Microglial/macrophage accumulation during cuprizone-induced demyelination in C57BL/6 mice.
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Oligodendrogenesis in the normal and pathological central nervous systemInhibition of p53 transcriptional activity: a potential target for future development of therapeutic strategies for primary demyelinationInfection as an Environmental Trigger of Multiple Sclerosis Disease ExacerbationDeletion of Jun proteins in adult oligodendrocytes does not perturb cell survival, or myelin maintenance in vivoTargeted ablation of oligodendrocytes triggers axonal damageThe Gas6/TAM System and Multiple SclerosisImmune Surveillance of the CNS following Infection and InjuryIntravenous transplantation of mouse embryonic stem cells attenuates demyelination in an ICR outbred mouse model of demyelinating diseasesPolymorphisms in the receptor tyrosine kinase MERTK gene are associated with multiple sclerosis susceptibilityDownregulation of oligodendrocyte transcripts is associated with impaired prefrontal cortex function in rats.Rostrocaudal analysis of corpus callosum demyelination and axon damage across disease stages refines diffusion tensor imaging correlations with pathological features.Inefficient clearance of myelin debris by microglia impairs remyelinating processesQuantitative MRI and ultrastructural examination of the cuprizone mouse model of demyelination.Histological correlation of diffusional kurtosis and white matter modeling metrics in cuprizone-induced corpus callosum demyelination.Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microgliaDevelopment and Pre-Clinical Evaluation of Recombinant Human Myelin Basic Protein Nano Therapeutic Vaccine in Experimental Autoimmune Encephalomyelitis Mice Animal Model.TGFβ signaling regulates the timing of CNS myelination by modulating oligodendrocyte progenitor cell cycle exit through SMAD3/4/FoxO1/Sp1.IkappaB kinase 2 determines oligodendrocyte loss by non-cell-autonomous activation of NF-kappaB in the central nervous systemThe pathogenesis of murine coronavirus infection of the central nervous system.GAS6 enhances repair following cuprizone-induced demyelination.N-cadherin promotes recruitment and migration of neural progenitor cells from the SVZ neural stem cell niche into demyelinated lesions.17beta-estradiol protects male mice from cuprizone-induced demyelination and oligodendrocyte loss.Down-regulation of Shadoo in prion infections traces a pre-clinical event inversely related to PrP(Sc) accumulation.Quantification of increased cellularity during inflammatory demyelination.Inhibiting poly(ADP-ribose) polymerase: a potential therapy against oligodendrocyte death.CNS-specific expression of C3a and C5a exacerbate demyelination severity in the cuprizone model.Neurobiological effects of sphingosine 1-phosphate receptor modulation in the cuprizone model.Critical role for protein tyrosine phosphatase SHP-1 in controlling infection of central nervous system glia and demyelination by Theiler's murine encephalomyelitis virusTime-dependent changes in the brain arachidonic acid cascade during cuprizone-induced demyelination and remyelination.Inhibition of 5-lipoxygenase activity in mice during cuprizone-induced demyelination attenuates neuroinflammation, motor dysfunction and axonal damageDeleterious role of IFNgamma in a toxic model of central nervous system demyelinationOligodendrocytes and progenitors become progressively depleted within chronically demyelinated lesionsGenetic programs and responses of neural stem/progenitor cells during demyelination: potential insights into repair mechanisms in multiple sclerosis.Demyelination and remyelination in anatomically distinct regions of the corpus callosum following cuprizone intoxicationIntravenous administration of human embryonic stem cell-derived neural precursor cells attenuates cuprizone-induced central nervous system (CNS) demyelination.Microglial recruitment, activation, and proliferation in response to primary demyelination.Diffusion kurtosis imaging probes cortical alterations and white matter pathology following cuprizone induced demyelination and spontaneous remyelinationFibroblast growth factor signaling in oligodendrocyte-lineage cells facilitates recovery of chronically demyelinated lesions but is redundant in acute lesions.Spatio-Temporal Patterns of Demyelination and Remyelination in the Cuprizone Mouse ModelReduced astrocytic NF-κB activation by laquinimod protects from cuprizone-induced demyelination.
P2860
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P2860
Microglial/macrophage accumulation during cuprizone-induced demyelination in C57BL/6 mice.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh
1998年學術文章
@zh-hant
name
Microglial/macrophage accumula ...... demyelination in C57BL/6 mice.
@en
Microglial/macrophage accumula ...... demyelination in C57BL/6 mice.
@nl
type
label
Microglial/macrophage accumula ...... demyelination in C57BL/6 mice.
@en
Microglial/macrophage accumula ...... demyelination in C57BL/6 mice.
@nl
prefLabel
Microglial/macrophage accumula ...... demyelination in C57BL/6 mice.
@en
Microglial/macrophage accumula ...... demyelination in C57BL/6 mice.
@nl
P2093
P1476
Microglial/macrophage accumula ...... demyelination in C57BL/6 mice.
@en
P2093
P356
10.1016/S0165-5728(98)00168-4
P577
1998-12-01T00:00:00Z