Chronic ethanol administration alters activity in ventral tegmental area neurons after cessation of withdrawal hyperexcitability.
about
Glial cell line-derived neurotrophic factor mediates the desirable actions of the anti-addiction drug ibogaine against alcohol consumptionAcamprosate: a prototypic neuromodulator in the treatment of alcohol dependenceEthanol action on dopaminergic neurons in the ventral tegmental area: interaction with intrinsic ion channels and neurotransmitter inputs.Decreased sensitivity of NMDA receptors on dopaminergic neurons from the posterior ventral tegmental area following chronic nondependent alcohol consumptionPhasic dopamine release in appetitive behaviors and drug addiction.The neurobiology, clinical efficacy and safety of acamprosate in the treatment of alcohol dependence.Drug dependence as a disorder of neural plasticity: focus on dopamine and glutamate.Voluntary ethanol intake enhances excitatory synaptic strength in the ventral tegmental area.Strain specific synaptic modifications on ventral tegmental area dopamine neurons after ethanol exposure.Neurodevelopmental liabilities in alcohol dependence: central serotonin and dopamine dysfunction.Microinjection of glycine into the ventral tegmental area selectively decreases ethanol consumption.Glutamate-dopamine cotransmission and reward processing in addiction.Motivational Processes Underlying Substance Abuse Disorder.Hyposensitivity to gamma-aminobutyric acid in the ventral tegmental area during alcohol withdrawal: reversal by histone deacetylase inhibitorsSizing up ethanol-induced plasticity: the role of small and large conductance calcium-activated potassium channels.Correlation between ethanol behavioral sensitization and midbrain dopamine neuron reactivity to ethanol.Dopaminergic neurons in the ventral tegmental area of C57BL/6J and DBA/2J mice differ in sensitivity to ethanol excitation.Increased ethanol excitation of dopaminergic neurons of the ventral tegmental area after chronic ethanol treatment.Anhedonia and substance dependence: clinical correlates and treatment options.Enduring effects of chronic ethanol in the CNS: basis for alcoholism.Acetaldehyde increases dopaminergic neuronal activity in the VTA.Withdrawal from intermittent ethanol exposure increases probability of burst firing in VTA neurons in vitro.
P2860
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P2860
Chronic ethanol administration alters activity in ventral tegmental area neurons after cessation of withdrawal hyperexcitability.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Chronic ethanol administration ...... withdrawal hyperexcitability.
@en
Chronic ethanol administration ...... withdrawal hyperexcitability.
@nl
type
label
Chronic ethanol administration ...... withdrawal hyperexcitability.
@en
Chronic ethanol administration ...... withdrawal hyperexcitability.
@nl
prefLabel
Chronic ethanol administration ...... withdrawal hyperexcitability.
@en
Chronic ethanol administration ...... withdrawal hyperexcitability.
@nl
P2093
P1433
P1476
Chronic ethanol administration ...... withdrawal hyperexcitability.
@en
P2093
P304
P356
10.1016/S0006-8993(98)00654-4
P407
P577
1998-08-01T00:00:00Z