c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
about
Elevation of c-FLIP in castrate-resistant prostate cancer antagonizes therapeutic response to androgen receptor-targeted therapyc-FLIP, a master anti-apoptotic regulatorHyperthermia enhances mapatumumab-induced apoptotic death through ubiquitin-mediated degradation of cellular FLIP(long) in human colon cancer cellsCalbindin 2 (CALB2) regulates 5-fluorouracil sensitivity in colorectal cancer by modulating the intrinsic apoptotic pathwayNanoencapsulation of ABT-737 and camptothecin enhances their clinical potential through synergistic antitumor effects and reduction of systemic toxicity.Expression profile and specific network features of the apoptotic machinery explain relapse of acute myeloid leukemia after chemotherapy.Early growth response-1 is a regulator of DR5-induced apoptosis in colon cancer cells.The eIF4E/eIF4G interaction inhibitor 4EGI-1 augments TRAIL-mediated apoptosis through c-FLIP Down-regulation and DR5 induction independent of inhibition of cap-dependent protein translationDissecting the molecular pathways of (testicular) germ cell tumour pathogenesis; from initiation to treatment-resistance.Prognostic significance of TRAIL signaling molecules in stage II and III colorectal cancer.Molecular mechanisms of tamoxifen therapy for cholangiocarcinoma: role of calmodulin.Chemosensitization of tumors by resveratrolDrug-induced caspase 8 upregulation sensitises cisplatin-resistant ovarian carcinoma cells to rhTRAIL-induced apoptosis.Evidence for two modes of synergistic induction of apoptosis by mapatumumab and oxaliplatin in combination with hyperthermia in human colon cancer cells.Chemotherapy overcomes TRAIL-R4-mediated TRAIL resistance at the DISC level4-(4-Chloro-2-methylphenoxy)-N-hydroxybutanamide (CMH) targets mRNA of the c-FLIP variants and induces apoptosis in MCF-7 human breast cancer cells.Decreased mtDNA, oxidative stress, cardiomyopathy, and death from transgenic cardiac targeted human mutant polymerase gamma.Targeting the Anti-Apoptotic Protein c-FLIP for Cancer Therapy.Regulation of chemoresistance via alternative messenger RNA splicingIdentification and Characterization of the Interaction Site between cFLIPL and CalmodulinThe relative contribution of pro-apoptotic p53-target genes in the triggering of apoptosis following DNA damage in vitro and in vivo.Transcriptional network governing the angiogenic switch in human pancreatic cancerCellular FLICE-like inhibitory protein (C-FLIP): a novel target for cancer therapy.Identification of an acetylation-dependant Ku70/FLIP complex that regulates FLIP expression and HDAC inhibitor-induced apoptosisHow many diseases are colorectal cancer?Mitochondrial inhibitor sensitizes non-small-cell lung carcinoma cells to TRAIL-induced apoptosis by reactive oxygen species and Bcl-X(L)/p53-mediated amplification mechanisms.Identification of galanin and its receptor GalR1 as novel determinants of resistance to chemotherapy and potential biomarkers in colorectal cancerCD44v3+/CD24- cells possess cancer stem cell-like properties in human oral squamous cell carcinomaDelivery of intracellular-acting biologics in pro-apoptotic therapies.c-FLIP knockdown induces ligand-independent DR5-, FADD-, caspase-8-, and caspase-9-dependent apoptosis in breast cancer cells.The effects of antioxidants on gene expression following gamma-radiation (GR) and proton radiation (PR) in mice in vivo.Expression and biological significance of c-FLIP in human hepatocellular carcinomasInhibition of c-FLIPL expression by miRNA-708 increases the sensitivity of renal cancer cells to anti-cancer drugs.The natural product honokiol preferentially inhibits cellular FLICE-inhibitory protein and augments death receptor-induced apoptosis.Expression of c-FLIP in pulmonary metastases in osteosarcoma patients and human xenografts.Fibroblast growth factor receptor 4 (FGFR4): a targetable regulator of drug resistance in colorectal cancer.Chemoprevention, chemotherapy, and chemoresistance in colorectal cancer.Celecoxib promotes c-FLIP degradation through Akt-independent inhibition of GSK3.Differences of immune disorders between Alzheimer's disease and breast cancer based on transcriptional regulation.Autocrine and Paracrine Mechanisms Promoting Chemoresistance in Cholangiocarcinoma.
P2860
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P2860
c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh
2006年學術文章
@zh-hant
name
c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
@en
c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
@nl
type
label
c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
@en
c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
@nl
prefLabel
c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
@en
c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
@nl
P2093
P356
P1433
P1476
c-FLIP inhibits chemotherapy-induced colorectal cancer cell death.
@en
P2093
D B Longley
L Galligan
P G Johnston
T R Wilson
P2888
P304
P356
10.1038/SJ.ONC.1209122
P407
P577
2006-02-01T00:00:00Z
P5875
P6179
1013055536