Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5. - Wikidata - awgldk - TriplyDB

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

http://www.wikidata.org/entity/Q48312558

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TRP channels coordinate ion signalling in astroglia Ultrasound modulates ion channel currents.Voltage-gated sodium channel Nav 1.5 contributes to astrogliosis in an in vitro model of glial injury via reverse Na+ /Ca2+ exchange.Sodium MRI of multiple sclerosis.Nonconventional MRI and microstructural cerebral changes in multiple sclerosis.Multiple Nav1.5 isoforms are functionally expressed in the brain and present distinct expression patterns compared with cardiac Nav1.5.Dynamics of sodium channel Nav1.5 expression in astrocytes in mouse models of multiple sclerosis 11C-acetate PET imaging in patients with multiple sclerosis.Regulation of neurovascular coupling in autoimmunity to water and ion channels Astrocyte response to IFN-γ limits IL-6-mediated microglia activation and progressive autoimmune encephalomyelitis.Astrogliosis during acute and chronic cuprizone demyelination and implications for remyelination Activity of NaV1.2 promotes neurodegeneration in an animal model of multiple sclerosis.hESC-derived Olig2+ progenitors generate a subtype of astroglia with protective effects against ischaemic brain injury.Blinders, phenotype, and fashionable genetic analysis: a critical examination of the current state of epilepsy genetic studies.Sustained-release fampridine and the role of ion channel dysfunction in multiple sclerosis.Polyphenols: multipotent therapeutic agents in neurodegenerative diseases.Sodium channels in astroglia and microglia.Astrocytes in multiple sclerosis.Voltage-gated sodium channel as a target for metastatic risk reduction with re-purposed drugs.Human astrocytes in the diseased brain.Targeting ion channels for the treatment of autoimmune neuroinflammation.Strength-duration time constant in peripheral nerve: no abnormality in multiple sclerosis.Topography of brain sodium accumulation in progressive multiple sclerosis.Nav1.5 sodium channels in macrophages in multiple sclerosis lesions.Molecular expression of multiple Nav1.5 splice variants in the frontal lobe of the human brain.Distribution and function of voltage-gated sodium channels in the nervous system.Astrocyte calcium signal and gliotransmission in human brain tissue.Increased expression of hyperpolarization-activated cyclic nucleotide-gated (HCN) channels in reactive astrocytes following ischemia.Brain intra- and extracellular sodium concentration in multiple sclerosis: a 7 T MRI study.Physiology of Astroglia.

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P2860

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

http://www.wikidata.org/entity/Q48312558

description

2010 nî lūn-bûn

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2010年の論文

@ja

2010年学术文章

@wuu

2010年学术文章

@zh

2010年学术文章

@zh-cn

2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh-hant

name

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

@en

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

@nl

type

label

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

@en

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

@nl

prefLabel

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

@en

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

@nl

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P1476

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5.

@en

P2093

Jia Newcombe

http://www.w3.org/2001/XMLSchema#string

Joel A Black

http://www.w3.org/2001/XMLSchema#string

Stephen G Waxman

http://www.w3.org/2001/XMLSchema#string

P2860

State-dependent trapping of flecainide in the cardiac sodium channel

Astrocytes in multiple sclerosis: a product of their environment

International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels

Involvement of Kv1.1 and Nav1.5 in proliferation of gastric epithelial cells

Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons

A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons

Sodium MR imaging of acute and subacute stroke for assessment of tissue viability.

Activation of the inducible form of nitric oxide synthase in the brains of patients with multiple sclerosis.

The neonatal splice variant of Nav1.5 potentiates in vitro invasive behaviour of MDA-MB-231 human breast cancer cells.

Molecular cloning of a putative tetrodotoxin-resistant rat heart Na+ channel isoform.

P304

835-846

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scholarly article

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10.1093/BRAIN/AWQ003

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P407

P433

Pt 3

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133

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2010-02-10T00:00:00Z

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41421841

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20147455

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P921

multiple sclerosis