Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

about

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

Item

http://www.wikidata.org/entity/Q47110397

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

@en

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

@nl

type

Item

label

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

@en

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

@nl

prefLabel

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

@en

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

@nl

P1476

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.

@en

P2093

Abigail E Woolums

http://www.w3.org/2001/XMLSchema#string

Donna M Wilcock

http://www.w3.org/2001/XMLSchema#string

Erica M Weekman

http://www.w3.org/2001/XMLSchema#string

Tiffany L Sudduth

http://www.w3.org/2001/XMLSchema#string

P2860

Effect of vitamins and aspirin on markers of platelet activation, oxidative stress and homocysteine in people at high risk of dementia

Folate dependence of hyperhomocysteinemia and vascular dysfunction in cystathionine beta-synthase-deficient mice

Induction of hyperhomocysteinemia models vascular dementia by induction of cerebral microhemorrhages and neuroinflammation.

Mice deficient in methylenetetrahydrofolate reductase exhibit hyperhomocysteinemia and decreased methylation capacity, with neuropathology and aortic lipid deposition.

Inflammation and white matter damage in vascular cognitive impairment.

Homocyst(e)ine and cardiovascular disease: a critical review of the epidemiologic evidence.

β-amyloid deposition is shifted to the vasculature and memory impairment is exacerbated when hyperhomocysteinemia is induced in APP/PS1 transgenic mice.

Homocysteine, silent brain infarcts, and white matter lesions: The Rotterdam Scan Study.

Lithium treatment of APPSwDI/NOS2-/- mice leads to reduced hyperphosphorylated tau, increased amyloid deposition and altered inflammatory phenotype.

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

P304

1759091417742296

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1177/1759091417742296

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2017-11-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29198136

http://www.w3.org/2001/XMLSchema#string

P932

5718317

http://www.w3.org/2001/XMLSchema#string