Selective coupling of mu-calpain activation with the NMDA receptor is independent of translocation and autolysis in primary cortical neurons.

Selective coupling of mu-calpain activation with the NMDA receptor is independent of translocation and autolysis in primary cortical neurons.

Item
http://www.wikidata.org/entity/Q48366651
Fast, non-competitive and reversible inhibition of NMDA-activated currents by 2-BFI confers neuroprotectionCellular interplay between neurons and glia: toward a comprehensive mechanism for excitotoxic neuronal loss in neurodegenerationNeuroprotection by sodium ferulate against glutamate-induced apoptosis is mediated by ERK and PI3 kinase pathways.Postsynaptic density-95 (PSD-95) and calcineurin control the sensitivity of N-methyl-D-aspartate receptors to calpain cleavage in cortical neurons.Switch to glutamate receptor 2-lacking AMPA receptors increases neuronal excitability in hypothalamus and sympathetic drive in hypertension.Mechanistic role of calpains in postischemic neurodegeneration.Nerve injury increases GluA2-lacking AMPA receptor prevalence in spinal cords: functional significance and signaling mechanisms.PARP-1 cleavage fragments: signatures of cell-death proteases in neurodegenerationSpecific proteolysis of the NR2 subunit at multiple sites by calpain.Real-time visualization of cytoplasmic calpain activation and calcium deregulation in acute glutamate excitotoxicity.Calpain activates caspase-3 during UV-induced neuronal death but only calpain is necessary for death.Chlortetracycline and demeclocycline inhibit calpains and protect mouse neurons against glutamate toxicity and cerebral ischemia.Calpain-mediated truncation of dihydropyrimidinase-like 3 protein (DPYSL3) in response to NMDA and H2O2 toxicity.TRPC6 expression in neurons is differentially regulated by NR2A- and NR2B-containing NMDA receptors.Calpain inhibitors confer biochemical, but not electrophysiological, protection against anoxia in rat optic nerves.
P2860

Selective coupling of mu-calpain activation with the NMDA receptor is independent of translocation and autolysis in primary cortical neurons.

Item
http://www.wikidata.org/entity/Q48366651
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@en
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@nl
type
label
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@en
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@nl
prefLabel
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@en
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@nl
P1433
P1476
P2093
P2860
P304
P31
P356
P433
P478
P577
P698
P356
P1433
P1476
Selective coupling of mu-calpa ...... s in primary cortical neurons.
@en
P2093
P2860
P304
P31
P356
10.1002/(SICI)1097-4547(19981015)54:2<223::AID-JNR10>3.0.CO;2-5
P433
P478
P577
P698