Trimetazidine inhibits mitochondrial permeability transition pore opening and prevents lethal ischemia-reperfusion injury.
about
The role of trimetazidine in cardiovascular disease: beyond an anti-anginal agentEffects of trimetazidine in patients with acute myocardial infarction: data from the Korean Acute Myocardial Infarction Registry.Protein Kinase Signaling at the Crossroads of Myocyte Life and Death in Ischemic Heart Disease.Trimetazidine, administered at the onset of reperfusion, ameliorates myocardial dysfunction and injury by activation of p38 mitogen-activated protein kinase and Akt signaling.Protective effect of an acute oral loading dose of trimetazidine on myocardial injury following percutaneous coronary interventionTrimetazidine does not alter metabolic substrate oxidation in cardiac mitochondria of target patient population.Mitochondria-mediated cardioprotection by trimetazidine in rabbit heart failure.Trimetazidine revisited: a comprehensive review of the pharmacological effects and analytical techniques for the determination of trimetazidine.Pharmacological strategies against cold ischemia reperfusion injury.Cardioprotective Effects of Oral Trimetazidine in Diabetic Patients With Anterior Wall Myocardial Infarction Treated with Thrombolysis.Postconditioning: from the bench to bedside.Mitochondrial dynamics in the heart as a novel therapeutic target for cardioprotection.Defining the role of trimetazidine in the treatment of cardiovascular disorders: some insights on its role in heart failure and peripheral artery disease.Trimetazidine protects against hypoxia-reperfusion-induced cardiomyocyte apoptosis by increasing microRNA-21 expression.In vitro dissolution and in vivo bioequivalence evaluation of two brands of trimetazidine tablets.Neuroprotective effects of Aceglutamide on motor function in a rat model of cerebral ischemia and reperfusion.Pharmacological preconditioning of mesenchymal stem cells with trimetazidine (1-[2,3,4-trimethoxybenzyl]piperazine) protects hypoxic cells against oxidative stress and enhances recovery of myocardial function in infarcted heart through Bcl-2 expressTrimetazidine demonstrated cardioprotective effects through mitochondrial pathway in a model of acute coronary ischemia.Exogenous hydrogen sulfide prevents cardiomyocyte apoptosis from cardiac hypertrophy induced by isoproterenol.Ex vivo administration of trimetazidine improves post-transplant lung function in pig model.Neuroprotective effect of hydroxy safflor yellow A against cerebral ischemia-reperfusion injury in rats: putative role of mPTP.Protective effects of ranolazine and propranolol, alone or combined, on the structural and functional alterations of cardiomyocyte mitochondria in a pig model of ischemia/reperfusion.Study of respiratory chain dysfunction in heart disease.
P2860
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P2860
Trimetazidine inhibits mitochondrial permeability transition pore opening and prevents lethal ischemia-reperfusion injury.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Trimetazidine inhibits mitocho ...... l ischemia-reperfusion injury.
@en
Trimetazidine inhibits mitocho ...... l ischemia-reperfusion injury.
@nl
type
label
Trimetazidine inhibits mitocho ...... l ischemia-reperfusion injury.
@en
Trimetazidine inhibits mitocho ...... l ischemia-reperfusion injury.
@nl
prefLabel
Trimetazidine inhibits mitocho ...... l ischemia-reperfusion injury.
@en
Trimetazidine inhibits mitocho ...... l ischemia-reperfusion injury.
@nl
P2093
P1476
Trimetazidine inhibits mitocho ...... l ischemia-reperfusion injury.
@en
P2093
Dominique Robert
Elisabeth Couture-Lepetit
Joseph Loufouat
Laurent Argaud
Ludovic Gomez
Michel Ovize
Odile Gateau-Roesch
P304
P356
10.1016/J.YJMCC.2005.09.012
P577
2005-10-21T00:00:00Z