Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
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Sex differences in estrogenic regulation of neuronal activity in neonatal cultures of ventromedial nucleus of the hypothalamusA ketogenic diet rescues the murine succinic semialdehyde dehydrogenase deficient phenotypehPSC-derived maturing GABAergic interneurons ameliorate seizures and abnormal behavior in epileptic mice.Loss of GABAergic neurons in the subiculum and its functional implications in temporal lobe epilepsyPlasticity of rat central inhibitory synapses through GABA metabolismInitiation of network bursts by Ca2+-dependent intrinsic bursting in the rat pilocarpine model of temporal lobe epilepsySubunit-specific trafficking of GABA(A) receptors during status epilepticusGABA progenitors grafted into the adult epileptic brain control seizures and abnormal behaviorDysfunction of the dentate basket cell circuit in a rat model of temporal lobe epilepsy.Neuronal density and architecture (Gray Level Index) in the brains of autistic patients.Pre & postsynaptic tuning of action potential timing by spontaneous GABAergic activityPresynaptic inhibitory terminals are functionally abnormal in a rat model of posttraumatic epilepsyNeuronal chloride accumulation and excitatory GABA underlie aggravation of neonatal epileptiform activities by phenobarbital.What is GABAergic inhibition? How is it modified in epilepsy?Pathophysiological mechanisms underlying increased anxiety after soman exposure: reduced GABAergic inhibition in the basolateral amygdala.Progressive NKCC1-dependent neuronal chloride accumulation during neonatal seizures.Initial loss but later excess of GABAergic synapses with dentate granule cells in a rat model of temporal lobe epilepsy.Synaptic reorganization of inhibitory hilar interneuron circuitry after traumatic brain injury in miceDifferent mechanisms of ripple-like oscillations in the human epileptic subiculumMolecular biology and ontogeny of glutamate receptors in the mammalian central nervous system.Selective reduction of cholecystokinin-positive basket cell innervation in a model of temporal lobe epilepsyCooperative regulation of neurotransmitter release by Rab3a and synapsin II.Functional alterations in GABAergic fast-spiking interneurons in chronically injured epileptogenic neocortex.Depolarising and hyperpolarising actions of GABA(A) receptor activation on gonadotrophin-releasing hormone neurones: towards an emerging consensus.More Docked Vesicles and Larger Active Zones at Basket Cell-to-Granule Cell Synapses in a Rat Model of Temporal Lobe Epilepsy.Persistent Hyperactivity of Hippocampal Dentate Interneurons After a Silent Period in the Rat Pilocarpine Model of Epilepsy.Impaired expression and function of group II metabotropic glutamate receptors in pilocarpine-treated chronically epileptic rats.Epilepsies and neuronal plasticity: for better or for worse?Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis.Targeted disruption of layer 4 during development increases GABAA receptor neurotransmission in the neocortex.Differential control of spontaneous and evoked GABA release by presynaptic L-type Ca(2+) channels in the rat medial preoptic nucleus.Dual and opposing roles of presynaptic Ca2+ influx for spontaneous GABA release from rat medial preoptic nerve terminals.In vitro formation of a secondary epileptogenic mirror focus by interhippocampal propagation of seizures.Changes in mIPSCs and sIPSCs after kainate treatment: evidence for loss of inhibitory input to dentate granule cells and possible compensatory responses.Metabolism is normal in astrocytes in chronically epileptic rats: a (13)C NMR study of neuronal-glial interactions in a model of temporal lobe epilepsy.Mini-I on epilepsy. Focus on "Changes in mIPSCs and sIPSCs after kainate treatment: evidence for loss of inhibitory input to dentate granule cells and possible compensatory responses".GABA transporter-1 (GAT1)-deficient mice: differential tonic activation of GABAA versus GABAB receptors in the hippocampus.A brief period of epileptiform activity strengthens excitatory synapses in the rat hippocampus in vitro.Hyperexcitability of the CA1 hippocampal region during epileptogenesis.Commonalities in epileptogenic processes from different acute brain insults: Do they translate?
P2860
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P2860
Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
@zh-hant
name
Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
@en
Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
@nl
type
label
Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
@en
Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
@nl
prefLabel
Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
@en
Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
@nl
P2093
P2860
P356
P1433
P1476
Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.
@en
P2093
Agassandian C
Esclapez M
Merchán-Pérez A
P2860
P2888
P304
P356
10.1038/9142
P407
P577
1999-06-01T00:00:00Z
P5875
P6179
1005156306