Leukocyte engagement of platelet glycoprotein Ibalpha via the integrin Mac-1 is critical for the biological response to vascular injury. - Wikidata - awgldk - TriplyDB

Leukocyte engagement of platelet glycoprotein Ibalpha via the integrin Mac-1 is critical for the biological response to vascular injury.

Leukocyte engagement of platelet glycoprotein Ibalpha via the integrin Mac-1 is critical for the biological response to vascular injury.

http://www.wikidata.org/entity/Q46783981

about

Myeloid-related protein-8/14 is critical for the biological response to vascular injury Inflammation and vascular injury: basic discovery to drug development Platelets Guide Leukocytes to Their Sites of Extravasation Juxtamembrane basic residues in glycoprotein Ibbeta cytoplasmic domain are required for assembly and surface expression of glycoprotein Ib-IX complex.Platelet-derived S100 family member myeloid-related protein-14 regulates thrombosis.CD40 ligand promotes Mac-1 expression, leukocyte recruitment, and neointima formation after vascular injury.The role of platelet adhesion receptor GPIbalpha far exceeds that of its main ligand, von Willebrand factor, in arterial thrombosis.Cleavage of the CD11b extracellular domain by the leukocyte serprocidins is critical for neutrophil detachment during chemotaxis.Neutrophil AKT2 regulates heterotypic cell-cell interactions during vascular inflammation Small molecule agonists of integrin CD11b/CD18 do not induce global conformational changes and are significantly better than activating antibodies in reducing vascular injury.Genetic deletion of platelet glycoprotein Ib alpha but not its extracellular domain protects from atherosclerosis.Small molecule-mediated activation of the integrin CD11b/CD18 reduces inflammatory disease.Directed transport of neutrophil-derived extracellular vesicles enables platelet-mediated innate immune response Mac-1 (CD11b/CD18) links inflammation and thrombosis after glomerular injury.Platelet depletion and aspirin treatment protect mice in a two-event model of transfusion-related acute lung injury.Role for platelet glycoprotein Ib-IX and effects of its inhibition in endotoxemia-induced thrombosis, thrombocytopenia, and mortality.Developmental endothelial locus-1 attenuates complement-dependent phagocytosis through inhibition of Mac-1-integrin.Myeloid-related protein-14 regulates deep vein thrombosis Leukocyte integrin Mac-1 regulates thrombosis via interaction with platelet GPIbα.Leukocyte-endothelial interactions in inflammation.The intrinsic complement regulator decay-accelerating factor modulates the biological response to vascular injury Thrombospondin-4 regulates vascular inflammation and atherogenesis.Critical role for Syk in responses to vascular injury.Lymphocyte function-associated antigen-1-dependent inhibition of corneal wound healing.Src family kinases mediate neutrophil adhesion to adherent platelets.Platelet-neutrophil interactions under thromboinflammatory conditions Platelet glycoprotein Ib alpha supports experimental lung metastasis In vivo targeting of inflammation-associated myeloid-related protein 8/14 via gadolinium immunonanoparticles.Platelets contribute to the pathogenesis of experimental autoimmune encephalomyelitis.Novel uses for anti-platelet agents as anti-inflammatory drugs.Circulating inflammatory cells are associated with vein graft stenosis Integrins α4 and αM, collagen1A1, and matrix metalloproteinase 7 are upregulated in acute Kawasaki disease vasculopathy.Expression, activation, and function of integrin alphaMbeta2 (Mac-1) on neutrophil-derived microparticles Properdin-Mediated C5a Production Enhances Stable Binding of Platelets to Granulocytes in Human Whole Blood.Platelet IκB kinase-β deficiency increases mouse arterial neointima formation via delayed glycoprotein Ibα shedding.Periodontal therapy alters gene expression of peripheral blood monocytes.Inhibition of platelet activation by clopidogrel prevents hypertension-induced cardiac inflammation and fibrosis.Platelets and Their Interactions with Other Immune Cells Enhanced biocompatibility of CD47-functionalized vascular stents.Platelet glycoprotein Ib-IX as a regulator of systemic inflammation.

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Leukocyte engagement of platelet glycoprotein Ibalpha via the integrin Mac-1 is critical for the biological response to vascular injury.

http://www.wikidata.org/entity/Q46783981

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年学术文章

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2005年學術文章

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2005年學術文章

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name

Leukocyte engagement of platel ...... l response to vascular injury.

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Leukocyte engagement of platel ...... l response to vascular injury.

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type

label

Leukocyte engagement of platel ...... l response to vascular injury.

@en

Leukocyte engagement of platel ...... l response to vascular injury.

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Leukocyte engagement of platel ...... l response to vascular injury.

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Leukocyte engagement of platel ...... l response to vascular injury.

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Leukocyte engagement of platel ...... l response to vascular injury.

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Alexandre C Zago

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Can Shi

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Daniel I Simon

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Edward Plow

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Jose Lopez

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Yunmei Wang

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