A human homolog of Additional sex combs, ADDITIONAL SEX COMBS-LIKE 1, maps to chromosome 20q11
about
A Chronic Fatigue Syndrome - related proteome in human cerebrospinal fluid.Epigenetic regulators and their impact on therapy in acute myeloid leukemiaLoss-of-function Additional sex combs like 1 mutations disrupt hematopoiesis but do not cause severe myelodysplasia or leukemiaAdditional sex combs-like 1 belongs to the enhancer of trithorax and polycomb group and genetically interacts with Cbx2 in miceThe H3K27me3 demethylase UTX in normal development and disease.Great challenges in molecular medicine: toward personalized medicine.Additional sex combs-like 2 is required for polycomb repressive complex 2 binding at select targetsASXL1 mutations in younger adult patients with acute myeloid leukemia: a study by the German-Austrian Acute Myeloid Leukemia Study Group.Acquired mutations in ASXL1 in acute myeloid leukemia: prevalence and prognostic value.Epigenetic abnormalities in myeloproliferative neoplasms: a target for novel therapeutic strategies.Functional and cancer genomics of ASXL family membersDe Novo Truncating Variants in ASXL2 Are Associated with a Unique and Recognizable Clinical Phenotype.Loss of Asxl1 leads to myelodysplastic syndrome-like disease in miceInducible gene trapping with drug-selectable markers and Cre/loxP to identify developmentally regulated genes.Asxl1 deficiency in embryonic fibroblasts leads to cellular senescence via impairment of the AKT-E2F pathway and Ezh2 inactivation.The Role of Additional Sex Combs-Like Proteins in Cancer.ASXL1 interacts with the cohesin complex to maintain chromatid separation and gene expression for normal hematopoiesis.Additional sex combs interacts with enhancer of zeste and trithorax and modulates levels of trimethylation on histone H3K4 and H3K27 during transcription of hsp70.ASXL gain-of-function truncation mutants: defective and dysregulated forms of a natural ribosomal frameshifting product?Gain-of-function of ASXL1 truncating protein in the pathogenesis of myeloid malignancies.Clonal Hematopoiesis and Evolution to Hematopoietic Malignancies.Clinical management of patients with ASXL1 mutations and Bohring-Opitz syndrome, emphasizing the need for Wilms tumor surveillance.ASXL1 and SETBP1 mutations promote leukaemogenesis by repressing TGFβ pathway genes through histone deacetylation
P2860
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P2860
A human homolog of Additional sex combs, ADDITIONAL SEX COMBS-LIKE 1, maps to chromosome 20q11
description
2003 nî lūn-bûn
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2003 թվականի մարտին հրատարակված գիտական հոդված
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2003年の論文
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2003年論文
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2003年論文
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2003年論文
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name
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@ast
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@en
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@nl
type
label
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@ast
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@en
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@nl
prefLabel
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@ast
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@en
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@nl
P2093
P1433
P1476
A human homolog of Additional ...... KE 1, maps to chromosome 20q11
@en
P2093
Cynthia L Fisher
Filippo Randazzo
Hugh W Brock
Joel Berger
P304
P407
P577
2003-03-13T00:00:00Z