New functions in translation termination uncovered for NMD factor UPF3B.

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New functions in translation termination uncovered for NMD factor UPF3B.

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2017 nî lūn-bûn

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2017年学术文章

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2017年学术文章

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2017年学术文章

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New functions in translation termination uncovered for NMD factor UPF3B.

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New functions in translation termination uncovered for NMD factor UPF3B.

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New functions in translation termination uncovered for NMD factor UPF3B.

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New functions in translation termination uncovered for NMD factor UPF3B.

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New functions in translation termination uncovered for NMD factor UPF3B.

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New functions in translation termination uncovered for NMD factor UPF3B.

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P1476

New functions in translation termination uncovered for NMD factor UPF3B.

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P2093

Evangelos D Karousis

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P2860

Binding of a novel SMG-1-Upf1-eRF1-eRF3 complex (SURF) to the exon junction complex triggers Upf1 phosphorylation and nonsense-mediated mRNA decay

Interactions between UPF1, eRFs, PABP and the exon junction complex suggest an integrated model for mammalian NMD pathways

Upf1 ATPase-dependent mRNP disassembly is required for completion of nonsense- mediated mRNA decay

A competition between stimulators and antagonists of Upf complex recruitment governs human nonsense-mediated mRNA decay

Recycling of eukaryotic posttermination ribosomal complexes.

Full UPF3B function is critical for neuronal differentiation of neural stem cells.

Interaction of PABPC1 with the translation initiation complex is critical to the NMD resistance of AUG-proximal nonsense mutations.

PABP enhances release factor recruitment and stop codon recognition during translation termination.

Dual function of UPF3B in early and late translation termination.

A faux 3'-UTR promotes aberrant termination and triggers nonsense-mediated mRNA decay.

P304

2928-2930

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P31

scholarly article

P356

10.15252/EMBJ.201798063

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P407

English

P433

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P478

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P50

Oliver Mühlemann

P577

2017-09-21T00:00:00Z

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P698

28935685

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P932

5641677

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