Time-course of glial changes in the hyperhomocysteinemia model of vascular cognitive impairment and dementia (VCID).

about

Hyperhomocysteinemia-Induced Gene Expression Changes in the Cell Types of the Brain.An emerging role of astrocytes in vascular contributions to cognitive impairment and dementia.Hyperhomocysteinemia as a Risk Factor for Vascular Contributions to Cognitive Impairment and Dementia

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P2860

Time-course of glial changes in the hyperhomocysteinemia model of vascular cognitive impairment and dementia (VCID).

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2016 nî lūn-bûn

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2016年の論文

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年学术文章

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2016年學術文章

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name

Time-course of glial changes i ...... mpairment and dementia (VCID).

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Time-course of glial changes i ...... nitive impairment and dementia

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type

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Time-course of glial changes i ...... mpairment and dementia (VCID).

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Time-course of glial changes i ...... nitive impairment and dementia

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Time-course of glial changes i ...... mpairment and dementia (VCID).

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Time-course of glial changes i ...... nitive impairment and dementia

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P1476

Time-course of glial changes i ...... mpairment and dementia (VCID).

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P2093

Abigail Woolums

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Brittani R Price

http://www.w3.org/2001/XMLSchema#string

Christopher M Norris

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Donna M Wilcock

http://www.w3.org/2001/XMLSchema#string

Erica M Weekman

http://www.w3.org/2001/XMLSchema#string

Jennifer L Gooch

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Tiffany L Sudduth

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P2860

Passive immunotherapy against Abeta in aged APP-transgenic mice reverses cognitive deficits and depletes parenchymal amyloid deposits in spite of increased vascular amyloid and microhemorrhage

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Vascular contributions to cognitive impairment and dementia including Alzheimer's disease

Homocysteine and cardiovascular disease

B-vitamin deficiency causes hyperhomocysteinemia and vascular cognitive impairment in mice.

Induction of hyperhomocysteinemia models vascular dementia by induction of cerebral microhemorrhages and neuroinflammation.

IL-17A is implicated in lipopolysaccharide-induced neuroinflammation and cognitive impairment in aged rats via microglial activation.

Alpha-syntrophin deletion removes the perivascular but not endothelial pool of aquaporin-4 at the blood-brain barrier and delays the development of brain edema in an experimental model of acute hyponatremia.

p38gamma MAPK cooperates with c-Jun in trans-activating matrix metalloproteinase 9.

β-amyloid deposition is shifted to the vasculature and memory impairment is exacerbated when hyperhomocysteinemia is induced in APP/PS1 transgenic mice.

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42-51

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scholarly article

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10.1016/J.NEUROSCIENCE.2016.11.024

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English

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341

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2016-11-25T00:00:00Z

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27890830

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5911565

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Time-course of glial changes in the hyperhomocysteinemia model of vascular cognitive impairment and dementia (VCID).

about

P2860

P2860

Time-course of glial changes in the hyperhomocysteinemia model of vascular cognitive impairment and dementia (VCID).

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P577

P698

P932