IL-12Rβ2 is essential for the development of experimental cerebral malaria.
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Endothelin-1 Mediates Brain Microvascular Dysfunction Leading to Long-Term Cognitive Impairment in a Model of Experimental Cerebral MalariaExperimental cerebral malaria pathogenesis--hemodynamics at the blood brain barrierToxoplasma gondii upregulates interleukin-12 to prevent Plasmodium berghei-induced experimental cerebral malaria.Imaging Plasmodium immunobiology in the liver, brain, and lung.IL-33 receptor ST2 regulates the cognitive impairments associated with experimental cerebral malariaGenetic variations of IL-12B, IL-12Rβ1, IL-12Rβ2 in Behcet's disease and VKH syndromeInterferons and interferon regulatory factors in malariaLipoxin A₄ and 15-epi-lipoxin A₄ protect against experimental cerebral malaria by inhibiting IL-12/IFN-γ in the brainIL-12Rβ2 is critical for survival of primary Francisella tularensis LVS infectionImprovement of the efficacy of dihydroartemisinin with atorvastatin in an experimental cerebral malaria murine model.A new hypothesis on the manifestation of cerebral malaria: the secret is in the liverFunctional roles for C5a and C5aR but not C5L2 in the pathogenesis of human and experimental cerebral malaria.Imaging of small-animal models of infectious diseases.Immunobiology of Plasmodium in liver and brain.Steroid Pulse Therapy May Mitigate Prolonged Neurological Manifestations after Eradication of Severe Plasmodium falciparum ParasitemiaMyeloid expression of the AP-1 transcription factor JUNB modulates outcomes of type 1 and type 2 parasitic infections.Critical role of IL-33 receptor ST2 in experimental cerebral malaria development.Profiling MHC II immunopeptidome of blood-stage malaria reveals that cDC1 control the functionality of parasite-specific CD4 T cells.Type I interferons contribute to experimental cerebral malaria development in response to sporozoite or blood-stage Plasmodium berghei ANKA.Interleukin-15 Complex Treatment Protects Mice from Cerebral Malaria by Inducing Interleukin-10-Producing Natural Killer Cells.The Glycosylphosphatidylinositol Transamidase Complex Subunit PbGPI16 of Plasmodium berghei Is Important for Inducing Experimental Cerebral Malaria
P2860
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P2860
IL-12Rβ2 is essential for the development of experimental cerebral malaria.
description
2012 nî lūn-bûn
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2012年の論文
@ja
2012年学术文章
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2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
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2012年學術文章
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name
IL-12Rβ2 is essential for the development of experimental cerebral malaria.
@en
IL-12Rβ2 is essential for the development of experimental cerebral malaria.
@nl
type
label
IL-12Rβ2 is essential for the development of experimental cerebral malaria.
@en
IL-12Rβ2 is essential for the development of experimental cerebral malaria.
@nl
prefLabel
IL-12Rβ2 is essential for the development of experimental cerebral malaria.
@en
IL-12Rβ2 is essential for the development of experimental cerebral malaria.
@nl
P2093
P356
P1476
IL-12Rβ2 is essential for the development of experimental cerebral malaria.
@en
P2093
Adeline Danneels
Frédéric Szeremeta
Jean-Claude Beloeil
Jennifer Palomo
Marie-Laure Bourigault
Mathilde Fauconnier
Sabine Charron
Sandra Meme
Valérie F J Quesniaux
P304
P356
10.4049/JIMMUNOL.1101978
P407
P577
2012-01-11T00:00:00Z