Opening of the mitoKATP channel and decoupling of mitochondrial complex II and III contribute to the suppression of myocardial reperfusion hyperoxygenation.
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Mitochondrial reactive oxygen species: a double edged sword in ischemia/reperfusion vs preconditioningCardiovascular KATP channels and advanced agingKetones prevent oxidative impairment of hippocampal synaptic integrity through KATP channelsCardiac mitochondria and reactive oxygen species generationHypoxia. 4. Hypoxia and ion channel functionMultiplicity of effectors of the cardioprotective agent, diazoxide.Inhibitors of succinate: quinone reductase/Complex II regulate production of mitochondrial reactive oxygen species and protect normal cells from ischemic damage but induce specific cancer cell death.
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P2860
Opening of the mitoKATP channel and decoupling of mitochondrial complex II and III contribute to the suppression of myocardial reperfusion hyperoxygenation.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 23 October 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Opening of the mitoKATP channe ...... reperfusion hyperoxygenation.
@en
Opening of the mitoKATP channe ...... reperfusion hyperoxygenation.
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type
label
Opening of the mitoKATP channe ...... reperfusion hyperoxygenation.
@en
Opening of the mitoKATP channe ...... reperfusion hyperoxygenation.
@nl
prefLabel
Opening of the mitoKATP channe ...... reperfusion hyperoxygenation.
@en
Opening of the mitoKATP channe ...... reperfusion hyperoxygenation.
@nl
P2093
P2860
P1476
Opening of the mitoKATP channe ...... reperfusion hyperoxygenation.
@en
P2093
Chwen-Lih Chen
Guanglong He
Harold M Swartz
Xuehai Zhu
Yeong-Renn Chen
P2860
P2888
P356
10.1007/S11010-009-0283-2
P577
2009-10-23T00:00:00Z