Cerebellar deficits and hyperactivity in mice lacking Smad4. - Wikidata - awgldk - TriplyDB

Cerebellar deficits and hyperactivity in mice lacking Smad4.

Cerebellar deficits and hyperactivity in mice lacking Smad4.

http://www.wikidata.org/entity/Q48236427

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Effects of Transforming Growth Factor Beta 1 in Cerebellar Development: Role in Synapse Formation Loss of Usp9x disrupts cortical architecture, hippocampal development and TGFβ-mediated axonogenesis The DAF-7/TGF-β signaling pathway regulates abundance of the Caenorhabditis elegans glutamate receptor GLR-1 Forebrain-Specific Loss of BMPRII in Mice Reduces Anxiety and Increases Object Exploration.S-SCAM, a rare copy number variation gene, induces schizophrenia-related endophenotypes in transgenic mouse model.Growth factors in synaptic function.Smad7 regulates the adult neural stem/progenitor cell pool in a transforming growth factor beta- and bone morphogenetic protein-independent manner Molecular profiles of pyramidal neurons in the superior temporal cortex in schizophrenia Bone Morphogenetic Protein (BMP) signaling in development and human diseases Induction of intrahepatic cholangiocellular carcinoma by liver-specific disruption of Smad4 and Pten in mice.NT-3 facilitates hippocampal plasticity and learning and memory by regulating neurogenesis Smad2 protein disruption in the central nervous system leads to aberrant cerebellar development and early postnatal ataxia in mice Chronically increased transforming growth factor-beta1 strongly inhibits hippocampal neurogenesis in aged mice Smad4 regulates ureteral smooth muscle cell differentiation during mouse embryogenesis Common partner Smad-independent canonical bone morphogenetic protein signaling in the specification process of the anterior rhombic lip during cerebellum development.Myhre syndrome.BMP/Smad signaling and embryonic cerebellum development: stem cell specification and heterogeneity of anterior rhombic lip.TGF-β Family Signaling in Neural and Neuronal Differentiation, Development, and Function.SMAD4 is essential for generating subtypes of neurons during cerebellar development.Differential effects of BMP signaling on parvalbumin and somatostatin interneuron differentiation.Zfp423/ZNF423 regulates cell cycle progression, the mode of cell division and the DNA-damage response in Purkinje neuron progenitors.Astrocyte transforming growth factor beta 1 promotes inhibitory synapse formation via CaM kinase II signaling.SMAD4 Defect Causes Auditory Neuropathy Via Specialized Disruption of Cochlear Ribbon Synapses in Mice.

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P2860

Cerebellar deficits and hyperactivity in mice lacking Smad4.

http://www.wikidata.org/entity/Q48236427

description

2003 nî lūn-bûn

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2003年の論文

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

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2003年学术文章

@zh-my

2003年学术文章

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2003年學術文章

@yue

2003年學術文章

@zh-hant

name

Cerebellar deficits and hyperactivity in mice lacking Smad4.

@en

Cerebellar deficits and hyperactivity in mice lacking Smad4.

@nl

type

label

Cerebellar deficits and hyperactivity in mice lacking Smad4.

@en

Cerebellar deficits and hyperactivity in mice lacking Smad4.

@nl

prefLabel

Cerebellar deficits and hyperactivity in mice lacking Smad4.

@en

Cerebellar deficits and hyperactivity in mice lacking Smad4.

@nl

P1433

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P1433

Journal of Biological Chemistry

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Cerebellar deficits and hyperactivity in mice lacking Smad4.

@en

P2093

Bai Lu

http://www.w3.org/2001/XMLSchema#string

Chu-Xia Deng

http://www.w3.org/2001/XMLSchema#string

Dan Li

http://www.w3.org/2001/XMLSchema#string

Kazuhiro Shimazu

http://www.w3.org/2001/XMLSchema#string

Kazuko Sakata

http://www.w3.org/2001/XMLSchema#string

Mingrui Zhao

http://www.w3.org/2001/XMLSchema#string

Yong-Xing Zhou

http://www.w3.org/2001/XMLSchema#string

P2860

TGF-beta signal transduction

MADR2 is a substrate of the TGFbeta receptor and its phosphorylation is required for nuclear accumulation and signaling

TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4

MADR2 maps to 18q21 and encodes a TGFbeta-regulated MAD-related protein that is functionally mutated in colorectal carcinoma

Receptor-associated Mad homologues synergize as effectors of the TGF-beta response

Smad6 inhibits BMP/Smad1 signaling by specifically competing with the Smad4 tumor suppressor

Generalized lacZ expression with the ROSA26 Cre reporter strain

Signal transduction by the TGF-beta superfamily

The MAD-related protein Smad7 associates with the TGFbeta receptor and functions as an antagonist of TGFbeta signaling

Identification of Smad7, a TGFbeta-inducible antagonist of TGF-beta signalling

P304

42313-42320

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scholarly article

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10.1074/JBC.M308287200

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43

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278

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2003-08-01T00:00:00Z

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12896967

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