Amyloid deposition in a mouse model humanized at the transthyretin and retinol-binding protein 4 loci.

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Amyloid deposition in a mouse model humanized at the transthyretin and retinol-binding protein 4 loci.

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2018 nî lūn-bûn

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2018年の論文

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年学术文章

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2018年學術文章

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2018年學術文章

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2018年學術文章

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Amyloid deposition in a mouse ...... etinol-binding protein 4 loci.

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Amyloid deposition in a mouse ...... etinol-binding protein 4 loci.

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Item

label

Amyloid deposition in a mouse ...... etinol-binding protein 4 loci.

@en

Amyloid deposition in a mouse ...... etinol-binding protein 4 loci.

@nl

prefLabel

Amyloid deposition in a mouse ...... etinol-binding protein 4 loci.

@en

Amyloid deposition in a mouse ...... etinol-binding protein 4 loci.

@nl

P1476

Amyloid deposition in a mouse ...... retinol-binding protein 4 loci

@en

P2093

Jingling Shen

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Ken-Ichi Yamamura

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Kimi Araki

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Li Liu

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Lixia Qiang

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Shoude Jin

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Xiangshun Li

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Yanshuang Mu

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Yanyi Lyu

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Zhenghua Li

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P2860

Structural evidence for native state stabilization of a conformationally labile amyloidogenic transthyretin variant by fibrillogenesis inhibitors

Plasma Transthyretin as a Biomarker of Lean Body Mass and Catabolic States

Rational design of potent human transthyretin amyloid disease inhibitors

Human-murine transthyretin heterotetramers are kinetically stable and non-amyloidogenic. A lesson in the generation of transgenic models of diseases involving oligomeric proteins

Repurposing diflunisal for familial amyloid polyneuropathy: a randomized clinical trial

Bisaryloxime ethers as potent inhibitors of transthyretin amyloid fibril formation.

Pathogenesis of and therapeutic strategies to ameliorate the transthyretin amyloidoses.

Reference distributions for the negative acute-phase serum proteins, albumin, transferrin and transthyretin: a practical, simple and clinically relevant approach in a large cohort.

Tafamidis for transthyretin familial amyloid polyneuropathy: a randomized, controlled trial

Support for the multigenic hypothesis of amyloidosis: the binding stoichiometry of retinol-binding protein, vitamin A, and thyroid hormone influences transthyretin amyloidogenicity in vitro.

P2888

s41374-017-0019-y

P304

512-524

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scholarly article

P356

10.1038/S41374-017-0019-Y

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P433

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P478

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P50

Bruno Imbimbo

P577

2018-01-12T00:00:00Z

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P698

29330472

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