The intestine as source of cytotoxic mediators in shock: free fatty acids and degradation of lipid-binding proteins.
about
Protease activity increases in plasma, peritoneal fluid, and vital organs after hemorrhagic shock in ratsTransmural intestinal wall permeability in severe ischemia after enteral protease inhibition.Role of intestinal lymphatics in interstitial volume regulation and transmucosal water transport.Disruption of the mucosal barrier during gut ischemia allows entry of digestive enzymes into the intestinal wall.Small β2-glycoprotein I peptides protect from intestinal ischemia reperfusion injury.Removal of luminal content protects the small intestine during hemorrhagic shock but is not sufficient to prevent lung injury.Sympathetic overstimulation during critical illness: adverse effects of adrenergic stress.The autodigestion hypothesis for shock and multi-organ failure.Modified Mediterranean Diet for Enrichment of Short Chain Fatty Acids: Potential Adjunctive Therapeutic to Target Immune and Metabolic Dysfunction in Schizophrenia?Autodigestion: Proteolytic Degradation and Multiple Organ Failure in ShockExpression profiles and circulation dynamics of rat mesenteric lymph microRNAsProgression of apoptic signaling from mesenteric ischemia-reperfusion injury to lungs: correlation in the level of ER chaperones expression.The autodigestion hypothesis: Proteolytic receptor cleavage in rheological and cardiovascular cell dysfunction1.Digested formula but not digested fresh human milk causes death of intestinal cells in vitro: implications for necrotizing enterocolitis.Effect of digestion and storage of human milk on free fatty acid concentration and cytotoxicity.Pancreatic digestive enzyme blockade in the intestine increases survival after experimental shock.Breast Milk Protects Against Gastrointestinal Symptoms in Infants at High Risk for Autism During Early Development.Severe intestinal ischemia can trigger cardiovascular collapse and sudden death via a parasympathetic mechanism.The Autodigestion Hypothesis in Shock and Multi-Organ Failure: Degrading Protease Activity.2008 Landis Award lecture. Inflammation and the autodigestion hypothesis.Biomechanical aspects of the auto-digestion theory.
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The intestine as source of cytotoxic mediators in shock: free fatty acids and degradation of lipid-binding proteins.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
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2008年學術文章
@zh-hant
name
The intestine as source of cyt ...... ion of lipid-binding proteins.
@en
The intestine as source of cyt ...... ion of lipid-binding proteins.
@nl
type
label
The intestine as source of cyt ...... ion of lipid-binding proteins.
@en
The intestine as source of cyt ...... ion of lipid-binding proteins.
@nl
prefLabel
The intestine as source of cyt ...... ion of lipid-binding proteins.
@en
The intestine as source of cyt ...... ion of lipid-binding proteins.
@nl
P2860
P1476
The intestine as source of cyt ...... ion of lipid-binding proteins.
@en
P2093
Alexander H Penn
Geert W Schmid-Schönbein
P2860
P304
P356
10.1152/AJPHEART.00902.2007
P577
2008-02-08T00:00:00Z