Neuronal activity determines the protein synthesis dependence of long-term potentiation. - Wikidata - awgldk - TriplyDB

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

http://www.wikidata.org/entity/Q46987191

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Neural Protein Synthesis during Aging: Effects on Plasticity and Memory The roles of protein expression in synaptic plasticity and memory consolidation Ubiquitin ligase TRIM3 controls hippocampal plasticity and learning by regulating synaptic γ-actin levels A PARP1-ERK2 synergism is required for the induction of LTP Failed stabilization for long-term potentiation in the auditory cortex of FMR1 knockout mice Loss of GluN2B-containing NMDA receptors in CA1 hippocampus and cortex impairs long-term depression, reduces dendritic spine density, and disrupts learning.Improved preparation and preservation of hippocampal mouse slices for a very stable and reproducible recording of long-term potentiation.Protein synthesis inhibition and memory: formation vs amnesia Tag-trigger-consolidation: a model of early and late long-term-potentiation and depression State based model of long-term potentiation and synaptic tagging and capture Post-training dephosphorylation of eEF-2 promotes protein synthesis for memory consolidation.Rapid and long-lasting increase in sites for synapse assembly during late-phase potentiation in rat hippocampal neurons.The sensitivity of memory consolidation and reconsolidation to inhibitors of protein synthesis and kinases: computational analysis Global analysis of dynamical decision-making models through local computation around the hidden saddle.An activity-regulated microRNA, miR-188, controls dendritic plasticity and synaptic transmission by downregulating neuropilin-2.Pin1 and PKMzeta sequentially control dendritic protein synthesis.Long-lasting LTP requires neither repeated trains for its induction nor protein synthesis for its development A spinal analog of memory reconsolidation enables reversal of hyperalgesia.The protein kinase Mζ network as a bistable switch to store neuronal memory.Postinduction requirement of NMDA receptor activation for late-phase long-term potentiation of developing retinotectal synapses in vivo.Reconsolidation of long-term memory in Aplysia.Estrogen promotes learning-related plasticity by modifying the synaptic cytoskeleton.Role of Liver X Receptor in AD Pathophysiology.Protein Synthesis Inhibitors Did Not Interfere with Long-Term Depression Induced either Electrically in Juvenile Rats or Chemically in Middle-Aged Rats.Dbo/Henji Modulates Synaptic dPAK to Gate Glutamate Receptor Abundance and Postsynaptic Response.Proteasome inhibition enhances the induction and impairs the maintenance of late-phase long-term potentiation.Frequency-specific stimulations induce reconsolidation of long-term potentiation in freely moving rats.Local translation and directional steering in axons Neuroplasticity mediated by altered gene expression.Mechanism and time course of cocaine-induced long-term potentiation in the ventral tegmental area Intrahippocampal infusions of anisomycin produce amnesia: contribution of increased release of norepinephrine, dopamine, and acetylcholine.The interplay between neuronal activity and actin dynamics mimic the setting of an LTD synaptic tag Asymmetrical synaptic cooperation between cortical and thalamic inputs to the amygdale.Mapping Gene Expression in Excitatory Neurons during Hippocampal Late-Phase Long-Term Potentiation.Modulation of fear memory by retrieval and extinction: a clue for memory deconsolidation.Biological pathways to adaptability--interactions between genome, epigenome, nervous system and environment for adaptive behavior.Memory disrupting effects of nonmuscle myosin II inhibition depend on the class of abused drug and brain region.Temporal phases of long-term potentiation (LTP): myth or fact?Reconsolidation and the Dynamic Nature of Memory.The Kinase Function of MSK1 Regulates BDNF Signaling to CREB and Basal Synaptic Transmission, But Is Not Required for Hippocampal Long-Term Potentiation or Spatial Memory.

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Neuronal activity determines the protein synthesis dependence of long-term potentiation.

http://www.wikidata.org/entity/Q46987191

description

2006 nî lūn-bûn

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2006年の論文

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年学术文章

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2006年學術文章

@yue

2006年學術文章

@zh-hant

name

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

@en

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

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type

label

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

@en

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

@nl

prefLabel

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

@en

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

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Nature Neuroscience

P1476

Neuronal activity determines the protein synthesis dependence of long-term potentiation.

@en

P2860

Fear memories require protein synthesis in the amygdala for reconsolidation after retrieval

Activity level controls postsynaptic composition and signaling via the ubiquitin-proteasome system

Long-lasting forms of synaptic potentiation in the mammalian hippocampus.

Anisomycin, an inhibitor of protein synthesis, blocks late phases of LTP phenomena in the hippocampal CA1 region in vitro.

N-methyl-D-aspartate receptor signaling results in Aurora kinase-catalyzed CPEB phosphorylation and alpha CaMKII mRNA polyadenylation at synapses.

Memory reconsolidation and extinction have distinct temporal and biochemical signatures.

Reconsolidation of memory after its reactivation.

Retrograde amnesia produced by electroconvulsive shock after reactivation of a consolidated memory trace.

NMDA receptor antagonists sustain LTP and spatial memory: active processes mediate LTP decay.

Expression of constitutively active CREB protein facilitates the late phase of long-term potentiation by enhancing synaptic capture.

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478-480

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scholarly article

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10.1038/NN1667

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4

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9

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Tobias Bonhoeffer

Valentin Nägerl

Rosalina Fonseca

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2006-03-12T00:00:00Z

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16531998

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