Breathing dysfunctions associated with impaired control of postinspiratory activity in Mecp2-/y knockout mice. - Wikidata - awgldk - TriplyDB

Breathing dysfunctions associated with impaired control of postinspiratory activity in Mecp2-/y knockout mice.

Breathing dysfunctions associated with impaired control of postinspiratory activity in Mecp2-/y knockout mice.

http://www.wikidata.org/entity/Q48318692

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Pontine mechanisms of respiratory control GABA and glutamate pathways are spatially and developmentally affected in the brain of Mecp2-deficient mice A partial loss of function allele of methyl-CpG-binding protein 2 predicts a human neurodevelopmental syndrome A BDNF loop-domain mimetic acutely reverses spontaneous apneas and respiratory abnormalities during behavioral arousal in a mouse model of Rett syndrome.Brain activity mapping in Mecp2 mutant mice reveals functional deficits in forebrain circuits, including key nodes in the default mode network, that are reversed with ketamine treatment.Preclinical research in Rett syndrome: setting the foundation for translational success.Rodent models of sleep apnea.Autonomic breathing abnormalities in Rett syndrome: caregiver perspectives in an international database study.Pinpointing brainstem mechanisms responsible for autonomic dysfunction in Rett syndrome: therapeutic perspectives for 5-HT1A agonists.Effects of glycinergic inhibition failure on respiratory rhythm and pattern generation.Effect of Sarizotan, a 5-HT1a and D2-like receptor agonist, on respiration in three mouse models of Rett syndrome Brainstem respiratory networks: building blocks and microcircuits β2-Adrenergic receptor agonist ameliorates phenotypes and corrects microRNA-mediated IGF1 deficits in a mouse model of Rett syndrome Use-dependent learning and memory of the Hering-Breuer inflation reflex in rats Pontine norepinephrine defects in Mecp2-null mice involve deficient expression of dopamine beta-hydroxylase but not a loss of catecholaminergic neurons.Correction of respiratory disorders in a mouse model of Rett syndrome.The role of serotonin in respiratory function and dysfunction.A selective 5-HT1a receptor agonist improves respiration in a mouse model of Rett syndrome.Respiratory rhythm generation in vivo.Characterization of the MeCP2R168X knockin mouse model for Rett syndrome.Habituation without NMDA Receptor-Dependent Desensitization of Hering-Breuer Apnea Reflex in a Mecp2 Mutant Mouse Model of Rett Syndrome Multiscale fingerprinting of neuronal functional connectivity Network models predict that reduced excitatory fluctuations can give rise to hippocampal network hyper-excitability in MeCP2-null mice The disruption of central CO2 chemosensitivity in a mouse model of Rett syndrome.MeCP2 is critical within HoxB1-derived tissues of mice for normal lifespan Synaptic microcircuit dysfunction in genetic models of neurodevelopmental disorders: focus on Mecp2 and Met.Kölliker–Fuse neurons send collateral projections to multiple hypoxia-activated and nonactivated structures in rat brainstem and spinal cord.Control of breathing by interacting pontine and pulmonary feedback loops.Post-inspiratory discharges are the centrepiece of respiratory disrhythmia in a gene knockout model of Rett syndrome.Decreased Hering-Breuer input-output entrainment in a mouse model of Rett syndrome.Progressive Changes in a Distributed Neural Circuit Underlie Breathing Abnormalities in Mice Lacking MeCP2.Blockade of dorsolateral pontine 5HT1A receptors destabilizes the respiratory rhythm in C57BL6/J wild-type mice.Rett syndrome: recent research progress.Neural control of breathing: insights from genetic mouse models.Breathing dysfunction in Rett syndrome: understanding epigenetic regulation of the respiratory network GlyT2-Dependent Preservation of MECP2-Expression in Inhibitory Neurons Improves Early Respiratory Symptoms but Does Not Rescue Survival in a Mouse Model of Rett Syndrome.Breathing challenges in Rett syndrome: lessons learned from humans and animal models.Breathing disorders in Rett syndrome: progressive neurochemical dysfunction in the respiratory network after birth Pontine respiratory activity involved in inspiratory/expiratory phase transition.Inflammatory lung disease in Rett syndrome.

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Breathing dysfunctions associated with impaired control of postinspiratory activity in Mecp2-/y knockout mice.

http://www.wikidata.org/entity/Q48318692

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2007 nî lūn-bûn

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Breathing dysfunctions associa ...... ity in Mecp2-/y knockout mice.

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Breathing dysfunctions associa ...... ity in Mecp2-/y knockout mice.

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JPHYSIOL.2006.119966

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Breathing dysfunctions associa ...... ity in Mecp2-/y knockout mice.

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Cornelia Brendel

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Diethelm W Richter

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Georg M Stettner

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Jutta Gärtner

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Peter Huppke

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P2860

Epigenetic overlap in autism-spectrum neurodevelopmental disorders: MECP2 deficiency causes reduced expression of UBE3A and GABRB3.

Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2

Reduced cortical activity due to a shift in the balance between excitation and inhibition in a mouse model of Rett syndrome

Molecular genetics of Rett syndrome: when DNA methylation goes unrecognized

A mouse Mecp2-null mutation causes neurological symptoms that mimic Rett syndrome

Learning and memory and synaptic plasticity are impaired in a mouse model of Rett syndrome

Defect in normal developmental increase of the brain biogenic amine concentrations in the mecp2-null mouse

Mecp2 deficiency disrupts norepinephrine and respiratory systems in mice

Hippocampal synaptic plasticity is impaired in the Mecp2-null mouse model of Rett syndrome

Deficiency of methyl-CpG binding protein-2 in CNS neurons results in a Rett-like phenotype in mice

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Pt 3

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Mathias Dutschmann

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