Connecting TNF-alpha signaling pathways to iNOS expression in a mouse model of Alzheimer's disease: relevance for the behavioral and synaptic deficits induced by amyloid beta protein.
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Rapid improvement in verbal fluency and aphasia following perispinal etanercept in Alzheimer's disease.Perispinal etanercept produces rapid improvement in primary progressive aphasia: identification of a novel, rapidly reversible TNF-mediated pathophysiologic mechanismTargeting innate immunity for neurodegenerative disorders of the central nervous systemTNF-α/NF-κB signaling in the CNS: possible connection to EPHB2New insights into the impact of neuro-inflammation in rheumatoid arthritisiNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant miceS-nitrosylation of Drp1 links excessive mitochondrial fission to neuronal injury in neurodegenerationAberrant protein S-nitrosylation contributes to the pathophysiology of neurodegenerative diseasesRapid cognitive improvement in Alzheimer's disease following perispinal etanercept administration.Role of the macrophage inflammatory protein-1alpha/CC chemokine receptor 5 signaling pathway in the neuroinflammatory response and cognitive deficits induced by beta-amyloid peptideRapid intracerebroventricular delivery of Cu-DOTA-etanercept after peripheral administration demonstrated by PET imaging.Cyclooxygenase-1 null mice show reduced neuroinflammation in response to beta-amyloid.Tumor necrosis factor α Inhibition for Alzheimer's Disease.Reductions in amyloid-beta-derived neuroinflammation, with minocycline, restore cognition but do not significantly affect tau hyperphosphorylation.Necroptosis mediates TNF-induced toxicity of hippocampal neurons.Perispinal etanercept for post-stroke neurological and cognitive dysfunction: scientific rationale and current evidenceCDK5 is essential for soluble amyloid β-induced degradation of GKAP and remodeling of the synaptic actin cytoskeletonTumor necrosis factor-alpha-induced sickness behavior is impaired by central administration of an inhibitor of c-jun N-terminal kinase.The time course of adenosine, nitric oxide (NO) and inducible NO synthase changes in the brain with sleep loss and their role in the non-rapid eye movement sleep homeostatic cascade.Cognitive-enhancing effects of polygalasaponin hydrolysate in aβ(25-35)-induced amnesic mice.Herbal substance, acteoside, alleviates intestinal mucositis in mice.Atherosclerosis and Alzheimer--diseases with a common cause? Inflammation, oxysterols, vasculatureMicroglial phagocytosis induced by fibrillar β-amyloid is attenuated by oligomeric β-amyloid: implications for Alzheimer's disease.Microcystin-LR induces apoptosis via NF-κB/iNOS pathway in INS-1 cellsAblation of TNF-RI/RII expression in Alzheimer's disease mice leads to an unexpected enhancement of pathology: implications for chronic pan-TNF-α suppressive therapeutic strategies in the brain.Effects of centrally administered etanercept on behavior, microglia, and astrocytes in mice following a peripheral immune challenge.Smad3-dependent signaling underlies the TGF-β1-mediated enhancement in astrocytic iNOS expressionRole of nitrative and oxidative DNA damage in inflammation-related carcinogenesis.Subventricular zone neural progenitors reverse TNF-alpha effects in cortical neurons.Therapeutic Effect of Yi-Chi-Tsung-Ming-Tang on Amyloid β-Induced Alzheimer's Disease-Like Phenotype via an Increase of Acetylcholine and Decrease of Amyloid β.Increased Contextual Fear Conditioning in iNOS Knockout Mice: Additional Evidence for the Involvement of Nitric Oxide in Stress-Related Disorders and Contribution of the Endocannabinoid SystemEarly intervention with a small molecule inhibitor for tumor necrosis factor-α prevents cognitive deficits in a triple transgenic mouse model of Alzheimer's diseaseIndomethacin-loaded lipid-core nanocapsules reduce the damage triggered by Aβ1-42 in Alzheimer's disease models.Microglial Inhibitory Mechanism of Coenzyme Q10 Against Aβ (1-42) Induced Cognitive Dysfunctions: Possible Behavioral, Biochemical, Cellular, and Histopathological Alterations.Advances in the pathogenesis of Alzheimer's disease: a re-evaluation of amyloid cascade hypothesis.Suppression of LPS-induced tau hyperphosphorylation by serum amyloid A.Mitochondrial dysfunction and cell death in neurodegenerative diseases through nitroxidative stressProtective Effects of Melatonin on Retinal Inflammation and Oxidative Stress in Experimental Diabetic Retinopathy.Neuroprotective Effects of Herbal Extract (Rosa canina, Tanacetum vulgare and Urtica dioica) on Rat Model of Sporadic Alzheimer's Disease.Beneficial Effect of 7-O-Galloyl-D-sedoheptulose, a Polyphenol Isolated from Corni Fructus, against Diabetes-Induced Alterations in Kidney and Adipose Tissue of Type 2 Diabetic db/db Mice
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P2860
Connecting TNF-alpha signaling pathways to iNOS expression in a mouse model of Alzheimer's disease: relevance for the behavioral and synaptic deficits induced by amyloid beta protein.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh
2007年學術文章
@zh-hant
name
Connecting TNF-alpha signaling ...... duced by amyloid beta protein.
@en
Connecting TNF-alpha signaling ...... duced by amyloid beta protein.
@nl
type
label
Connecting TNF-alpha signaling ...... duced by amyloid beta protein.
@en
Connecting TNF-alpha signaling ...... duced by amyloid beta protein.
@nl
prefLabel
Connecting TNF-alpha signaling ...... duced by amyloid beta protein.
@en
Connecting TNF-alpha signaling ...... duced by amyloid beta protein.
@nl
P2093
P50
P1476
Connecting TNF-alpha signaling ...... nduced by amyloid beta protein
@en
P2093
Filipe S Duarte
Gabriella Di Giunta
Giselle F Passos
Pablo Pandolfo
Reinaldo N Takahashi
P304
P356
10.1523/JNEUROSCI.5047-06.2007
P407
P50
P577
2007-05-01T00:00:00Z