pSTAT1, pSTAT3, and T-bet expression in peripheral blood mononuclear cells from relapsing-remitting multiple sclerosis patients correlates with disease activity.
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Regulation of translocator protein 18 kDa (TSPO) expression in health and disease statesAPC-derived cytokines and T cell polarization in autoimmune inflammationSarcoidosis: Immunopathogenesis and Immunological MarkersRTL551 treatment of EAE reduces CD226 and T-bet+ CD4 T cells in periphery and prevents infiltration of T-bet+ IL-17, IFN-γ producing T cells into CNSSOCS1 and SOCS3 in the control of CNS immunitySHP-1 deficiency and increased inflammatory gene expression in PBMCs of multiple sclerosis patients.Involvement of the janus kinase/signal transducer and activator of transcription signaling pathway in multiple sclerosis and the animal model of experimental autoimmune encephalomyelitis.Gammaherpesvirus latency accentuates EAE pathogenesis: relevance to Epstein-Barr virus and multiple sclerosis.More than just a T-box: the role of T-bet as a possible biomarker and therapeutic target in autoimmune diseases.Seeking balance: Potentiation and inhibition of multiple sclerosis autoimmune responses by IL-6 and IL-10.Interleukin-33 upregulation in peripheral leukocytes and CNS of multiple sclerosis patients.Quantitative differences in the immunomodulatory effects of Rebif and Avonex in IFN-β 1a treated multiple sclerosis patientsPromoter-specific induction of the phosphatase SHP-1 by viral infection and cytokines in CNS gliaActivation of the JAK-STAT Signaling Pathway after In Vitro Stimulation with IFNß in Multiple Sclerosis Patients According to the Therapeutic Response to IFNß.Next-generation sequencing identifies microRNAs that associate with pathogenic autoimmune neuroinflammation in ratsModulation of macrophage infiltration and inflammatory activity by the phosphatase SHP-1 in virus-induced demyelinating diseaseMacrophages of multiple sclerosis patients display deficient SHP-1 expression and enhanced inflammatory phenotypeHypothesis: sarcoidosis is a STAT1-mediated disease.Highly polarized Th17 cells induce EAE via a T-bet independent mechanism.Interferon-beta treatment in multiple sclerosis attenuates inflammatory gene expression through inducible activity of the phosphatase SHP-1.yuDetecting the percent of peripheral blood mononuclear cells displaying p-STAT-3 in malignant glioma patients.Regulation of effector function of CNS autoreactive CD4 T cells through inhibitory receptors and IL-7Rα.Glatiramer acetate attenuates the activation of CD4+ T cells by modulating STAT1 and -3 signaling in glia.Therapeutic efficacy of suppressing the Jak/STAT pathway in multiple models of experimental autoimmune encephalomyelitis.Opportunities for Translation from the Bench: Therapeutic Intervention of the JAK/STAT Pathway in Neuroinflammatory Diseases.Lentiviral vector-mediated gene transfer and RNA silencing technology in neuronal dysfunctions.T-bet in disease.Noradrenaline transmission reducing drugs may protect against a broad range of diseases.Upregulation of T-cell Immunoglobulin and Mucin-Domain Containing-3 (Tim-3) in Monocytes/Macrophages Associates with Gastric Cancer Progression.Smad7 in T cells drives T helper 1 responses in multiple sclerosis and experimental autoimmune encephalomyelitis.Heterogeneity in multiple sclerosis: scratching the surface of a complex disease.Increased promoter methylation of the immune regulatory gene SHP-1 in leukocytes of multiple sclerosis subjects.Increased CXCL10 expression in MS MSCs and monocytes is unaffected by AHSCT.In active relapsing-remitting multiple sclerosis, effector T cell resistance to adaptive T(regs) involves IL-6-mediated signaling.Cerebral gene expression of superantigen encephalitis in the lewis rat induced by staphylococcal enterotoxin a.Absence of Notch1 in murine myeloid cells attenuates the development of experimental autoimmune encephalomyelitis by affecting Th1 and Th17 priming.Molecular Genetic and Epigenetic Basis of Multiple Sclerosis.Resveratrol Ameliorates Dysregulation of Th1, Th2, Th17, and T Regulatory Cell-Related Transcription Factor Signaling in a BTBR T + tf/J Mouse Model of Autism.Dysregulation of Th1, Th2, Th17, and T regulatory cell-related transcription factor signaling in children with autism.The monocyte transcriptome during pregnancy in multiple sclerosis: prominent expression of the Fc-receptor CD64.
P2860
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P2860
pSTAT1, pSTAT3, and T-bet expression in peripheral blood mononuclear cells from relapsing-remitting multiple sclerosis patients correlates with disease activity.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
pSTAT1, pSTAT3, and T-bet expr ...... relates with disease activity.
@en
pSTAT1, pSTAT3, and T-bet expr ...... relates with disease activity.
@nl
type
label
pSTAT1, pSTAT3, and T-bet expr ...... relates with disease activity.
@en
pSTAT1, pSTAT3, and T-bet expr ...... relates with disease activity.
@nl
prefLabel
pSTAT1, pSTAT3, and T-bet expr ...... relates with disease activity.
@en
pSTAT1, pSTAT3, and T-bet expr ...... relates with disease activity.
@nl
P2093
P2860
P356
P1476
pSTAT1, pSTAT3, and T-bet expr ...... relates with disease activity.
@en
P2093
Agata Katia Patanella
Anna Paola Batocchi
Cristina Sancricca
Francesco Angelucci
Giovanni Frisullo
Marcella Caggiula
Massimiliano Mirabella
Pietro Attilio Tonali
Viviana Nociti
P2860
P304
P356
10.1002/JNR.20995
P577
2006-10-01T00:00:00Z