LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice - Wikidata - awgldk - TriplyDB

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

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Targeting innate immunity for neurodegenerative disorders of the central nervous system Fundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease in focal cerebral ischemic rats Fundamental role of pan-inflammation and oxidative-nitrosative pathways in neuropathogenesis of Alzheimer's disease Individuality, phenotypic differentiation, dormancy and 'persistence' in culturable bacterial systems: commonalities shared by environmental, laboratory, and clinical microbiology Curcumin and Apigenin - novel and promising therapeutics against chronic neuroinflammation in Alzheimer's disease Non-steroidal anti-inflammatory drugs and cognitive function: are prostaglandins at the heart of cognitive impairment in dementia and delirium?Who fans the flames of Alzheimer's disease brains? Misfolded tau on the crossroad of neurodegenerative and inflammatory pathways Neuroinflammation in Alzheimer's disease; A source of heterogeneity and target for personalized therapy A Bacterial Component to Alzheimer’s-Type Dementia Seen via a Systems Biology Approach that Links Iron Dysregulation and Inflammagen Shedding to Disease Ameliorative Effects of Antioxidants on the Hippocampal Accumulation of Pathologic Tau in a Rat Model of Blast-Induced Traumatic Brain Injury Histone deacetylase 6 inhibition improves memory and reduces total tau levels in a mouse model of tau deposition.Ketogenic diet improves motor performance but not cognition in two mouse models of Alzheimer's pathology R-flurbiprofen improves tau, but not Aß pathology in a triple transgenic model of Alzheimer's disease.Glial contributions to neurodegeneration in tauopathies.Sevoflurane induces tau phosphorylation and glycogen synthase kinase 3β activation in young mice Epitope analysis following active immunization with tau proteins reveals immunogens implicated in tau pathogenesis.Amyloid β and Tau Alzheimer's disease related pathology is reduced by Toll-like receptor 9 stimulation.The role of tau protein in HIV-associated neurocognitive disorders Lipolysaccharide-Induced Neuroinflammation Is Associated with Alzheimer-Like Amyloidogenic Axonal Pathology and Dendritic Degeneration in Rats.Low-level laser therapy regulates microglial function through Src-mediated signaling pathways: implications for neurodegenerative diseases New perspectives for natural antimicrobial peptides: application as antinflammatory drugs in a murine model Inflammasomes as therapeutic targets for Alzheimer's disease.Neurovascular dysfunction, inflammation and endothelial activation: implications for the pathogenesis of Alzheimer's disease.Expression of Tau40 induces activation of cultured rat microglial cells.Preconditioning of microglia by α-synuclein strongly affects the response induced by toll-like receptor (TLR) stimulation.Lipopolysaccharide-induced tau phosphorylation and kinase activity--modulation, but not mediation, by corticotropin-releasing factor receptors.Tau depletion prevents progressive blood-brain barrier damage in a mouse model of tauopathy Impact of CRFR1 Ablation on Amyloid-β Production and Accumulation in a Mouse Model of Alzheimer's Disease.Protective Effect of RNase on Unilateral Nephrectomy-Induced Postoperative Cognitive Dysfunction in Aged Mice Neuronal Cell Death and Degeneration through Increased Nitroxidative Stress and Tau Phosphorylation in HIV-1 Transgenic Rats.Microglial malfunction: the third rail in the development of Alzheimer's disease.Recent Advances in the Study of Bipolar/Rod-Shaped Microglia and their Roles in Neurodegeneration.Cyclic AMP is a key regulator of M1 to M2a phenotypic conversion of microglia in the presence of Th2 cytokines Suppression of LPS-induced tau hyperphosphorylation by serum amyloid A.Neuroinflammatory phenotype in early Alzheimer's disease.The blockage of the Nogo/NgR signal pathway in microglia alleviates the formation of Aβ plaques and tau phosphorylation in APP/PS1 transgenic mice.Thinking laterally about neurodegenerative proteinopathies.Sustained interleukin-1β overexpression exacerbates tau pathology despite reduced amyloid burden in an Alzheimer's mouse model.Diverse activation of microglia by chemokine (C-C motif) ligand 2 overexpression in brain.Amyloid oligomers exacerbate tau pathology in a mouse model of tauopathy

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LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

http://www.wikidata.org/entity/Q34173381

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

@zh-tw

2010年论文

@wuu

name

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@ast

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@en

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@nl

type

label

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@ast

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@en

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@nl

prefLabel

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@ast

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@en

LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

@nl

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Journal of Neuroinflammation

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LPS- induced inflammation exacerbates phospho-tau pathology in rTg4510 mice

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Amelia Johnson

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Chad A Dickey

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Clara Kraft

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Daniel C Lee

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Dave Morgan

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Justin Rizer

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Maj-Linda B Selenica

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Marcia N Gordon

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Patrick Reid

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P2860

Time course of the development of Alzheimer-like pathology in the doubly transgenic PS1+APP mouse

The crystal structure of a novel mammalian lectin, Ym1, suggests a saccharide binding site

Interleukin-1 mediates pathological effects of microglia on tau phosphorylation and on synaptophysin synthesis in cortical neurons through a p38-MAPK pathway

Alternative activation of macrophages: an immunologic functional perspective

Tau suppression in a neurodegenerative mouse model improves memory function

Inflammation, microglia, and Alzheimer's disease.

Inhibition of glycogen synthase kinase-3 by lithium correlates with reduced tauopathy and degeneration in vivo

Massive gliosis induced by interleukin-6 suppresses Abeta deposition in vivo: evidence against inflammation as a driving force for amyloid deposition.

Region-specific dissociation of neuronal loss and neurofibrillary pathology in a mouse model of tauopathy.

Expression profiles for macrophage alternative activation genes in AD and in mouse models of AD

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10.1186/1742-2094-7-56

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1019856959

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20846376

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2949628

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