Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
about
Somatic Mutations and Clonal Hematopoiesis: Unexpected Potential New Drivers of Age-Related Cardiovascular Disease.ASXL1 and SETBP1 mutations promote leukaemogenesis by repressing TGFβ pathway genes through histone deacetylationAsxl1 exerts an antiproliferative effect on mouse lung maturation via epigenetic repression of the E2f1-Nmyc axis
P2860
Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年學術文章
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2017年學術文章
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name
Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
@en
Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
@nl
type
label
Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
@en
Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
@nl
prefLabel
Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
@en
Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.
@nl
P2093
P2860
P50
P1433
P1476
Gain of function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies
@en
P2093
Claes Wahlestedt
Feng-Chun Yang
Hassan Al-Ali
Ines Lohse
Jianping Li
Lingxiao Li
Matthew G Field
Mingjiang Xu
P2860
P304
P356
10.1182/BLOOD-2017-06-789669
P407
P577
2017-11-07T00:00:00Z