated with early-onset of persistent lone atrial fibrillation.
description
2013 nî lūn-bûn
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name
A novel KCND3 gain-of-function ...... tent lone atrial fibrillation.
@en
A novel KCND3 gain-of-function ...... tent lone atrial fibrillation.
@nl
type
label
A novel KCND3 gain-of-function ...... tent lone atrial fibrillation.
@en
A novel KCND3 gain-of-function ...... tent lone atrial fibrillation.
@nl
prefLabel
A novel KCND3 gain-of-function ...... tent lone atrial fibrillation.
@en
A novel KCND3 gain-of-function ...... tent lone atrial fibrillation.
@nl
P2860
P50
P356
P1476
A novel KCND3 gain-of-function ...... tent lone atrial fibrillation.
@en
P2093
Anders Peter Larsen
Stig Haunsø
P2860
P304
P356
10.1093/CVR/CVT028
P50
P577
2013-02-11T00:00:00Z