The time course of selected malarial infections in cytokine-deficient mice.
about
Complex genetic control of susceptibility to malaria: positional cloning of the Char9 locusInterferon-γ: The Jekyll and Hyde of MalariaProtection and pathology during parasite infection: IL-10 strikes the balanceInduction of proinflammatory responses in macrophages by the glycosylphosphatidylinositols of Plasmodium falciparum: cell signaling receptors, glycosylphosphatidylinositol (GPI) structural requirement, and regulation of GPI activityOn the control of acute rodent malaria infections by innate immunityImmune responses of NIH mice infected with avirulent and virulent strains of Plasmodium chabaudi adami single and mixed infections.A role for cytokines in potentiation of malaria vaccines through immunological modulation of blood stage infection.Roles of CD4(+) T cells and gamma interferon in protective immunity against Babesia microti infection in mice.Central role of endogenous gamma interferon in protective immunity against blood-stage Plasmodium chabaudi AS infection.Complement contributes to protective immunity against reinfection by Plasmodium chabaudi chabaudi parasites.Antigen-presenting cell function during Plasmodium yoelii infection.Gammadelta T cells but not NK cells are essential for cell-mediated immunity against Plasmodium chabaudi malaria.Osteopontin participates in Th1-mediated host resistance against nonlethal malaria parasite Plasmodium chabaudi chabaudi infection in miceSplenic gammadelta T cells regulated by CD4+ T cells are required to control chronic Plasmodium chabaudi malaria in the B-cell-deficient mouseIrf8-regulated genomic responses drive pathological inflammation during cerebral malaria.Signalling through the IL-2 receptor γ(c) peptide (CD132) is essential for the expression of immunity to Plasmodium chabaudi adami blood-stage malaria.Dissemination of Chlamydia trachomatis chronic genital tract infection in gamma interferon gene knockout mice.Vaccination with novel immunostimulatory adjuvants against blood-stage malaria in mice.Macrophage migration inhibitory factor: a downregulator of early T cell-dependent IFN-gamma responses in Plasmodium chabaudi adami (556 KA)-infected mice.CD8+ T Cells Induce Fatal Brainstem Pathology during Cerebral Malaria via Luminal Antigen-Specific Engagement of Brain Vasculature.Mice deficient in interleukin-4 (IL-4) or IL-4 receptor alpha have higher resistance to sporozoite infection with Plasmodium berghei (ANKA) than do naive wild-type mice.Macrophage-mediated but gamma interferon-independent innate immune responses control the primary wave of Plasmodium yoelii parasitemiaTransforming growth factor beta production is inversely correlated with severity of murine malaria infection.Roles of IFN-γ and γδ T Cells in Protective Immunity Against Blood-Stage MalariaIL-18-induced expression of high-affinity IL-2R on murine NK cells is essential for NK-cell IFN-γ production during murine Plasmodium yoelii infection.Type I Interferons Regulate Immune Responses in Humans with Blood-Stage Plasmodium falciparum Infection.Protection against Plasmodium chabaudi malaria induced by immunization with apical membrane antigen 1 and merozoite surface protein 1 in the absence of gamma interferon or interleukin-4.CD28 costimulation is required for the expression of T-cell-dependent cell-mediated immunity against blood-stage Plasmodium chabaudi malaria parasites.NK Cells: Uncertain Allies against Malaria.Interferon-γ--central mediator of protective immune responses against the pre-erythrocytic and blood stage of malaria.Host resistance to malaria: using mouse models to explore the host response.Do you see what I see: Recognition of protozoan parasites by Toll-like receptors.Mechanism of splenic cell death and host mortality in a Plasmodium yoelii malaria model.Immunological memory to blood-stage malaria infection is controlled by the histamine releasing factor (HRF) of the parasite.Plasmodium attenuation: connecting the dots between early immune responses and malaria disease severityDivergent roles of IRAK4-mediated innate immune responses in two experimental models of severe malaria.Mapping of Char10, a novel malaria susceptibility locus on mouse chromosome 9.11β-hydroxysteroid dehydrogenase type 1 has no effect on survival during experimental malaria but affects parasitemia in a parasite strain-specific manner.Development of a Novel CD4+ TCR Transgenic Line That Reveals a Dominant Role for CD8+ Dendritic Cells and CD40 Signaling in the Generation of Helper and CTL Responses to Blood-Stage Malaria.CD41 is a reliable identification and activation marker for murine basophils in the steady state and during helminth and malarial infections.
P2860
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P2860
The time course of selected malarial infections in cytokine-deficient mice.
description
1997 nî lūn-bûn
@nan
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
1997年學術文章
@zh-hant
name
The time course of selected malarial infections in cytokine-deficient mice.
@en
The time course of selected malarial infections in cytokine-deficient mice.
@nl
type
label
The time course of selected malarial infections in cytokine-deficient mice.
@en
The time course of selected malarial infections in cytokine-deficient mice.
@nl
prefLabel
The time course of selected malarial infections in cytokine-deficient mice.
@en
The time course of selected malarial infections in cytokine-deficient mice.
@nl
P2093
P356
P1476
The time course of selected malarial infections in cytokine-deficient mice.
@en
P2093
Batchelder J
Weidanz WP
van der Heyde HC
P304
P356
10.1006/EXPR.1996.4132
P577
1997-02-01T00:00:00Z