Energy expenditure by intravenous administration of glucagon-like peptide-1 mediated by the lower brainstem and sympathoadrenal system.
about
The GLP-1 agonist, liraglutide, as a pharmacotherapy for obesityGLP-1 receptor stimulation depresses heart rate variability and inhibits neurotransmission to cardiac vagal neurons.Leptin and the systems neuroscience of meal size control.High-amylose resistant starch increases hormones and improves structure and function of the gastrointestinal tract: a microarray study.Gut hormones such as amylin and GLP-1 in the control of eating and energy expenditure.Long term exendin-4 treatment reduces food intake and body weight and alters expression of brain homeostatic and reward markers.Oxyntomodulin increases energy expenditure in addition to decreasing energy intake in overweight and obese humans: a randomised controlled trial.Emerging role of GLP-1 receptor agonists in the treatment of obesity.Spinally projecting preproglucagon axons preferentially innervate sympathetic preganglionic neuronsWeight loss effect of glucagon-like peptide-1 mimetics on obese/overweight adults without diabetes: A systematic review and meta-analysis of randomized controlled trials.Induction of Energy Expenditure by Sitagliptin Is Dependent on GLP-1 ReceptorMice Deficient in Proglucagon-Derived Peptides Exhibit Glucose Intolerance on a High-Fat Diet but Are Resistant to Obesity.Direct control of brown adipose tissue thermogenesis by central nervous system glucagon-like peptide-1 receptor signaling.Cardiovascular biology of the incretin system.Dietary-resistant starch improves maternal glycemic control in Goto-Kakizaki rat.Caudal brainstem processing is sufficient for behavioral, sympathetic, and parasympathetic responses driven by peripheral and hindbrain glucagon-like-peptide-1 receptor stimulation.Acute peripheral GLP-1 receptor agonism or antagonism does not alter energy expenditure in rats after Roux-en-Y gastric bypassCNS GLP-1 regulation of peripheral glucose homeostasis.Dietary resistant starch increases hypothalamic POMC expression in rats.The nucleus tractus solitarius: a portal for visceral afferent signal processing, energy status assessment and integration of their combined effects on food intake.Surgical weight loss: impact on energy expenditure.Substance P increases sympathetic activity during combined angiotensin-converting enzyme and dipeptidyl peptidase-4 inhibition.Role of the glucagon-like-peptide-1 receptor in the control of energy balance.Hypothalamic control of energy and glucose metabolism.Impact of leucine on energy balance.Peripheral signalling involved in energy homeostasis control.Glucagon-like peptide-1 receptor in the brain: role in neuroendocrine control of energy metabolism and treatment target for obesity.At the centennial of Michaelis and Menten, competing Michaelis-Menten steps explain effect of GLP-1 on blood-brain transfer and metabolism of glucose.GLP-1: benefits beyond pancreas.Transplantation of PC1/3-Expressing alpha-cells improves glucose handling and cold tolerance in leptin-resistant mice.Loss of dorsomedial hypothalamic GLP-1 signaling reduces BAT thermogenesis and increases adiposity.Oxyntomodulin analogue increases energy expenditure via the glucagon receptor.Intestinal bitter taste receptor activation alters hormone secretion and imparts metabolic benefits
P2860
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P2860
Energy expenditure by intravenous administration of glucagon-like peptide-1 mediated by the lower brainstem and sympathoadrenal system.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Energy expenditure by intraven ...... em and sympathoadrenal system.
@en
Energy expenditure by intraven ...... em and sympathoadrenal system.
@nl
type
label
Energy expenditure by intraven ...... em and sympathoadrenal system.
@en
Energy expenditure by intraven ...... em and sympathoadrenal system.
@nl
prefLabel
Energy expenditure by intraven ...... em and sympathoadrenal system.
@en
Energy expenditure by intraven ...... em and sympathoadrenal system.
@nl
P2093
P1433
P1476
Energy expenditure by intraven ...... em and sympathoadrenal system.
@en
P2093
Midori Yamakawa
Shuji Inoue
Toshimasa Osaka
P304
P356
10.1016/J.PEPTIDES.2005.02.016
P577
2005-03-17T00:00:00Z