Glucocorticoids specifically enhance L-type calcium current amplitude and affect calcium channel subunit expression in the mouse hippocampus.
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Role of Glia in Stress-Induced Enhancement and Impairment of MemoryThe stress hormone corticosterone increases synaptic alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptors via serum- and glucocorticoid-inducible kinase (SGK) regulation of the GDI-Rab4 complexCorticosteroids: way upstreamN-type calcium current, Cav2.2, is enhanced in small-diameter sensory neurons isolated from Nf1+/- mice.Mifepristone pretreatment reduces ethanol withdrawal severity in vivo.Impact of corticosterone treatment on spontaneous seizure frequency and epileptiform activity in mice with chronic epilepsy.Elucidating the Complex Interactions between Stress and Epileptogenic PathwaysDelayed effects of corticosterone on slow after-hyperpolarization potentials in mouse hippocampal versus prefrontal cortical pyramidal neuronsGlucocorticoid actions on synapses, circuits, and behavior: implications for the energetics of stress.Acute restraint stress enhances hippocampal endocannabinoid function via glucocorticoid receptor activation.Glucocorticoid receptors recruit the CaMKIIα-BDNF-CREB pathways to mediate memory consolidation.Dissociation between rat hippocampal CA1 and dentate gyrus cells in their response to corticosterone: effects on calcium channel protein and current.Puberty, steroids and GABA(A) receptor plasticity.Stress and glucocorticoid receptor-dependent mechanisms in long-term memory: from adaptive responses to psychopathologiesSteroid modulation of hippocampal plasticity: switching between cognitive and emotional memories.Repeated social defeat stress enhances glutamatergic synaptic plasticity in the VTA and cocaine place conditioning.Differential effects of corticosterone on the slow afterhyperpolarization in the basolateral amygdala and CA1 region: possible role of calcium channel subunits.Outgroup emotion processing in the vACC is modulated by childhood trauma and CACNA1C risk variant.Role of CACNA1C gene polymorphisms and protein expressions in the pathogenesis of schizophrenia: a case-control study in a Chinese population.Corticosterone targets distinct steps of synaptic transmission via concentration specific activation of mineralocorticoid and glucocorticoid receptors.NADPH oxidase-mediated Rac1 GTP activity is necessary for nongenomic actions of the mineralocorticoid receptor in the CA1 region of the rat hippocampus.
P2860
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P2860
Glucocorticoids specifically enhance L-type calcium current amplitude and affect calcium channel subunit expression in the mouse hippocampus.
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name
Glucocorticoids specifically e ...... sion in the mouse hippocampus.
@en
Glucocorticoids specifically e ...... sion in the mouse hippocampus.
@nl
type
label
Glucocorticoids specifically e ...... sion in the mouse hippocampus.
@en
Glucocorticoids specifically e ...... sion in the mouse hippocampus.
@nl
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Glucocorticoids specifically e ...... sion in the mouse hippocampus.
@en
Glucocorticoids specifically e ...... sion in the mouse hippocampus.
@nl
P2093
P2860
P356
P1476
Glucocorticoids specifically e ...... sion in the mouse hippocampus.
@en
P2093
August Benjamin Smit
Marian Joëls
Yongjun Qin
P2860
P356
10.1152/JN.00821.2006
P407
P577
2006-10-04T00:00:00Z