Deficit in PINK1/PARKIN-mediated mitochondrial autophagy at late stages of dystrophic cardiomyopathy.

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Deficit in PINK1/PARKIN-mediated mitochondrial autophagy at late stages of dystrophic cardiomyopathy.

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http://www.wikidata.org/entity/Q47646874

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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name

Deficit in PINK1/PARKIN-mediat ...... of dystrophic cardiomyopathy.

@en

Deficit in PINK1/PARKIN-mediat ...... of dystrophic cardiomyopathy.

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type

Item

label

Deficit in PINK1/PARKIN-mediat ...... of dystrophic cardiomyopathy.

@en

Deficit in PINK1/PARKIN-mediat ...... of dystrophic cardiomyopathy.

@nl

prefLabel

Deficit in PINK1/PARKIN-mediat ...... of dystrophic cardiomyopathy.

@en

Deficit in PINK1/PARKIN-mediat ...... of dystrophic cardiomyopathy.

@nl

P1476

Deficit in PINK1/PARKIN-mediat ...... s of dystrophic cardiomyopathy

@en

P2093

Chifei Kang

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Myriam A Badr

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Viktoriia Kyrychenko

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P2860

PINK1 is selectively stabilized on impaired mitochondria to activate Parkin

Guidelines for the use and interpretation of assays for monitoring autophagy

Parkin is activated by PINK1-dependent phosphorylation of ubiquitin at Ser65

Leaky RyR2 trigger ventricular arrhythmias in Duchenne muscular dystrophy

Hierarchical accumulation of RyR post-translational modifications drives disease progression in dystrophic cardiomyopathy

Reactive oxygen species are essential for autophagy and specifically regulate the activity of Atg4

Mitochondrial biogenesis and dynamics in the developing and diseased heart

The three 'P's of mitophagy: PARKIN, PINK1, and post-translational modifications

Parkin structure and function

Mitochondrial structure and function are disrupted by standard isolation methods

P304

90-102

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P31

scholarly article

P356

10.1093/CVR/CVX201

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P433

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P478

114

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P50

Eeva-Liisa Eskelinen

Natalia Shirokova

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2018-01-01T00:00:00Z

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29036556

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P932

5852524

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