Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy. - Wikidata - awgldk - TriplyDB

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

http://www.wikidata.org/entity/Q48134588

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Sex differences in estrogenic regulation of neuronal activity in neonatal cultures of ventromedial nucleus of the hypothalamus A ketogenic diet rescues the murine succinic semialdehyde dehydrogenase deficient phenotype hPSC-derived maturing GABAergic interneurons ameliorate seizures and abnormal behavior in epileptic mice.Loss of GABAergic neurons in the subiculum and its functional implications in temporal lobe epilepsy Plasticity of rat central inhibitory synapses through GABA metabolism Initiation of network bursts by Ca2+-dependent intrinsic bursting in the rat pilocarpine model of temporal lobe epilepsy Subunit-specific trafficking of GABA(A) receptors during status epilepticus GABA progenitors grafted into the adult epileptic brain control seizures and abnormal behavior Dysfunction of the dentate basket cell circuit in a rat model of temporal lobe epilepsy.Neuronal density and architecture (Gray Level Index) in the brains of autistic patients.Pre & postsynaptic tuning of action potential timing by spontaneous GABAergic activity Presynaptic inhibitory terminals are functionally abnormal in a rat model of posttraumatic epilepsy Neuronal chloride accumulation and excitatory GABA underlie aggravation of neonatal epileptiform activities by phenobarbital.What is GABAergic inhibition? How is it modified in epilepsy?Pathophysiological mechanisms underlying increased anxiety after soman exposure: reduced GABAergic inhibition in the basolateral amygdala.Progressive NKCC1-dependent neuronal chloride accumulation during neonatal seizures.Initial loss but later excess of GABAergic synapses with dentate granule cells in a rat model of temporal lobe epilepsy.Synaptic reorganization of inhibitory hilar interneuron circuitry after traumatic brain injury in mice Different mechanisms of ripple-like oscillations in the human epileptic subiculum Molecular biology and ontogeny of glutamate receptors in the mammalian central nervous system.Selective reduction of cholecystokinin-positive basket cell innervation in a model of temporal lobe epilepsy Cooperative regulation of neurotransmitter release by Rab3a and synapsin II.Functional alterations in GABAergic fast-spiking interneurons in chronically injured epileptogenic neocortex.Depolarising and hyperpolarising actions of GABA(A) receptor activation on gonadotrophin-releasing hormone neurones: towards an emerging consensus.More Docked Vesicles and Larger Active Zones at Basket Cell-to-Granule Cell Synapses in a Rat Model of Temporal Lobe Epilepsy.Persistent Hyperactivity of Hippocampal Dentate Interneurons After a Silent Period in the Rat Pilocarpine Model of Epilepsy.Impaired expression and function of group II metabotropic glutamate receptors in pilocarpine-treated chronically epileptic rats.Epilepsies and neuronal plasticity: for better or for worse?Pathological alterations in GABAergic interneurons and reduced tonic inhibition in the basolateral amygdala during epileptogenesis.Targeted disruption of layer 4 during development increases GABAA receptor neurotransmission in the neocortex.Differential control of spontaneous and evoked GABA release by presynaptic L-type Ca(2+) channels in the rat medial preoptic nucleus.Dual and opposing roles of presynaptic Ca2+ influx for spontaneous GABA release from rat medial preoptic nerve terminals.In vitro formation of a secondary epileptogenic mirror focus by interhippocampal propagation of seizures.Changes in mIPSCs and sIPSCs after kainate treatment: evidence for loss of inhibitory input to dentate granule cells and possible compensatory responses.Metabolism is normal in astrocytes in chronically epileptic rats: a (13)C NMR study of neuronal-glial interactions in a model of temporal lobe epilepsy.Mini-I on epilepsy. Focus on "Changes in mIPSCs and sIPSCs after kainate treatment: evidence for loss of inhibitory input to dentate granule cells and possible compensatory responses".GABA transporter-1 (GAT1)-deficient mice: differential tonic activation of GABAA versus GABAB receptors in the hippocampus.A brief period of epileptiform activity strengthens excitatory synapses in the rat hippocampus in vitro.Hyperexcitability of the CA1 hippocampal region during epileptogenesis.Commonalities in epileptogenic processes from different acute brain insults: Do they translate?

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P2860

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

http://www.wikidata.org/entity/Q48134588

description

1999 nî lūn-bûn

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1999年の論文

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年学术文章

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1999年學術文章

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1999年學術文章

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name

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

@en

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

@nl

type

label

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

@en

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

@nl

prefLabel

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

@en

Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

@nl

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Deficit of quantal release of GABA in experimental models of temporal lobe epilepsy.

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Agassandian C

http://www.w3.org/2001/XMLSchema#string

Ben-Ari Y

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Bernard C

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Esclapez M

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Hirsch JC

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Merchán-Pérez A

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P2860

Minireview. Kainic acid as a tool for the study of temporal lobe epilepsy

Essential functions of synapsins I and II in synaptic vesicle regulation

Interneurons of the hippocampus

Operative GABAergic inhibition in hippocampal CA1 pyramidal neurons in experimental epilepsy.

Genetic dissection of synaptic transmission in Drosophila.

Limbic seizures produced by pilocarpine in rats: behavioural, electroencephalographic and neuropathological study.

A functional role for GTP-binding proteins in synaptic vesicle cycling.

Heterogeneous release properties of visualized individual hippocampal synapses.

Newly formed excitatory pathways provide a substrate for hyperexcitability in experimental temporal lobe epilepsy.

Perpetual inhibitory activity in mammalian brain slices generated by spontaneous GABA release.

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6

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2

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Javier de Felipe

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