15d-prostaglandin J2 protects brain from ischemia-reperfusion injury. - Wikidata - awgldk - TriplyDB

15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

http://www.wikidata.org/entity/Q46872642

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Mechanisms and potential therapeutic applications of microglial activation after brain injury PGJ(2) provides prolonged CNS stroke protection by reducing white matter edema Mitochondrial dynamics associated with oxygen-glucose deprivation in rat primary neuronal cultures Ligand-activated peroxisome proliferator-activated receptor-gamma protects against ischemic cerebral infarction and neuronal apoptosis by 14-3-3 epsilon upregulation.Peroxisome Proliferator-Activated Receptors Protect against Apoptosis via 14-3-3.Extension of the neuroprotective time window for thiazolidinediones in ischemic stroke is dependent on time of reperfusion.Peroxisome proliferator-activated receptor-γ agonist 15d-prostaglandin J2 mediates neuronal autophagy after cerebral ischemia-reperfusion injury.Bardoxolone methyl (BARD) ameliorates ischemic AKI and increases expression of protective genes Nrf2, PPARγ, and HO-1.12/15-Lipoxygenase metabolites of arachidonic acid activate PPARγ: a possible neuroprotective effect in ischemic brain Hyperbaric oxygen preconditioning attenuates hemorrhagic transformation through increasing PPARγ in hyperglycemic MCAO rats.Sustained neuroprotection from a single intravitreal injection of PGJ₂ in a nonhuman primate model of nonarteritic anterior ischemic optic neuropathy.Treatment targets in intracerebral hemorrhage.Activation of peroxisome proliferator-activated receptor gamma contributes to the survival of T lymphoma cells by affecting cellular metabolism.New insights into the role of peroxisome proliferator-activated receptors in regulating the inflammatory response after tissue injury.Reversal of the Caspase-Dependent Apoptotic Cytotoxicity Pathway by Taurine from Lycium barbarum (Goji Berry) in Human Retinal Pigment Epithelial Cells: Potential Benefit in Diabetic Retinopathy.Emerging PPARγ-Independent Role of PPARγ Ligands in Lung Diseases.Role of transcription factors in mediating post-ischemic cerebral inflammation and brain damage.Peroxisome proliferator-activated receptors γ/mitochondrial uncoupling protein 2 signaling protects against seizure-induced neuronal cell death in the hippocampus following experimental status epilepticus.Ischemic preconditioning via epsilon protein kinase C activation requires cyclooxygenase-2 activation in vitro Prevention of JNK phosphorylation as a mechanism for rosiglitazone in neuroprotection after transient cerebral ischemia: activation of dual specificity phosphatase.Smurf2-mediated degradation of EZH2 enhances neuron differentiation and improves functional recovery after ischaemic stroke The arachidonic acid metabolite 11β-ProstaglandinF2α controls intestinal epithelial healing: deficiency in patients with Crohn's disease.Increased generation of cyclopentenone prostaglandins after brain ischemia and their role in aggregation of ubiquitinated proteins in neurons Mechanisms of anti-inflammatory and neuroprotective actions of PPAR-gamma agonists.Rosiglitazone attenuates the severity of hyperlipidemic severe acute pancreatitis in rats.Transient receptor potential A1 is a sensory receptor for multiple products of oxidative stress.Sustained neuroprotection from a single intravitreal injection of PGJ2 in a rodent model of anterior ischemic optic neuropathy PPARs as new therapeutic targets for the treatment of cerebral ischemia/reperfusion injury.Albumin-binding and tumor vasculature determine the antitumor effect of 15-deoxy-Delta-(12,14)-prostaglandin-J(2) in vivo Autophagy Modulation in Disease Therapy: Where Do We Stand?Therapeutic Potential of PPARγ Activation in Stroke.Therapeutic implications of disorders of cell death signalling: membranes, micro-environment, and eicosanoid and docosanoid metabolism Peroxisome proliferator-activated receptor gamma (PPAR-γ) and neurodegenerative disorders.Physiological and Pathological Roles of 15-Deoxy-Δ12,14-Prostaglandin J2 in the Central Nervous System and Neurological Diseases.The omega-6 fatty acid derivative 15-deoxy-Δ¹²,¹⁴-prostaglandin J2 is involved in neuroprotection by enteric glial cells against oxidative stress.Thiazolidinediones protect mouse pancreatic β-cells directly from cytokine-induced cytotoxicity through PPARγ-dependent mechanisms.15-Deoxy-Δ(12,14)-prostaglandin J2 Induces Apoptosis and Upregulates SOCS3 in Human Thyroid Cancer Cells.Modification of ubiquitin-C-terminal hydrolase-L1 by cyclopentenone prostaglandins exacerbates hypoxic injury.Clinacanthus nutans Mitigates Neuronal Apoptosis and Ischemic Brain Damage Through Augmenting the C/EBPβ-Driven PPAR-γ Transcription.15d-PGJ2 induces apoptosis of mouse oligodendrocyte precursor cells.

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15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

http://www.wikidata.org/entity/Q46872642

description

2005 nî lūn-bûn

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2005年の論文

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2005年学术文章

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2005年学术文章

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2005年学术文章

@zh-cn

2005年学术文章

@zh-hans

2005年学术文章

@zh-my

2005年学术文章

@zh-sg

2005年學術文章

@yue

2005年學術文章

@zh-hant

name

15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

@en

15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

@nl

type

label

15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

@en

15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

@nl

prefLabel

15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

@en

15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

@nl

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01.ATV.0000201933.53964.5B

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Arteriosclerosis, Thrombosis, and Vascular Biology

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15d-prostaglandin J2 protects brain from ischemia-reperfusion injury.

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Heng Lin

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Jean-Ju Chen

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Jin-Jer Chen

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Jui-Sheng Wu

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Jun-Yang Liou

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Kenneth K Wu

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Song-Kun Shyue

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Teng-Nan Lin

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Wai-Mui Cheung

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481-487

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scholarly article

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10.1161/01.ATV.0000201933.53964.5B

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3

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26

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2005-12-29T00:00:00Z

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16385084

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reperfusion injury