Trimetazidine, administered at the onset of reperfusion, ameliorates myocardial dysfunction and injury by activation of p38 mitogen-activated protein kinase and Akt signaling. - Wikidata - awgldk - TriplyDB

Trimetazidine, administered at the onset of reperfusion, ameliorates myocardial dysfunction and injury by activation of p38 mitogen-activated protein kinase and Akt signaling.

Trimetazidine, administered at the onset of reperfusion, ameliorates myocardial dysfunction and injury by activation of p38 mitogen-activated protein kinase and Akt signaling.

http://www.wikidata.org/entity/Q33861991

about

Autophagy, a process within reperfusion injury: an update Effects of trimetazidine in patients with acute myocardial infarction: data from the Korean Acute Myocardial Infarction Registry.Central Beneficial Effects of Trimetazidine on Psychomotor Performance in Normal Healthy Volunteers.Trimetazidine protects against smoking-induced left ventricular remodeling via attenuating oxidative stress, apoptosis, and inflammation Atorvastatin ameliorates myocardial ischemia/reperfusion injury through attenuation of endoplasmic reticulum stress-induced apoptosis.Arterial levels of oxygen stimulate intimal hyperplasia in human saphenous veins via a ROS-dependent mechanism.Carvedilol enhances mesenchymal stem cell therapy for myocardial infarction via inhibition of caspase-3 expression.Mitochondria as a drug target in ischemic heart disease and cardiomyopathy Sivelestat attenuates myocardial reperfusion injury during brief low flow postischemic infusion.The protective effect of trimetazidine on myocardial ischemia/reperfusion injury through activating AMPK and ERK signaling pathway.Trimetazidine protects against cardiac ischemia/reperfusion injury via effects on cardiac miRNA‑21 expression, Akt and the Bcl‑2/Bax pathway.The effects of trimetazidine on lipopolysaccharide-induced oxidative stress in mice.Oxygen and oxygenation in stem-cell therapy for myocardial infarction c-Kit-positive cardiac stem cells nested in hypoxic niches are activated by stem cell factor reversing the aging myopathy.Pharmacological Agents Targeting Myocardial Metabolism for the Management of Chronic Stable Angina : an Update.Oxygen cycling in conjunction with stem cell transplantation induces NOS3 expression leading to attenuation of fibrosis and improved cardiac function A molecular paramagnetic spin-doped biopolymeric oxygen sensor.Attenuation of endoplasmic reticulum stress and mitochondrial injury in kidney with ischemic postconditioning application and trimetazidine treatment.How to protect liver graft with nitric oxide.Effects of trimetazidine on the Akt/eNOS signaling pathway and oxidative stress in an in vivo rat model of renal ischemia-reperfusion.The protective effect of trimetazidine against cisplatin-induced nephrotoxicity in rats.Metabolic Modulation of Cardiac Metabolism in Heart Failure

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Trimetazidine, administered at the onset of reperfusion, ameliorates myocardial dysfunction and injury by activation of p38 mitogen-activated protein kinase and Akt signaling.

http://www.wikidata.org/entity/Q33861991

description

2010 nî lūn-bûn

@nan

2010 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Trimetazidine, administered at ...... tein kinase and Akt signaling.

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Trimetazidine, administered at ...... tein kinase and Akt signaling.

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JPET.109.165175

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Journal of Pharmacology and Experimental Therapeutics

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Trimetazidine, administered at ...... tein kinase and Akt signaling.

@en

P2093

Kàlmàn Hideg

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Mahmood Khan

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Mahmoud Mostafa

http://www.w3.org/2001/XMLSchema#string

Periannan Kuppusamy

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Saniya Khan

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Sarath Meduru

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P2860

Trimetazidine. A review of its use in stable angina pectoris and other coronary conditions

The antianginal drug trimetazidine shifts cardiac energy metabolism from fatty acid oxidation to glucose oxidation by inhibiting mitochondrial long-chain 3-ketoacyl coenzyme A thiolase

Molecular and cellular mechanisms of myocardial stunning.

Sulfaphenazole protects heart against ischemia-reperfusion injury and cardiac dysfunction by overexpression of iNOS, leading to enhancement of nitric oxide bioavailability and tissue oxygenation.

Inhibition of p38 mitogen-activated protein kinase decreases cardiomyocyte apoptosis and improves cardiac function after myocardial ischemia and reperfusion.

Reperfusion injury induces apoptosis in rabbit cardiomyocytes.

Demonstration of free radical generation in "stunned" myocardium of intact dogs with the use of the spin trap alpha-phenyl N-tert-butyl nitrone

Oxidative stress and cardiovascular injury: Part I: basic mechanisms and in vivo monitoring of ROS.

Protein kinase activation and myocardial ischemia/reperfusion injury.

EF24 induces G2/M arrest and apoptosis in cisplatin-resistant human ovarian cancer cells by increasing PTEN expression

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421-429

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10.1124/JPET.109.165175

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41486478

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20167841

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2872960

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