The PRKAG2 gene and hypertrophic cardiomyopathy: an energetically imbalanced relationship.

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The PRKAG2 gene and hypertrophic cardiomyopathy: an energetically imbalanced relationship.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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The PRKAG2 gene and hypertroph ...... cally imbalanced relationship.

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The PRKAG2 gene and hypertroph ...... cally imbalanced relationship.

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type

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The PRKAG2 gene and hypertroph ...... cally imbalanced relationship.

@en

The PRKAG2 gene and hypertroph ...... cally imbalanced relationship.

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The PRKAG2 gene and hypertroph ...... cally imbalanced relationship.

@en

The PRKAG2 gene and hypertroph ...... cally imbalanced relationship.

@nl

P1476

The PRKAG2 gene and hypertroph ...... cally imbalanced relationship.

@en

P2093

Martina Calore

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P2860

Metformin and phenformin activate AMP-activated protein kinase in the heart by increasing cytosolic AMP concentration

Mutations in the gamma(2) subunit of AMP-activated protein kinase cause familial hypertrophic cardiomyopathy: evidence for the central role of energy compromise in disease pathogenesis

Insulin-dependent stimulation of protein synthesis by phosphorylation of a regulator of 5'-cap function

Mutations in the gene for cardiac myosin-binding protein C and late-onset familial hypertrophic cardiomyopathy

Characterization of the AMP-activated protein kinase kinase from rat liver and identification of threonine 172 as the major site at which it phosphorylates AMP-activated protein kinase

Hypertrophic cardiomyopathy.

Aberrant activation of AMP-activated protein kinase remodels metabolic network in favor of cardiac glycogen storage.

AMP-activated protein kinase: the energy charge hypothesis revisited.

Functional analysis of mutations in the gamma 2 subunit of AMP-activated protein kinase associated with cardiac hypertrophy and Wolff-Parkinson-White syndrome.

Transgenic mouse model of ventricular preexcitation and atrioventricular reentrant tachycardia induced by an AMP-activated protein kinase loss-of-function mutation responsible for Wolff-Parkinson-White syndrome.

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H248-H250

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scholarly article

editorial

P356

10.1152/AJPHEART.00316.2017

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313

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M Calore

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2017-06-16T00:00:00Z

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28626079

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