Mitochondrial dysfunction in oocytes of obese mothers: transmission to offspring and reversal by pharmacological endoplasmic reticulum stress inhibitors.
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The Programming Power of the PlacentaModeling dietary influences on offspring metabolic programming in Drosophila melanogasterAdiponectin stimulates lipid metabolism via AMPK in rabbit blastocysts.Endoplasmic Reticulum Stress and Homeostasis in Reproductive Physiology and Pathology.Joint MiRNA/mRNA expression profiling reveals changes consistent with development of dysfunctional corpus luteum after weight gain.Differing roles of pyruvate dehydrogenase kinases during mouse oocyte maturation.Research Priorities for Fertility and Conception Research as Identified by Multidisciplinary Health Care Practitioners and Researchers.The GTPase SPAG-1 orchestrates meiotic program by dictating meiotic resumption and cytoskeleton architecture in mouse oocytesMaternal obesity reduces oxidative capacity in fetal skeletal muscle of Japanese macaques.Maternal obesity programs mitochondrial and lipid metabolism gene expression in infant umbilical vein endothelial cells.Embryos, DOHaD and David Barker.The early origins of obesity and insulin resistance: timing, programming and mechanisms.Epigenetic germline inheritance in mammals: Looking to the past to understand the future.Drosophila melanogaster: An emerging model of transgenerational effects of maternal obesity.Transmission of Metabolic Dysfunction Across Generations.Next Generation Sequencing-Based Comprehensive Chromosome Screening in Mouse Polar Bodies, Oocytes, and Embryos.Epigenetic Mechanisms of Transmission of Metabolic Disease across Generations.Intergenerational Transmission of Maternal Childhood Maltreatment Exposure: Implications for Fetal Brain Development.Activation of Endoplasmic Reticulum Stress in Granulosa Cells from Patients with Polycystic Ovary Syndrome Contributes to Ovarian Fibrosis.High-sucrose-induced maternal obesity disrupts ovarian function and decreases fertility in Drosophila melanogaster.A pre-in vitro maturation medium containing cumulus oocyte complex ligand-receptor signaling molecules maintains meiotic arrest, supports the cumulus oocyte complex and improves oocyte developmental competence.BGP-15 prevents the death of neurons in a mouse model of familial dysautonomia.Nicotinamide mononucleotide (NMN) supplementation ameliorates the impact of maternal obesity in mice: comparison with exercise.Live-cell quantification and comparison of mammalian oocyte cytosolic lipid content between species, during development, and in relation to body composition using nonlinear vibrational microscopy.Effects of Female Dietary Restriction in a Rabbit Growth Line During Rearing on Reproductive Performance and Embryo Quality.Human female meiosis revised: new insights into the mechanisms of chromosome segregation and aneuploidies from advanced genomics and time-lapse imaging.In rats gestational iron deficiency does not change body fat or hepatic mitochondria in the aged offspring.Maternal obesity, diabetes during pregnancy and epigenetic mechanisms that influence the developmental origins of cardiometabolic disease in the offspring.Periconception onset diabetes is associated with embryopathy and fetal growth retardation, reproductive tract hyperglycosylation and impaired immune adaptation to pregnancy.Fat eggs shape offspring health.When two obese parents are worse than one! Impacts on embryo and fetal development.Combined parental obesity negatively impacts preimplantation mouse embryo development, kinetics, morphology and metabolism.Influence of follicular fluid and cumulus cells on oocyte quality: clinical implications.Maternal obesity increases the risk of metabolic disease and impacts renal health in offspring.The fetal programming of telomere biology hypothesis: an update.Analysis of Endoplasmic Reticulum (ER) Stress Induced during Somatic Cell Nuclear Transfer (SCNT) Process in Porcine SCNT Embryos.Maternal overnutrition by hypercaloric diets programs hypothalamic mitochondrial fusion and metabolic dysfunction in rat male offspring.
P2860
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P2860
Mitochondrial dysfunction in oocytes of obese mothers: transmission to offspring and reversal by pharmacological endoplasmic reticulum stress inhibitors.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年学术文章
@wuu
2015年学术文章
@zh-cn
2015年学术文章
@zh-hans
2015年学术文章
@zh-my
2015年学术文章
@zh-sg
2015年學術文章
@yue
2015年學術文章
@zh
2015年學術文章
@zh-hant
name
Mitochondrial dysfunction in o ...... c reticulum stress inhibitors.
@en
Mitochondrial dysfunction in o ...... c reticulum stress inhibitors.
@nl
type
label
Mitochondrial dysfunction in o ...... c reticulum stress inhibitors.
@en
Mitochondrial dysfunction in o ...... c reticulum stress inhibitors.
@nl
prefLabel
Mitochondrial dysfunction in o ...... c reticulum stress inhibitors.
@en
Mitochondrial dysfunction in o ...... c reticulum stress inhibitors.
@nl
P2093
P50
P921
P356
P1433
P1476
Mitochondrial dysfunction in o ...... c reticulum stress inhibitors.
@en
P2093
Justin C St John
Linda L Wu
Mark A Febbraio
Miaoxin Chen
Robert J Norman
Siew L Wong
P304
P356
10.1242/DEV.114850
P407
P577
2015-02-01T00:00:00Z