The direct physiological effects of mitoK(ATP) opening on heart mitochondria.
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Mitochondrial Mechanisms in Septic CardiomyopathyPhysiological consequences of complex II inhibition for aging, disease, and the mKATP channelIntramitochondrial signaling: interactions among mitoKATP, PKCepsilon, ROS, and MPTCross talk between mitochondria and NADPH oxidasesMitochondrial reactive oxygen species production in excitable cells: modulators of mitochondrial and cell function.Mitochondrial matrix K+ flux independent of large-conductance Ca2+-activated K+ channel openingA novel mitochondrial K(ATP) channel assay.Mitochondrial depolarization underlies delay in permeability transition by preconditioning with isoflurane: roles of ROS and Ca2+.The mitochondrial bioenergetic phenotype for protection from cardiac ischemia in SUR2 mutant miceMitochondrial Ca2+-activated K+ channels more efficiently reduce mitochondrial Ca2+ overload in rat ventricular myocytes.The role of mitochondria in protection of the heart by preconditioning.Oxidative stress in atrial fibrillation: an emerging role of NADPH oxidase.Decreased brain K(ATP) channel contributes to exacerbating ischemic brain injury and the failure of neuroprotection by sevoflurane post-conditioning in diabetic rats.Respiratory uncoupling by increased H(+) or K(+) flux is beneficial for heart mitochondrial turnover of reactive oxygen species but not for permeability transitionEffect of Diazoxide Preconditioning on Cultured Rat Myocardium Microvascular Endothelial Cells against Apoptosis and Relation of PI3K/Akt Pathway.Enhanced charge-independent mitochondrial free Ca(2+) and attenuated ADP-induced NADH oxidation by isoflurane: Implications for cardioprotectionAge- and gender-related differences in ischemia/reperfusion injury and cardioprotection: effects of diazoxideProlonged Subcutaneous Administration of Oxytocin Accelerates Angiotensin II-Induced Hypertension and Renal Damage in Male Rats.The mitochondrial death pathway: a promising therapeutic target in diseasesNicorandil protects mesenchymal stem cells against hypoxia and serum deprivation-induced apoptosis.Isoflurane differentially modulates mitochondrial reactive oxygen species production via forward versus reverse electron transport flow: implications for preconditioningBKCa channel activation increases cardiac contractile recovery following hypothermic ischemia/reperfusion.Cardioprotection and mitochondrial S-nitrosation: effects of S-nitroso-2-mercaptopropionyl glycine (SNO-MPG) in cardiac ischemia-reperfusion injuryProtection against cardiac injury by small Ca(2+)-sensitive K(+) channels identified in guinea pig cardiac inner mitochondrial membraneRegulation of mitochondrial matrix volume.Diabetes, perioperative ischaemia and volatile anaesthetics: consequences of derangements in myocardial substrate metabolism.The mitochondrial K(ATP) channel--fact or fiction?S-nitrosylation of TRIM72 mends the broken heart: a molecular modifier-mediated cardioprotection.The endogenous mitochondrial complex II inhibitor malonate regulates mitochondrial ATP-sensitive potassium channels: implications for ischemic preconditioning.Inhibition of Succinate Dehydrogenase by Diazoxide Is Independent of the ATP-Sensitive Potassium Channel Subunit Sulfonylurea Type 1 Receptor.Conditioning the heart induces formation of signalosomes that interact with mitochondria to open mitoKATP channelsConnexin 43 impacts on mitochondrial potassium uptake.Molecular physiology of preconditioning-induced brain tolerance to ischemia.Opening of the mitoKATP channel and decoupling of mitochondrial complex II and III contribute to the suppression of myocardial reperfusion hyperoxygenation.Mitochondrial therapeutics for cardioprotection.Priming adult stem cells by hypoxic pretreatments for applications in regenerative medicine.Crosstalk signaling between mitochondrial Ca2+ and ROS.Mitochondrial reactive oxygen species: which ROS signals cardioprotection?Nox2 as a potential target of mitochondrial superoxide and its role in endothelial oxidative stressRelationship between mitochondrial matrix volume and cellular volume in response to stress and the role of ATP-sensitive potassium channel.
P2860
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P2860
The direct physiological effects of mitoK(ATP) opening on heart mitochondria.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
The direct physiological effects of mitoK
@nl
The direct physiological effects of mitoK(ATP) opening on heart mitochondria.
@en
type
label
The direct physiological effects of mitoK
@nl
The direct physiological effects of mitoK(ATP) opening on heart mitochondria.
@en
prefLabel
The direct physiological effects of mitoK
@nl
The direct physiological effects of mitoK(ATP) opening on heart mitochondria.
@en
P2093
P1476
The direct physiological effects of mitoK(ATP) opening on heart mitochondria
@en
P2093
Alexandre D T Costa
Anastasia Andrukhiv
Ian C West
Martin Jabůrek
P304
P356
10.1152/AJPHEART.00794.2005
P577
2005-09-02T00:00:00Z