Type I interferons contribute to experimental cerebral malaria development in response to sporozoite or blood-stage Plasmodium berghei ANKA.
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Cerebral malaria: gamma-interferon reduxType I Interferons Induce T Regulatory 1 Responses and Restrict Humoral Immunity during Experimental MalariaIL-33 receptor ST2 regulates the cognitive impairments associated with experimental cerebral malariaReduction of experimental cerebral malaria and its related proinflammatory responses by the novel liposome-based β-methasone nanodrug.Interferons and interferon regulatory factors in malariaCytokine and chemokine profile of the innate and adaptive immune response of Schistosoma haematobium and Plasmodium falciparum single and co-infected school-aged children from an endemic area of Lambaréné, Gabon.Transcriptomic profiling of microglia reveals signatures of cell activation and immune response, during experimental cerebral malaria.Strain-specific innate immune signaling pathways determine malaria parasitemia dynamics and host mortality.Cytokines and Chemokines in Cerebral Malaria Pathogenesis.Critical role of IL-33 receptor ST2 in experimental cerebral malaria development.Plasmodium attenuation: connecting the dots between early immune responses and malaria disease severityIL-25, IL-33 and TSLP receptor are not critical for development of experimental murine malaria.USP15 regulates type I interferon response and is required for pathogenesis of neuroinflammation.cGAS-mediated control of blood-stage malaria promotes Plasmodium-specific germinal center responses.Type I Interferon Signaling Is Required for CpG-Oligodesoxynucleotide-Induced Control of Leishmania major, but Not for Spontaneous Cure of Subcutaneous Primary or Secondary L. major Infection.Fetal and Maternal Innate Immunity Receptors Have Opposing Effects on the Severity of Experimental Malaria in Pregnancy: Beneficial Roles for Fetus-Derived Toll-Like Receptor 4 and Type I Interferon Receptor 1.Type I Interferon Receptor Variants in Gene Regulatory Regions are Associated with Susceptibility to Cerebral Malaria in Malawi.CXCL10 stabilizes T cell-brain endothelial cell adhesion leading to the induction of cerebral malaria.Plasmodium and intestinal parasite perturbations of the infected host's inflammatory responses: a systematic reviewEffects of type I interferons in malariaA Cross-Stage Antigen Contributes to the Development of Experimental Cerebral Malaria
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P2860
Type I interferons contribute to experimental cerebral malaria development in response to sporozoite or blood-stage Plasmodium berghei ANKA.
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2013 nî lūn-bûn
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name
Type I interferons contribute ...... stage Plasmodium berghei ANKA.
@en
Type I interferons contribute ...... stage Plasmodium berghei ANKA.
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type
label
Type I interferons contribute ...... stage Plasmodium berghei ANKA.
@en
Type I interferons contribute ...... stage Plasmodium berghei ANKA.
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prefLabel
Type I interferons contribute ...... stage Plasmodium berghei ANKA.
@en
Type I interferons contribute ...... stage Plasmodium berghei ANKA.
@nl
P2093
P2860
P921
P356
P1476
Type I interferons contribute ...... stage Plasmodium berghei ANKA.
@en
P2093
Dieudonnée Togbe
Dominique Mazier
Frederic Szeremeta
Jean-Claude Beloeil
Jennifer Palomo
Laurie Coquard
Lizette Fick
Mathilde Fauconnier
Maïlys Gilles
Muazzam Jacobs
P2860
P304
P356
10.1002/EJI.201343327
P407
P577
2013-07-19T00:00:00Z