Quantitative relationship between inhibition of respiratory complexes and formation of reactive oxygen species in isolated nerve terminals.
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The role of Nox2-derived ROS in the development of cognitive impairment after sepsisOxidative stress in schizophreniaThree toxic gases meet in the mitochondriaOxidative stress as activators of sensory nerves for coughMitochondrial dysfunction in schizophrenia: evidence for compromised brain metabolism and oxidative stressComplex III releases superoxide to both sides of the inner mitochondrial membraneThe mitochondrion: a perpetrator of acquired hearing loss.Sensory nerve terminal mitochondrial dysfunction induces hyperexcitability in airway nociceptors via protein kinase CDopamine quinone modifies and decreases the abundance of the mitochondrial selenoprotein glutathione peroxidase 4.Age related changes in NAD+ metabolism oxidative stress and Sirt1 activity in wistar ratsAlpha-ketoglutarate dehydrogenase: a target and generator of oxidative stressCalcium and mitochondrial reactive oxygen species generation: how to read the facts.Mitochondria: key players in the neurotoxic effects of amphetamines.Optical and pharmacological tools to investigate the role of mitochondria during oxidative stress and neurodegeneration.Aging-related decrease of human ASC angiogenic potential is reversed by hypoxia preconditioning through ROS production.AIF, reactive oxygen species, and neurodegeneration: a "complex" problemInteraction of nitric oxide with a functional model of cytochrome c oxidase.The Psi(m) depolarization that accompanies mitochondrial Ca2+ uptake is greater in mutant SOD1 than in wild-type mouse motor terminals.Complex I is rate-limiting for oxygen consumption in the nerve terminalInvolvement of ROS in BBB dysfunction.Reactive oxygen species regulation by AIF- and complex I-depleted brain mitochondria.Conditional knockout of Mn-SOD targeted to type IIB skeletal muscle fibers increases oxidative stress and is sufficient to alter aerobic exercise capacity.MT-CYB mutations in hypertrophic cardiomyopathy.Mechanisms associated with mitochondrial-generated reactive oxygen species in cancer.Association of glycogen synthase kinase-3β with Parkinson's disease (review).Natural antioxidants in hybrids for the treatment of neurodegenerative diseases: a successful strategy?Role of Mitochondria in Methamphetamine-Induced Dopaminergic Neurotoxicity: Involvement in Oxidative Stress, Neuroinflammation, and Pro-apoptosis-A Review.Methylenedioxymethamphetamine inhibits mitochondrial complex I activity in mice: a possible mechanism underlying neurotoxicity.Status Epilepticus in Immature Rats Is Associated with Oxidative Stress and Mitochondrial Dysfunction.Complex I and complex III inhibition specifically increase cytosolic hydrogen peroxide levels without inducing oxidative stress in HEK293 cells.Energy failure in astrocytes increases the vulnerability of neurons to spreading depression.Protein phosphatases but not reactive oxygen species play functional role in acute amphetamine-mediated dopamine release.Intracellular dopamine oxidation mediates rotenone-induced apoptosis in PC12 cells.'SNO'-Storms Compromise Protein Activity and Mitochondrial Metabolism in Neurodegenerative Disorders.Guidelines on experimental methods to assess mitochondrial dysfunction in cellular models of neurodegenerative diseases.Diverse fission yeast genes required for responding to oxidative and metal stress: Comparative analysis of glutathione-related and other defense gene deletions.Implications of enzyme deficiencies on mitochondrial energy metabolism and reactive oxygen species formation of neurons involved in rotenone-induced Parkinson's disease: a model-based analysis.Effects of partial inhibition of respiratory complex I on H2O 2 production by isolated brain mitochondria in different respiratory states.Agaricus blazei extract attenuates rotenone-induced apoptosis through its mitochondrial protective and antioxidant properties in SH-SY5Y neuroblastoma cells.Mitochondria in Excitatory and Inhibitory Synapses have Similar Susceptibility to Amyloid-β Peptides Modeling Alzheimer's Disease.
P2860
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P2860
Quantitative relationship between inhibition of respiratory complexes and formation of reactive oxygen species in isolated nerve terminals.
description
2003 nî lūn-bûn
@nan
2003年の論文
@ja
2003年学术文章
@wuu
2003年学术文章
@zh
2003年学术文章
@zh-cn
2003年学术文章
@zh-hans
2003年学术文章
@zh-my
2003年学术文章
@zh-sg
2003年學術文章
@yue
2003年學術文章
@zh-hant
name
Quantitative relationship betw ...... s in isolated nerve terminals.
@en
Quantitative relationship betw ...... s in isolated nerve terminals.
@nl
type
label
Quantitative relationship betw ...... s in isolated nerve terminals.
@en
Quantitative relationship betw ...... s in isolated nerve terminals.
@nl
prefLabel
Quantitative relationship betw ...... s in isolated nerve terminals.
@en
Quantitative relationship betw ...... s in isolated nerve terminals.
@nl
P2860
P1476
Quantitative relationship betw ...... s in isolated nerve terminals.
@en
P2093
Ildikó Sipos
Laszlo Tretter
P2860
P304
P356
10.1046/J.1471-4159.2003.01513.X
P407
P577
2003-01-01T00:00:00Z