Selective coupling of mu-calpain activation with the NMDA receptor is independent of translocation and autolysis in primary cortical neurons.
about
Fast, non-competitive and reversible inhibition of NMDA-activated currents by 2-BFI confers neuroprotectionCellular interplay between neurons and glia: toward a comprehensive mechanism for excitotoxic neuronal loss in neurodegenerationNeuroprotection by sodium ferulate against glutamate-induced apoptosis is mediated by ERK and PI3 kinase pathways.Postsynaptic density-95 (PSD-95) and calcineurin control the sensitivity of N-methyl-D-aspartate receptors to calpain cleavage in cortical neurons.Switch to glutamate receptor 2-lacking AMPA receptors increases neuronal excitability in hypothalamus and sympathetic drive in hypertension.Mechanistic role of calpains in postischemic neurodegeneration.Nerve injury increases GluA2-lacking AMPA receptor prevalence in spinal cords: functional significance and signaling mechanisms.PARP-1 cleavage fragments: signatures of cell-death proteases in neurodegenerationSpecific proteolysis of the NR2 subunit at multiple sites by calpain.Real-time visualization of cytoplasmic calpain activation and calcium deregulation in acute glutamate excitotoxicity.Calpain activates caspase-3 during UV-induced neuronal death but only calpain is necessary for death.Chlortetracycline and demeclocycline inhibit calpains and protect mouse neurons against glutamate toxicity and cerebral ischemia.Calpain-mediated truncation of dihydropyrimidinase-like 3 protein (DPYSL3) in response to NMDA and H2O2 toxicity.TRPC6 expression in neurons is differentially regulated by NR2A- and NR2B-containing NMDA receptors.Calpain inhibitors confer biochemical, but not electrophysiological, protection against anoxia in rat optic nerves.
P2860
Q21090747-C51A4BCB-2BE0-499D-8D54-B19A2CC3BC6CQ24654460-CB83F0BD-93E4-4E1B-AC7E-7EC453AF1395Q33306762-1384A9BD-2B8E-4691-A451-6E098CC9D7A7Q33806466-4DA44F04-76B6-4C59-B1F0-CB2B8C55BAF7Q36594018-F6016D14-9794-4C32-8874-CBCD5705E51AQ37029897-435C0717-CA8A-416B-8990-41BC71FD6EC9Q37331824-AB662DB0-5EFA-4BCD-A542-5AACA811087BQ37822920-2395F784-BEE0-4054-BFCA-23AFBFF75543Q40781019-8709AA1A-AE15-4CDF-B335-F64F97D4C3EBQ42003134-EB677711-980F-4D76-B09E-062F62263AA9Q44163246-C1CC3BE9-D6A9-44D9-8708-F82AB6CF8B5EQ46642858-5B4C8249-A502-47B3-A375-0AB84438C1FCQ46680604-4B8478B4-8854-4C5B-A39A-0AB6B81B8113Q47809841-6D218FD2-F183-40F8-80E5-C7E7ECB4A572Q51402691-693679FF-1CB7-4658-9F43-2AB0C5523F16
P2860
Selective coupling of mu-calpain activation with the NMDA receptor is independent of translocation and autolysis in primary cortical neurons.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@en
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@nl
type
label
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@en
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@nl
prefLabel
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@en
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@nl
P2093
P2860
P1476
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@en
P2093
P2860
P304
P356
10.1002/(SICI)1097-4547(19981015)54:2<223::AID-JNR10>3.0.CO;2-5
P577
1998-10-01T00:00:00Z