Environmental factors determine DAP12 deficiency to either enhance or suppress immunopathogenic processes.

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2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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Environmental factors determin ...... ss immunopathogenic processes.

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Environmental factors determin ...... ss immunopathogenic processes.

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Environmental factors determin ...... ss immunopathogenic processes.

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Environmental factors determin ...... ss immunopathogenic processes.

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Environmental factors determin ...... ss immunopathogenic processes.

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Environmental factors determin ...... ss immunopathogenic processes.

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P1476

Environmental factors determin ...... ss immunopathogenic processes.

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P2093

Barbara P Vistica

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Daniel W McVicar

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Igal Gery

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Kimberly C Boelte

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Laura Quigley

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Lindsey F Nugent

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Toshiyuki Takai

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Vanessa Montalvo

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P2860

Cutting edge: inflammatory responses can be triggered by TREM-1, a novel receptor expressed on neutrophils and monocytes

Immunoreceptor DAP12 bearing a tyrosine-based activation motif is involved in activating NK cells

TREM-1 amplifies inflammation and is a crucial mediator of septic shock

Osteopetrosis and thalamic hypomyelinosis with synaptic degeneration in DAP12-deficient mice

DAP12-mediated signal transduction in natural killer cells. A dominant role for the Syk protein-tyrosine kinase

TREM2-transduced myeloid precursors mediate nervous tissue debris clearance and facilitate recovery in an animal model of multiple sclerosis.

The linker for activation of B cells (LAB)/non-T cell activation linker (NTAL) regulates triggering receptor expressed on myeloid cells (TREM)-2 signaling and macrophage inflammatory responses independently of the linker for activation of T cells.

Role of the innate immune system in the pathogenesis of multiple sclerosis.

Immune adaptations that maintain homeostasis with the intestinal microbiota.

The microbiome in infectious disease and inflammation.

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475-482

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scholarly article

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10.1111/IMM.12158

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P478

140

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2013-12-01T00:00:00Z

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23906311

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3839651

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