How AMPK and PKA Interplay to Regulate Mitochondrial Function and Survival in Models of Ischemia and Diabetes.

about

How AMPK and PKA Interplay to Regulate Mitochondrial Function and Survival in Models of Ischemia and Diabetes.

Item

http://www.wikidata.org/entity/Q48042214

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

How AMPK and PKA Interplay to ...... dels of Ischemia and Diabetes.

@en

How AMPK and PKA Interplay to ...... dels of Ischemia and Diabetes.

@nl

type

Item

label

How AMPK and PKA Interplay to ...... dels of Ischemia and Diabetes.

@en

How AMPK and PKA Interplay to ...... dels of Ischemia and Diabetes.

@nl

prefLabel

How AMPK and PKA Interplay to ...... dels of Ischemia and Diabetes.

@en

How AMPK and PKA Interplay to ...... dels of Ischemia and Diabetes.

@nl

P1476

How AMPK and PKA Interplay to ...... dels of Ischemia and Diabetes.

@en

P2093

Jingdian Zhang

http://www.w3.org/2001/XMLSchema#string

Xiaofeng Liu

http://www.w3.org/2001/XMLSchema#string

Ying Zhang

http://www.w3.org/2001/XMLSchema#string

Yumeng Wang

http://www.w3.org/2001/XMLSchema#string

P275

Creative Commons Attribution 4.0 International

P2860

Cardiolipin externalization to the outer mitochondrial membrane acts as an elimination signal for mitophagy in neuronal cells

Phosphorylation and activation of heart PFK-2 by AMPK has a role in the stimulation of glycolysis during ischaemia

Adiponectin stimulates glucose utilization and fatty-acid oxidation by activating AMP-activated protein kinase

The AMPK signalling pathway coordinates cell growth, autophagy and metabolism

Chemical Conditioning as an Approach to Ischemic Stroke Tolerance: Mitochondria as the Target

Role of protein kinase A in regulating mitochondrial function and neuronal development: implications to neurodegenerative diseases

After the banquet: mitochondrial biogenesis, mitophagy, and cell survival

Akap1 Deficiency Promotes Mitochondrial Aberrations and Exacerbates Cardiac Injury Following Permanent Coronary Ligation via Enhanced Mitophagy and Apoptosis

Mechanism of neuroprotective mitochondrial remodeling by PKA/AKAP1

Metabolism. AMP-activated protein kinase mediates mitochondrial fission in response to energy stress.

P304

4353510

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

review article

P356

10.1155/2017/4353510

http://www.w3.org/2001/XMLSchema#string

P478

2017

http://www.w3.org/2001/XMLSchema#string

P50

P5008

ScienceSource

P577

2017-12-17T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29391924

http://www.w3.org/2001/XMLSchema#string

P921

P932

5748092

http://www.w3.org/2001/XMLSchema#string