about
Hierarchical accumulation of RyR post-translational modifications drives disease progression in dystrophic cardiomyopathyA preferred amplitude of calcium sparks in skeletal muscle.Calcium sparks: release packets of uncertain origin and fundamental roleInvolvement of multiple intracellular release channels in calcium sparks of skeletal muscle.Mitochondrial dysfunctions during progression of dystrophic cardiomyopathyPivotal role of miR-448 in the development of ROS-induced cardiomyopathy.The spark and its ember: separately gated local components of Ca(2+) release in skeletal muscleCa2+ release from the sarcoplasmic reticulum compared in amphibian and mammalian skeletal muscle.A damped oscillation in the intramembranous charge movement and calcium release flux of frog skeletal muscle fibers.Calcium release flux underlying Ca2+ sparks of frog skeletal muscle.Type 1 inositol (1,4,5)-trisphosphate receptor activates ryanodine receptor 1 to mediate calcium spark signaling in adult mammalian skeletal muscleA guide to sparkology: the taxonomy of elementary cellular Ca2+ signaling events.Pathways of abnormal stress-induced Ca2+ influx into dystrophic mdx cardiomyocytesMetabolic regulation of Ca2+ release in permeabilized mammalian skeletal muscle fibres.Transfer and tunneling of Ca2+ from sarcoplasmic reticulum to mitochondria in skeletal muscle.Reciprocal amplification of ROS and Ca(2+) signals in stressed mdx dystrophic skeletal muscle fibers.Reactive oxygen species contribute to Ca2+ signals produced by osmotic stress in mouse skeletal muscle fibres.Deficit in PINK1/PARKIN-mediated mitochondrial autophagy at late stages of dystrophic cardiomyopathy.Small Fractions of Muscular Dystrophy Embryonic Stem Cells Yield Severe Cardiac and Skeletal Muscle Defects in Adult Mouse Chimeras.Small event Ca2+ release: a probable precursor of Ca2+ sparks in frog skeletal muscle.Normalization of connexin 43 protein levels prevents cellular and functional signs of dystrophic cardiomyopathy in mice.Local calcium release in mammalian skeletal muscle.Activation of Ca2+ release by caffeine and voltage in frog skeletal muscleCaffeine enhances intramembranous charge movement in frog skeletal muscle by increasing cytoplasmic Ca2+ concentrationProperties and roles of an intramembranous charge mobilized at high voltages in frog skeletal muscle'Quantal' calcium release operated by membrane voltage in frog skeletal muscleDystrophic cardiomyopathy: amplification of cellular damage by Ca2+ signalling and reactive oxygen species-generating pathways
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description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Natalia Shirokova
@ast
Natalia Shirokova
@en
Natalia Shirokova
@es
Natalia Shirokova
@nl
Natalia Shirokova
@sl
type
label
Natalia Shirokova
@ast
Natalia Shirokova
@en
Natalia Shirokova
@es
Natalia Shirokova
@nl
Natalia Shirokova
@sl
altLabel
Natalia Pivovarova
@en
prefLabel
Natalia Shirokova
@ast
Natalia Shirokova
@en
Natalia Shirokova
@es
Natalia Shirokova
@nl
Natalia Shirokova
@sl
P106
P1153
7003628999
P21
P31
P496
0000-0003-4648-4592