Connexin43 null mutation increases infarct size after stroke.
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Purinergic receptor stimulation reduces cytotoxic edema and brain infarcts in mouse induced by photothrombosis by energizing glial mitochondriaRole of gap junction protein connexin43 in astrogliosis induced by brain injuryThe complex of ciliary neurotrophic factor-ciliary neurotrophic factor receptor alpha up-regulates connexin43 and intercellular coupling in astrocytes via the Janus tyrosine kinase/signal transducer and activator of transcription pathwayConnexin43, the major gap junction protein of astrocytes, is down-regulated in inflamed white matter in an animal model of multiple sclerosis.The role of gap junction channels during physiologic and pathologic conditions of the human central nervous systemmicroRNAs Involved in Regulating Spontaneous Recovery in Embolic Stroke Model.Gap junctions and neuronal injury: protectants or executioners?Gap junction hemichannels in astrocytes of the CNS.Neurons and brain macrophages regulate connexin expression in cultured astrocytes.Connexin 43 is an emerging therapeutic target in ischemia/reperfusion injury, cardioprotection and neuroprotectionGap junctions mediate human immunodeficiency virus-bystander killing in astrocytesEnhanced connexin 43 expression following neural stem cell transplantation in a rat model of traumatic brain injury.Staphylococcus aureus-derived peptidoglycan induces Cx43 expression and functional gap junction intercellular communication in microglia.Neuroprotective Role of Gap Junctions in a Neuron Astrocyte Network Model.Modulation of brain hemichannels and gap junction channels by pro-inflammatory agents and their possible role in neurodegenerationIschemia alters the expression of connexins in the aged human brain.The role of astrocytes in mediating exogenous cell-based restorative therapy for strokeRole of astrocytes in brain function and disease.Increased apoptosis and inflammation after focal brain ischemia in mice lacking connexin43 in astrocytes.Upregulation of Connexin 43 Expression Via C-Jun N-Terminal Kinase Signaling in Prion Disease.Cellular expression of connexins in the rat brain: neuronal localization, effects of kainate-induced seizures and expression in apoptotic neuronal cells.Protective effects of carbenoxolone are associated with attenuation of oxidative stress in ischemic brain injuryA neuroprotective role for gap junctions.Modulation of gene expression during early stages of reconnection of the turtle spinal cord.Blocked gap junctional coupling increases glutamate-induced neurotoxicity in neuron-astrocyte co-cultures.Loss of astrocyte connexins 43 and 30 does not significantly alter susceptibility or severity of acute experimental autoimmune encephalomyelitis in mice.Connexin 43 mimetic peptides reduce swelling, astrogliosis, and neuronal cell death after spinal cord injury.Acute connexin43 temporal and spatial expression in response to ischemic stroke.Phosphorylation of connexin 43 induced by traumatic brain injury promotes exosome release.Connexins in Cardiovascular and Neurovascular Health and Disease: Pharmacological Implications.Neuroprotective role of astrocytic gap junctions in ischemic stroke.Gap-junction blocker carbenoxolone differentially enhances NMDA-induced cell death in hippocampal neurons and astrocytes in co-culture.The connexin43 C-terminal region mediates neuroprotection during stroke.Conflicting Roles of Connexin43 in Tumor Invasion and Growth in the Central Nervous System.Roles of astrocytic connexin-43, hemichannels, and gap junctions in oxygen-glucose deprivation/reperfusion injury induced neuroinflammation and the possible regulatory mechanisms of salvianolic acid B and carbenoxolone.
P2860
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P2860
Connexin43 null mutation increases infarct size after stroke.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年学术文章
@wuu
2001年学术文章
@zh
2001年学术文章
@zh-cn
2001年学术文章
@zh-hans
2001年学术文章
@zh-my
2001年学术文章
@zh-sg
2001年學術文章
@yue
2001年學術文章
@zh-hant
name
Connexin43 null mutation increases infarct size after stroke.
@en
Connexin43 null mutation increases infarct size after stroke.
@nl
type
label
Connexin43 null mutation increases infarct size after stroke.
@en
Connexin43 null mutation increases infarct size after stroke.
@nl
prefLabel
Connexin43 null mutation increases infarct size after stroke.
@en
Connexin43 null mutation increases infarct size after stroke.
@nl
P2093
P2860
P356
P1476
Connexin43 null mutation increases infarct size after stroke.
@en
P2093
Bechberger JF
Cechetto DF
Hachinski VC
Siushansian R
P2860
P304
P356
10.1002/CNE.1392
P407
P577
2001-11-01T00:00:00Z