Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
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Angiotensin-(1-7): new perspectives in atherosclerosis treatmentVascular stiffness in insulin resistance and obesityReview article: pancreatic renin-angiotensin systems in health and diseaseObesity associated hypertension: new insights into mechanismDivergent mechanism regulating fluid intake and metabolism by the brain renin-angiotensin systemModulation of glucose metabolism by the renin-angiotensin-aldosterone systemAnti-Inflammatory Effects of Ang-(1-7) in Ameliorating HFD-Induced Renal Injury through LDLr-SREBP2-SCAP PathwayPhysiology of kidney reninLong-term systemic angiotensin II type 1 receptor blockade regulates mRNA expression of dorsomedial medulla renin-angiotensin system componentsThe metabolic syndromeThe renin angiotensin system and the metabolic syndromeACE2 and diabetes: ACE of ACEs?The brain Renin-angiotensin system controls divergent efferent mechanisms to regulate fluid and energy balance.Renin-angiotensin system blockers protect pancreatic islets against diet-induced obesity and insulin resistance in miceChronic treatment with angiotensin-(1-7) improves renal endothelial dysfunction in apolipoproteinE-deficient miceReduced levels of two modifiers of epigenetic gene silencing, Dnmt3a and Trim28, cause increased phenotypic noise.Atypical signaling and functional desensitization response of MAS receptor to peptide ligands.The combination of high-fat diet-induced obesity and chronic ulcerative colitis reciprocally exacerbates adipose tissue and colon inflammationAngiotensin I-converting enzyme type 2 (ACE2) gene therapy improves glycemic control in diabetic mice.Deficiency of ACE2 in Bone-Marrow-Derived Cells Increases Expression of TNF-α in Adipose Stromal Cells and Augments Glucose Intolerance in Obese C57BL/6 MiceMAS C-Terminal Tail Interacting Proteins Identified by Mass Spectrometry- Based Proteomic Approach.The link between the renin-angiotensin-aldosterone system and renal injury in obesity and the metabolic syndrome.Polymorphism in miR-31 and miR-584 binding site in the angiotensinogen gene differentially influences body fat distribution in both sexes.Angiotensin converting enzyme 2: a new important player in the regulation of glycemia.International Union of Basic and Clinical Pharmacology. XCIX. Angiotensin Receptors: Interpreters of Pathophysiological Angiotensinergic Stimuli [corrected].Physical activity prevents alterations in mitochondrial ultrastructure and glucometabolic parameters in a high-sugar diet model.Effect of Angiotensin(1-7) on Heart Function in an Experimental Rat Model of ObesityOpposing tissue-specific roles of angiotensin in the pathogenesis of obesity, and implications for obesity-related hypertensionLoss of ACE2 exaggerates high-calorie diet-induced insulin resistance by reduction of GLUT4 in mice.The renin angiotensin aldosterone system and insulin resistance in humans.Angiotensin-converting enzyme 2/angiotensin-(1-7)/Mas axis activates Akt signaling to ameliorate hepatic steatosisMetabolic rate regulation by the renin-angiotensin system: brain vs. body.Mice lacking angiotensin-converting enzyme have increased energy expenditure, with reduced fat mass and improved glucose clearanceAngiotensin 1-7 as means to prevent the metabolic syndrome: lessons from the fructose-fed rat model.Chronic Angiotensin-(1-7) Improves Insulin Sensitivity in High-Fat Fed Mice Independent of Blood Pressure.ACE2 is expressed in mouse adipocytes and regulated by a high-fat diet.Angiotensin (1-7)/Mas receptor axis activation ameliorates the changes in fatty acid composition in diabetic rats with nephropathy.Control of adipogenesis by the autocrine interplays between angiotensin 1-7/Mas receptor and angiotensin II/AT1 receptor signaling pathwaysUpregulation of the angiotensin-converting enzyme 2/angiotensin-(1-7)/Mas receptor axis in the heart and the kidney of growth hormone receptor knock-out mice.Angiotensin-converting enzyme 2 and the kidney.
P2860
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P2860
Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
@en
Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
@nl
type
label
Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
@en
Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
@nl
prefLabel
Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
@en
Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
@nl
P2093
P356
P1433
P1476
Mas deficiency in FVB/N mice produces marked changes in lipid and glycemic metabolism.
@en
P2093
Adaliene Versiani M Ferreira
Erica Guilhen Mario
Jaqueline Isaura Alvarez-Leite
Laura Cristina J Pôrto
Luciana Rodrigues Fernandes
Natalia Alenina
Robson Augusto S Santos
Sérgio Henrique S Santos
P304
P356
10.2337/DB07-0953
P407
P577
2007-11-19T00:00:00Z