Characterization of the EBV-Induced Persistent DNA Damage Response.

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Characterization of the EBV-Induced Persistent DNA Damage Response.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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Characterization of the EBV-Induced Persistent DNA Damage Response.

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Characterization of the EBV-Induced Persistent DNA Damage Response.

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Characterization of the EBV-Induced Persistent DNA Damage Response.

@en

Characterization of the EBV-Induced Persistent DNA Damage Response.

@nl

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Characterization of the EBV-Induced Persistent DNA Damage Response.

@en

Characterization of the EBV-Induced Persistent DNA Damage Response.

@nl

P1476

Characterization of the EBV-Induced Persistent DNA Damage Response.

@en

P2093

Amy Y Hafez

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P2860

Telomere shortening triggers senescence of human cells through a pathway involving ATM, p53, and p21(CIP1), but not p16(INK4a)

Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a

Sex hormones, acting on the TERT gene, increase telomerase activity in human primary hematopoietic cells

Danazol Treatment for Telomere Diseases

The serial cultivation of human diploid cell strains

Activation of the DNA damage checkpoint and genomic instability in human precancerous lesions

DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis

Senescence-associated secretory phenotypes reveal cell-nonautonomous functions of oncogenic RAS and the p53 tumor suppressor

A DNA damage checkpoint response in telomere-initiated senescence

Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints

P31

scholarly article

P356

10.3390/V9120366

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P433

http://www.w3.org/2001/XMLSchema#string

P478

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P50

Micah A Luftig

P577

2017-12-01T00:00:00Z

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P698

29194355

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P932

5744141

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